(Q73270833)

English

Calcium influx triggers the sequential proteolysis of extracellular and cytoplasmic domains of E-cadherin, leading to loss of beta-catenin from cell-cell contacts

scientific article published on 01 November 1999

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scholarly article

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Europe PubMed Central

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:10597309%20AND%20SRC:MED&resulttype=core&format=json

2 November 2019

Calcium influx triggers the sequential proteolysis of extracellular and cytoplasmic domains of E-cadherin, leading to loss of beta-catenin from cell-cell contacts (English)

1 reference

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2 November 2019

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Ito K

1

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2 November 2019

Okamoto I

2

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2 November 2019

Araki N

3

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2 November 2019

Kawano Y

4

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2 November 2019

Nakao M

5

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2 November 2019

Fujiyama S

6

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2 November 2019

Tomita K

7

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2 November 2019

Mimori T

8

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2 November 2019

Saya H

9

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2 November 2019

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publication date

1 November 1999

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2 November 2019

1 reference

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2 November 2019

18

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2 November 2019

7080-7090

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2 November 2019

50

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2 November 2019

https://scigraph.springernature.com/pub.10.1038/sj.onc.1203191

0 references

Calcium - a life and death signal

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Cleavage of beta-catenin and plakoglobin and shedding of VE-cadherin during endothelial apoptosis: evidence for a role for caspases and metalloproteinases

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Inactivation of the E-cadherin-mediated cell adhesion system in human cancers

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Membrane protein secretases

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Nuclear localization of beta-catenin by interaction with transcription factor LEF-1

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Co-culture of human breast adenocarcinoma MCF-7 cells and human dermal fibroblasts enhances the production of matrix metalloproteinases 1, 2 and 3 in fibroblasts

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Soluble E-cadherin fragments increased in circulation of cancer patients

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Matrix metalloproteinase stromelysin-1 triggers a cascade of molecular alterations that leads to stable epithelial-to-mesenchymal conversion and a premalignant phenotype in mammary epithelial cells

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