(Q57804107)

Laura Stronati

2

0 references

Giovanni Di Nardo

Giovanni Di Nardo

4

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Paolo Rossi

Paolo Rossi

7

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Salvatore Cucchiara

Salvatore Cucchiara

8

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Anna Negroni

Anna Negroni

3

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author name string

Maria Pierdomenico

5

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Emanuela del Giudice

The American Journal of Gastroenterology

6

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language of work or name

English

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publication date

26 July 2011

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published in

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volume

106

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issue

11

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page(s)

2029-2040

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cites work

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Hydrophobicity: an ancient damage-associated molecular pattern that initiates innate immune responses.

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HMG-1 as a late mediator of endotoxin lethality in mice

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HMG1 and 2: architectural DNA-binding proteins

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Targeting HMGB1 in inflammation

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High-mobility group box 1 (HMGB1) protein at the crossroads between innate and adaptive immunity

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High-mobility group box 1 (HMGB1) protein: friend and foe.

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High-mobility group box 1 protein (HMGB1): nuclear weapon in the immune arsenal

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The nuclear protein HMGB1 is secreted by monocytes via a non-classical, vesicle-mediated secretory pathway

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HMGB1 as a cytokine and therapeutic target

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Release of chromatin protein HMGB1 by necrotic cells triggers inflammation

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HMGB1 and RAGE in inflammation and cancer

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Extracellular role of HMGB1 in inflammation and sepsis.

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The role of HMGB1 in the pathogenesis of rheumatic disease

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Damage-associated molecular patterns--emerging targets for biologic therapy of childhood arthritides

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High Mobility Group Box 1: a potential therapeutic target for systemic lupus erythematosus.

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HMGB1 B box increases the permeability of Caco-2 enterocytic monolayers and impairs intestinal barrier function in mice

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Ethyl pyruvate decreases HMGB1 release and ameliorates murine colitis

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Development, validation, and evaluation of a pediatric ulcerative colitis activity index: a prospective multicenter study.

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Comparison of several activity indices for the evaluation of endoscopic activity in UC: inter- and intraobserver consistency

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Post-translational methylation of high mobility group box 1 (HMGB1) causes its cytoplasmic localization in neutrophils.

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Anti-high-mobility group box chromosomal protein 1 antibodies improve survival of rats with sepsis.

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The N-terminal domain of thrombomodulin sequesters high-mobility group-B1 protein, a novel antiinflammatory mechanism

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The HMGB1 receptor RAGE mediates ischemic brain damage

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The nuclear factor HMGB1 mediates hepatic injury after murine liver ischemia-reperfusion

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Protective effect of HMGB1 a box on organ injury of acute pancreatitis in mice

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Contributions of high mobility group box protein in experimental and clinical acute lung injury.

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Role of endoscopy in predicting the disease course in inflammatory bowel disease

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Monocytic cells hyperacetylate chromatin protein HMGB1 to redirect it towards secretion

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Nucleocytoplasmic shuttling of HMGB1 is regulated by phosphorylation that redirects it toward secretion

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HMGB1 is phosphorylated by classical protein kinase C and is secreted by a calcium-dependent mechanism

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