(Q73450523)

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The neuronal Golgi apparatus is fragmented in transgenic mice expressing a mutant human SOD1, but not in mice expressing the human NF-H gene

scientific article published on 01 February 2000

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4 November 2019

The neuronal Golgi apparatus is fragmented in transgenic mice expressing a mutant human SOD1, but not in mice expressing the human NF-H gene (English)

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4 November 2019

Golgi apparatus

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A Stieber

1

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4 November 2019

J O Gonatas

2

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4 November 2019

J Collard

3

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4 November 2019

J Meier

4

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4 November 2019

J Julien

5

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4 November 2019

P Schweitzer

6

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4 November 2019

N K Gonatas

7

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4 November 2019

publication date

1 February 2000

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4 November 2019

Journal of the Neurological Sciences

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4 November 2019

173

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4 November 2019

1

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4 November 2019

63-72

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4 November 2019

Mutations in Cu/Zn superoxide dismutase gene are associated with familial amyotrophic lateral sclerosis

1 reference

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7 January 2021

Amyotrophic lateral sclerosis and structural defects in Cu,Zn superoxide dismutase

1 reference

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7 January 2021

Genetics of amyotrophic lateral sclerosis

1 reference

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7 January 2021

Motor neuron degeneration in mice that express a human Cu,Zn superoxide dismutase mutation

1 reference

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7 January 2021

Variants of the heavy neurofilament subunit are associated with the development of amyotrophic lateral sclerosis

1 reference

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7 January 2021

Progressive neuronopathy in transgenic mice expressing the human neurofilament heavy gene: a mouse model of amyotrophic lateral sclerosis

1 reference

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7 January 2021

Increased expression of neurofilament subunit NF-L produces morphological alterations that resemble the pathology of human motor neuron disease

1 reference

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7 January 2021

Fragmentation of the Golgi apparatus of motor neurons in amyotrophic lateral sclerosis revealed by organelle-specific antibodies

1 reference

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7 January 2021

The Golgi apparatus of motor neurons in amyotrophic lateral sclerosis.

1 reference

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7 January 2021

The Golgi apparatus of spinal cord motor neurons in transgenic mice expressing mutant Cu,Zn superoxide dismutase becomes fragmented in early, preclinical stages of the disease

1 reference

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7 January 2021

Fragmentation of the Golgi apparatus of Betz cells in patients with amyotrophic lateral sclerosis.

1 reference

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7 January 2021

HISTOCHEMICAL AND ULTRASTRUCTURAL STUDIES ON HELA CELL CULTURES EXPOSED TO SPINDLE INHIBITORS WITH SPECIAL REFERENCE TO THE INTERPHASE CELL

1 reference

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7 January 2021

Evidence that all newly synthesized proteins destined for fast axonal transport pass through the Golgi apparatus

1 reference

https://api.crossref.org/works/10.1016%2FS0022-510X%2899%2900301-9

7 January 2021

A low expressor line of transgenic mice carrying a mutant human Cu,Zn superoxide dismutase (SOD1) gene develops pathological changes that most closely resemble those in human amyotrophic lateral sclerosis

1 reference

https://api.crossref.org/works/10.1016%2FS0022-510X%2899%2900301-9

7 January 2021

Immunocytochemical visualization of the Golgi apparatus in several species, including human, and tissues with an antiserum against MG-160, a sialoglycoprotein of rat Golgi apparatus

1 reference

https://api.crossref.org/works/10.1016%2FS0022-510X%2899%2900301-9

7 January 2021

The early stages of absorption of injected horseradish peroxidase in the proximal tubules of mouse kidney: ultrastructural cytochemistry by a new technique

1 reference

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7 January 2021

Immunoperoxidase methods for the localization of antigens in cultured cells and tissue sections by electron microscopy

1 reference

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7 January 2021

Developmental regulation of membrane traffic organization during synaptogenesis in mouse diaphragm muscle

1 reference

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7 January 2021

Speculating about spectrin: new insights into the Golgi-associated cytoskeleton

1 reference

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7 January 2021

Superoxide dismutase 1 with mutations linked to familial amyotrophic lateral sclerosis possesses significant activity

1 reference

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7 January 2021

Defective axonal transport in a transgenic mouse model of amyotrophic lateral sclerosis

1 reference

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7 January 2021

Neurofilaments and orthograde transport are reduced in ventral root axons of transgenic mice that express human SOD1 with a G93A mutation

1 reference

https://api.crossref.org/works/10.1016%2FS0022-510X%2899%2900301-9

7 January 2021

SOD1 mutation is associated with accumulation of neurofilaments in amyotrophic lateral sclerosis.

1 reference

https://api.crossref.org/works/10.1016%2FS0022-510X%2899%2900301-9

7 January 2021

Transgenic mice carrying a human mutant superoxide dismutase transgene develop neuronal cytoskeletal pathology resembling human amyotrophic lateral sclerosis lesions

1 reference

https://api.crossref.org/works/10.1016%2FS0022-510X%2899%2900301-9

7 January 2021

Protective effect of neurofilament heavy gene overexpression in motor neuron disease induced by mutant superoxide dismutase

1 reference

https://api.crossref.org/works/10.1016%2FS0022-510X%2899%2900301-9

7 January 2021

Absence of neurofilaments reduces the selective vulnerability of motor neurons and slows disease caused by a familial amyotrophic lateral sclerosis-linked superoxide dismutase 1 mutant

1 reference

https://api.crossref.org/works/10.1016%2FS0022-510X%2899%2900301-9

7 January 2021

Fine structural study of neurofibrillary changes in a family with amyotrophic lateral sclerosis

1 reference

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7 January 2021

Focal accumulation of phosphorylated neurofilaments within anterior horn cell in familial amyotrophic lateral sclerosis

1 reference

https://api.crossref.org/works/10.1016%2FS0022-510X%2899%2900301-9

7 January 2021

Superoxide dismutase 1 subunits with mutations linked to familial amyotrophic lateral sclerosis do not affect wild-type subunit function.

1 reference

https://api.crossref.org/works/10.1016%2FS0022-510X%2899%2900301-9

7 January 2021

Altered reactivity of superoxide dismutase in familial amyotrophic lateral sclerosis

1 reference

https://api.crossref.org/works/10.1016%2FS0022-510X%2899%2900301-9

7 January 2021

A gain-of-function of an amyotrophic lateral sclerosis-associated Cu,Zn-superoxide dismutase mutant: An enhancement of free radical formation due to a decrease in Km for hydrogen peroxide

1 reference

https://api.crossref.org/works/10.1016%2FS0022-510X%2899%2900301-9

7 January 2021

Aggregation and Motor Neuron Toxicity of an ALS-Linked SOD1 Mutant Independent from Wild-Type SOD1

1 reference

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7 January 2021

Peroxynitrite-mediated tyrosine nitration catalyzed by superoxide dismutase

1 reference

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7 January 2021

Kinetics of superoxide dismutase- and iron-catalyzed nitration of phenolics by peroxynitrite

1 reference

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7 January 2021

ALS, SOD and peroxynitrite

1 reference

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7 January 2021

The familial amyotrophic lateral sclerosis-associated amino acid substitutions E100G, G93A, and G93R do not influence the rate of inactivation of copper- and zinc-containing superoxide dismutase by H2O2

1 reference

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7 January 2021

Pathogenesis of two axonopathies does not require axonal neurofilaments.

1 reference

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7 January 2021

Identifiers

10.1016/S0022-510X(99)00301-9

1 reference

Europe PubMed Central

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4 November 2019

1 reference

Europe PubMed Central

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:10675581%20AND%20SRC:MED&resulttype=core&format=json

4 November 2019

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