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Multiple sclerosis-associated single-nucleotide polymorphisms in CLEC16A correlate with reduced SOCS1 and DEXI expression in the thymus
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Europe PubMed Central
PubMed publication ID
23151489
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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:23151489%20AND%20SRC:MED&resulttype=core&format=json
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17 February 2020
title
Multiple sclerosis-associated single-nucleotide polymorphisms in CLEC16A correlate with reduced SOCS1 and DEXI expression in the thymus
(English)
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stated in
Europe PubMed Central
PubMed publication ID
23151489
reference URL
https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:23151489%20AND%20SRC:MED&resulttype=core&format=json
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17 February 2020
main subject
single-nucleotide polymorphism
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Maria K. Dahle
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3
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Europe PubMed Central
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23151489
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17 February 2020
Anne Spurkland
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6
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23151489
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17 February 2020
Hanne F Harbo
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5
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H F Harbo
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23151489
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17 February 2020
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I S Leikfoss
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1
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23151489
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17 February 2020
I-L Mero
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2
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23151489
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17 February 2020
B A Lie
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4
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23151489
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17 February 2020
T Berge
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23151489
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17 February 2020
publication date
15 November 2012
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23151489
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retrieved
17 February 2020
published in
Genes and Immunity
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PubMed publication ID
23151489
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17 February 2020
volume
14
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23151489
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17 February 2020
issue
1
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23151489
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17 February 2020
page(s)
62-66
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Europe PubMed Central
PubMed publication ID
23151489
reference URL
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retrieved
17 February 2020
exact match
https://scigraph.springernature.com/pub.10.1038/gene.2012.52
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The expanding genetic overlap between multiple sclerosis and type I diabetes
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Polymorphisms in CLEC16A and CIITA at 16p13 are associated with primary adrenal insufficiency
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Specific association of a CLEC16A/KIAA0350 polymorphism with NOD2/CARD15(-) Crohn's disease patients
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Replication of KIAA0350, IL2RA, RPL5 and CD58 as multiple sclerosis susceptibility genes in Australians
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Genetic risk and a primary role for cell-mediated immune mechanisms in multiple sclerosis
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Association of IFIH1 and other autoimmunity risk alleles with selective IgA deficiency
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A CLEC16A variant confers risk for juvenile idiopathic arthritis and anti-cyclic citrullinated peptide antibody negative rheumatoid arthritis
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Exploring the CLEC16A gene reveals a MS-associated variant with correlation to the relative expression of CLEC16A isoforms in thymus
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Risk alleles for multiple sclerosis identified by a genomewide study
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More CLEC16A gene variants associated with multiple sclerosis
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CIITA variation in the presence of HLA-DRB1*1501 increases risk for multiple sclerosis
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A cytokine gene screen uncovers SOCS1 as genetic risk factor for multiple sclerosis
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Genetic variation in the IL7RA/IL7 pathway increases multiple sclerosis susceptibility
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Interrogating the complex role of chromosome 16p13.13 in multiple sclerosis susceptibility: independent genetic signals in the CIITA-CLEC16A-SOCS1 gene complex
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Replication of CD58 and CLEC16A as genome-wide significant risk genes for multiple sclerosis
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Long-range DNA looping and gene expression analyses identify DEXI as an autoimmune disease candidate gene
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Candidate causal regulatory effects by integration of expression QTLs with complex trait genetic associations
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SOCS1 negatively regulates the production of Foxp3+ CD4+ T cells in the thymus.
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T-helper 17 cells expand in multiple sclerosis and are inhibited by interferon-beta.
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SOCS1 is essential for regulatory T cell functions by preventing loss of Foxp3 expression as well as IFN-{gamma} and IL-17A production
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Glucocorticoids target suppressor of cytokine signaling 1 (SOCS1) and type 1 interferons to regulate Toll-like receptor-induced STAT1 activation
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Identifiers
DOI
10.1038/GENE.2012.52
1 reference
stated in
Europe PubMed Central
PubMed publication ID
23151489
reference URL
https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:23151489%20AND%20SRC:MED&resulttype=core&format=json
retrieved
17 February 2020
PubMed publication ID
23151489
1 reference
stated in
Europe PubMed Central
PubMed publication ID
23151489
reference URL
https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:23151489%20AND%20SRC:MED&resulttype=core&format=json
retrieved
17 February 2020
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