(Q57251838)

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The non-synonymous coding IKr-channel variant KCNH2-K897T is associated with atrial fibrillation: results from a systematic candidate gene-based analysis of KCNH2 (HERG)

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The non-synonymous coding IKr-channel variant KCNH2-K897T is associated with atrial fibrillation: results from a systematic candidate gene-based analysis of KCNH2 (HERG) (English)

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atrial fibrillation

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based on heuristic

inferred from title

Thomas Meitinger

object named as

Thomas Meitinger

15

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Albert Schömig

13

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Albert Schömig

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9

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Thomas Illig

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Britt Maria Beckmann

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Britt-Maria Beckmann

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Moritz F Sinner

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Moritz F. Sinner

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16

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Stefan Kääb

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Wiebke Sauter

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2

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Arne Pfeufer

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Martin Hinterseer

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Martin Hinterseer

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Michael Nabauer

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Michael Näbauer

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7

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Siegfried Perz

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Gerhard Steinbeck

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Gerhard Steinbeck

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Annette Wacker-Gussmann

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Annette Wacker

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author name string

Mahmut Akyol

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Claus Schmitt

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H.-Erich Wichmann

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publication date

25 January 2008

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European Heart Journal

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29

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7

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907-914

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Prevalence of diagnosed atrial fibrillation in adults: national implications for rhythm management and stroke prevention: the AnTicoagulation and Risk Factors in Atrial Fibrillation (ATRIA) Study

1 reference

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De novo KCNQ1 mutation responsible for atrial fibrillation and short QT syndrome in utero.

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Kv1.5 channelopathy due to KCNA5 loss-of-function mutation causes human atrial fibrillation

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Identification of a KCNE2 gain-of-function mutation in patients with familial atrial fibrillation

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Identification of a genetic locus for familial atrial fibrillation

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Genome-wide linkage scan identifies a novel genetic locus on chromosome 5p13 for neonatal atrial fibrillation associated with sudden death and variable cardiomyopathy

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Somatic mutations in the connexin 40 gene (GJA5) in atrial fibrillation

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Short QT syndrome and atrial fibrillation caused by mutation in KCNH2.

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Controversies in atrial fibrillation

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Parental atrial fibrillation as a risk factor for atrial fibrillation in offspring

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Familial aggregation of atrial fibrillation in Iceland

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A common polymorphism in SCN5A is associated with lone atrial fibrillation

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Association of the human minK gene 38G allele with atrial fibrillation: evidence of possible genetic control on the pathogenesis of atrial fibrillation

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Relation of 97T polymorphism in KCNE5 to risk of atrial fibrillation

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Renin-angiotensin system gene polymorphisms and atrial fibrillation

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Variants conferring risk of atrial fibrillation on chromosome 4q25.

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A common polymorphism in KCNH2 (HERG) hastens cardiac repolarization

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Common Variants in Myocardial Ion Channel Genes Modify the QT Interval in the General Population

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Efficiency and power in genetic association studies

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A simple salting out procedure for extracting DNA from human nucleated cells

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Haploview: analysis and visualization of LD and haplotype maps

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The structure of haplotype blocks in the human genome

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From genotypes to genes: doubling the sample size

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Joint analysis is more efficient than replication-based analysis for two-stage genome-wide association studies

1 reference

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Meta-analysis of genetic association studies supports a contribution of common variants to susceptibility to common disease

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Genomic control for association studies

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SNP-based analysis of genetic substructure in the German population.

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New ideas about atrial fibrillation 50 years on.

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Cesium-induced atrial tachycardia degenerating into atrial fibrillation in dogs: atrial torsades de pointes?

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Reinduction of atrial fibrillation immediately after termination of the arrhythmia is mediated by late phase 3 early afterdepolarization-induced triggered activity

1 reference

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Atrial fibrillation-associated minK38G/S polymorphism modulates delayed rectifier current and membrane localization.

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The antihistamine fexofenadine does not affect I(Kr) currents in a case report of drug-induced cardiac arrhythmia

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Molecular and functional characterization of common polymorphisms in HERG (KCNH2) potassium channels

1 reference

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Functional characterization of the common amino acid 897 polymorphism of the cardiac potassium channel KCNH2 (HERG).

1 reference

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KCNH2-K897T is a genetic modifier of latent congenital long-QT syndrome

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Prolonged Atrial Action Potential Durations and Polymorphic Atrial Tachyarrhythmias in Patients with Long QT Syndrome

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21 January 2018

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10.1093/EURHEARTJ/EHM619

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