(Q59351731)

11

object named as

Zhiheng Xu

0 references

Cui Li

9

object named as

Cui Li

0 references

author name string

Haijun Zhang

1

0 references

Yafei Chang

2

0 references

Longbin Zhang

3

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Seung-Nam Kim

4

0 references

Gaizka Otaegi

5

0 references

Zhen Zhang

6

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Yanzhen Nie

7

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Taufif Mubarak

8

0 references

Tao Sun

12

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language of work or name

English

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publication date

27 September 2018

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number of pages

14

1 reference

on focus list of Wikimedia project

inferred from page(s)

published in

Molecular Neurobiology

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volume

56

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page(s)

4072-4085

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issue

6

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WikiProject Zika Corpus

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exact match

https://scigraph.springernature.com/pub.10.1007/s12035-018-1358-4

0 references

cites work

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IAPs are essential for GDNF-mediated neuroprotective effects in injured motor neurons in vivo

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Cortical excitatory neurons and glia, but not GABAergic neurons, are produced in the Emx1-expressing lineage.

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Regulation of natural cell death in dopaminergic neurons of the substantia nigra by striatal glial cell line-derived neurotrophic factor in vivo.

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Pax6, Tbr2, and Tbr1 are expressed sequentially by radial glia, intermediate progenitor cells, and postmitotic neurons in developing neocortex

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Less is more: progenitor death and cortical size.

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GDNF as a candidate striatal target-derived neurotrophic factor for the development of substantia nigra dopamine neurons.

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The MicroRNA miR-124 promotes neuronal differentiation by triggering brain-specific alternative pre-mRNA splicing

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MicroRNA-9 directs late organizer activity of the midbrain-hindbrain boundary

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MicroRNA-9 modulates Cajal-Retzius cell differentiation by suppressing Foxg1 expression in mouse medial pallium

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miRNAs are essential for survival and differentiation of newborn neurons but not for expansion of neural progenitors during early neurogenesis in the mouse embryonic neocortex

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Different timings of Dicer deletion affect neurogenesis and gliogenesis in the developing mouse central nervous system

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MicroRNAs potentiate neural development

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miR-9 controls the timing of neurogenesis through the direct inhibition of antagonistic factors

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Convergent repression of Foxp2 3'UTR by miR-9 and miR-132 in embryonic mouse neocortex: implications for radial migration of neurons.

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MicroRNA cluster miR-17-92 regulates neural stem cell expansion and transition to intermediate progenitors in the developing mouse neocortex

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Tuning the cell fate of neurons and glia by microRNAs

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MicroRNA function is required for neurite outgrowth of mature neurons in the mouse postnatal cerebral cortex

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MicroRNA-9 promotes the switch from early-born to late-born motor neuron populations by regulating Onecut transcription factor expression.

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Zika Virus Infects Human Cortical Neural Progenitors and Attenuates Their Growth

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Vertical transmission of Zika virus targeting the radial glial cells affects cortex development of offspring mice

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Zika Virus Disrupts Neural Progenitor Development and Leads to Microcephaly in Mice

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Western Zika Virus in Human Fetal Neural Progenitors Persists Long Term with Partial Cytopathic and Limited Immunogenic Effects

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The Brazilian Zika virus strain causes birth defects in experimental models

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Zika Virus NS4A and NS4B Proteins Deregulate Akt-mTOR Signaling in Human Fetal Neural Stem Cells to Inhibit Neurogenesis and Induce Autophagy

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Zika Virus Infects Neural Progenitors in the Adult Mouse Brain and Alters Proliferation

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Zika Virus Disrupts Phospho-TBK1 Localization and Mitosis in Human Neuroepithelial Stem Cells and Radial Glia

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12 December 2020

Molecular signatures associated with ZIKV exposure in human cortical neural progenitors

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Zika virus infection disrupts neurovascular development and results in postnatal microcephaly with brain damage.

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Recent Zika Virus Isolates Induce Premature Differentiation of Neural Progenitors in Human Brain Organoids

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High-Content Screening in hPSC-Neural Progenitors Identifies Drug Candidates that Inhibit Zika Virus Infection in Fetal-like Organoids and Adult Brain.

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Zika-Virus-Encoded NS2A Disrupts Mammalian Cortical Neurogenesis by Degrading Adherens Junction Proteins

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A single mutation in the prM protein of Zika virus contributes to fetal microcephaly.

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12 December 2020

Self-Organized Cerebral Organoids with Human-Specific Features Predict Effective Drugs to Combat Zika Virus Infection

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12 December 2020

Intranasal infection and contact transmission of Zika virus in guinea pigs

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