(Q73604815)

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Cyclin kinase inhibitors are increased during experimental membranous nephropathy: potential role in limiting glomerular epithelial cell proliferation in vivo

scientific article published on 01 August 1997

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Europe PubMed Central

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:9263996%20AND%20SRC:MED&resulttype=core&format=json

5 November 2019

Cyclin kinase inhibitors are increased during experimental membranous nephropathy: potential role in limiting glomerular epithelial cell proliferation in vivo (English)

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5 November 2019

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S J Shankland

1

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5 November 2019

J Floege

2

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5 November 2019

S E Thomas

3

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5 November 2019

M Nangaku

4

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5 November 2019

C Hugo

5

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5 November 2019

J Pippin

6

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5 November 2019

K Henne

7

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5 November 2019

D M Hockenberry

8

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5 November 2019

R J Johnson

9

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5 November 2019

W G Couser

10

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5 November 2019

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1 August 1997

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5 November 2019

Kidney International

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5 November 2019

52

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5 November 2019

2

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5 November 2019

404-413

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5 November 2019

https://scigraph.springernature.com/pub.10.1038/ki.1997.347

0 references

Podocyte damage is a critical step in the development of glomerulosclerosis in the uninephrectomised-desoxycorticosterone hypertensive rat

1 reference

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7 January 2021

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Rat glomerular mesangial cells synthesize basic fibroblast growth factor. Release, upregulated synthesis, and mitogenicity in mesangial proliferative glomerulonephritis

1 reference

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Development of tubular and glomerular cells of the kidney

1 reference

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Loss of mitotic activity and the expression of vimentin in glomerular epithelial cells of developing human kidneys

1 reference

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Mitosis and the presence of binucleate cells among glomerular podocytes in diseased human kidneys

1 reference

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Long-term treatment of rats with FGF-2 results in focal segmental glomerulosclerosis

1 reference

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7 January 2021

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Basic fibroblast growth factor augments podocyte injury and induces glomerulosclerosis in rats with experimental membranous nephropathy

1 reference

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7 January 2021

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Animal cell cycles and their control

1 reference

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7 January 2021

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Mammalian G1 cyclins

1 reference

https://api.crossref.org/works/10.1038%2FKI.1997.347

7 January 2021

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Principles of CDK regulation

1 reference

https://api.crossref.org/works/10.1038%2FKI.1997.347

7 January 2021

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Cdc2 regulatory factors

1 reference

https://api.crossref.org/works/10.1038%2FKI.1997.347

7 January 2021

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Changes in cell-cycle protein expression during experimental mesangial proliferative glomerulonephritis

1 reference

https://api.crossref.org/works/10.1038%2FKI.1997.347

7 January 2021

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Experimental membranous glomerulonephritis in rats. Quantitative studies of glomerular immune deposit formation in isolated glomeruli and whole animals

1 reference

https://api.crossref.org/works/10.1038%2FKI.1997.347

7 January 2021

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Increased urinary excretion of C5b-9 distinguishes passive Heymann nephritis in the rat.

1 reference

https://api.crossref.org/works/10.1038%2FKI.1997.347

7 January 2021

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Mesangial cell apoptosis: the major mechanism for resolution of glomerular hypercellularity in experimental mesangial proliferative nephritis

1 reference

https://api.crossref.org/works/10.1038%2FKI.1997.347

7 January 2021

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The glomerular response to injury: progression or resolution?

1 reference

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7 January 2021

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Cyclins and cyclin-dependent kinases: a biochemical view

1 reference

https://api.crossref.org/works/10.1038%2FKI.1997.347

7 January 2021

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Cyclin D1 is a nuclear protein required for cell cycle progression in G1

1 reference

https://api.crossref.org/works/10.1038%2FKI.1997.347

7 January 2021

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Human cyclin E, a nuclear protein essential for the G1-to-S phase transition

1 reference

https://api.crossref.org/works/10.1038%2FKI.1997.347

7 January 2021

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Cyclin A is required at two points in the human cell cycle

1 reference

https://api.crossref.org/works/10.1038%2FKI.1997.347

7 January 2021

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Inhibitors of mammalian G1 cyclin-dependent kinases

1 reference

https://api.crossref.org/works/10.1038%2FKI.1997.347

7 January 2021

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WAF1, a potential mediator of p53 tumor suppression

1 reference

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7 January 2021

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The p21 Cdk-interacting protein Cip1 is a potent inhibitor of G1 cyclin-dependent kinases

1 reference

https://api.crossref.org/works/10.1038%2FKI.1997.347

7 January 2021

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Cloning of senescent cell-derived inhibitors of DNA synthesis using an expression screen

1 reference

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p27, a novel inhibitor of G1 cyclin-Cdk protein kinase activity, is related to p21

1 reference

https://api.crossref.org/works/10.1038%2FKI.1997.347

7 January 2021

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Cloning of p27Kip1, a cyclin-dependent kinase inhibitor and a potential mediator of extracellular antimitogenic signals

1 reference

https://api.crossref.org/works/10.1038%2FKI.1997.347

7 January 2021

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p27Kip1, a cyclin-Cdk inhibitor, links transforming growth factor-beta and contact inhibition to cell cycle arrest

1 reference

https://api.crossref.org/works/10.1038%2FKI.1997.347

7 January 2021

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Inhibition of cyclin-dependent kinases by p21

1 reference

https://api.crossref.org/works/10.1038%2FKI.1997.347

7 January 2021

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Requirement of p27Kip1 for restriction point control of the fibroblast cell cycle

1 reference

https://api.crossref.org/works/10.1038%2FKI.1997.347

7 January 2021

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Impact of the cyclin-dependent kinase inhibitor p27Kip1 on resistance of tumor cells to anticancer agents

1 reference

https://api.crossref.org/works/10.1038%2FKI.1997.347

7 January 2021

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Differential expression of transforming growth factor-beta isoforms and receptors in experimental membranous nephropathy

1 reference

https://api.crossref.org/works/10.1038%2FKI.1997.347

7 January 2021

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Renal microvascular injury induced by antibody to glomerular endothelial cells is mediated by C5b-9

1 reference

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Role of the complement membrane attack complex (C5b-9) in mediating experimental mesangioproliferative glomerulonephritis

1 reference

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Complement-induced glomerular epithelial cell injury. Role of the membrane attack complex in rat membranous nephropathy

1 reference

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based on heuristic

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