(Q74045139)

English

Interaction of the receptor for advanced glycation end products (RAGE) with transthyretin triggers nuclear transcription factor kB (NF-kB) activation

scientific article published on 01 July 2000

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scholarly article

1 reference

Europe PubMed Central

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:10908156%20AND%20SRC:MED&resulttype=core&format=json

8 November 2019

Interaction of the receptor for advanced glycation end products (RAGE) with transthyretin triggers nuclear transcription factor kB (NF-kB) activation (English)

1 reference

Europe PubMed Central

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:10908156%20AND%20SRC:MED&resulttype=core&format=json

8 November 2019

author name string

M M Sousa

1

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8 November 2019

S D Yan

2

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8 November 2019

D Stern

3

1 reference

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8 November 2019

M J Saraiva

4

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8 November 2019

publication date

1 July 2000

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8 November 2019

Laboratory Investigation

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8 November 2019

80

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8 November 2019

7

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8 November 2019

1101-1110

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8 November 2019

https://scigraph.springernature.com/pub.10.1038/labinvest.3780116

0 references

Thyroxine binding to transthyretin Met 119. Comparative studies of different heterozygotic carriers and structural analysis

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Oxidative stress is found in amyloid deposits in systemic amyloidosis

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Hydrogen peroxide mediates amyloid beta protein toxicity

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Strjcture of human plasma prealbumin at 2-5 A resolution. A preliminary report on the polypeptide chain conformation, quaternary structure and thyroxine binding

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The serpin-enzyme complex receptor recognizes soluble, nontoxic amyloid-beta peptide but not aggregated, cytotoxic amyloid-beta peptide

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‘In vitro’ amyloid fibril formation from transthyretin: the influence of ions and the amyloidogenicity of TTR variants

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Accurate transcription initiation by RNA polymerase II in a soluble extract from isolated mammalian nuclei

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Amyloid-beta peptide-receptor for advanced glycation endproduct interaction elicits neuronal expression of macrophage-colony stimulating factor: a proinflammatory pathway in Alzheimer disease

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Senile plaques stimulate microglia to release a neurotoxin found in Alzheimer brain.

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The HHQK domain of beta-amyloid provides a structural basis for the immunopathology of Alzheimer's disease.

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A model for beta-amyloid aggregation and neurotoxicity based on free radical generation by the peptide: relevance to Alzheimer disease

1 reference

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7 January 2021

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RAGE mediates a novel proinflammatory axis: a central cell surface receptor for S100/calgranulin polypeptides

1 reference

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7 January 2021

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The receptor for advanced glycation end products (RAGE) is a cellular binding site for amphoterin. Mediation of neurite outgrowth and co-expression of rage and amphoterin in the developing nervous system

1 reference

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N(epsilon)-(carboxymethyl)lysine adducts of proteins are ligands for receptor for advanced glycation end products that activate cell signaling pathways and modulate gene expression

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Activation of macrophages by Alzheimer beta amyloid peptide.

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Neuronal cell death in Alzheimer's disease correlates with apoE uptake and intracellular Abeta stabilization

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Characterization and functional analysis of the promoter of RAGE, the receptor for advanced glycation end products

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7 January 2021

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Microglia in degenerative neurological disease.

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The receptor for advanced glycation end products (RAGE) is a central mediator of the interaction of AGE-beta2microglobulin with human mononuclear phagocytes via an oxidant-sensitive pathway. Implications for the pathogenesis of dialysis-related amyl

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7 January 2021

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The structure of human retinol-binding protein (RBP) with its carrier protein transthyretin reveals an interaction with the carboxy terminus of RBP

1 reference

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7 January 2021

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The Tetrameric Protein Transthyretin Dissociates to a Non-native Monomer in Solution

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7 January 2021

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Transthyretin mutations in health and disease.

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Amyloid fibril protein in familial amyloidotic polyneuropathy, Portuguese type. Definition of molecular abnormality in transthyretin (prealbumin).

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7 January 2021

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Receptor for advanced glycation end products (AGEs) has a central role in vessel wall interactions and gene activation in response to circulating AGE proteins

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Activation of receptor for advanced glycation end products: a mechanism for chronic vascular dysfunction in diabetic vasculopathy and atherosclerosis

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7 January 2021

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The crystal structure of amyloidogenic Leu55 --> Pro transthyretin variant reveals a possible pathway for transthyretin polymerization into amyloid fibrils

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Translating cell biology into therapeutic advances in Alzheimer's disease

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Role of the beta-amyloid protein in Alzheimer's disease.

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Oxidative damage in Alzheimer's.

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Binding of beta-amyloid to the p75 neurotrophin receptor induces apoptosis. A possible mechanism for Alzheimer's disease.

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RAGE and amyloid-beta peptide neurotoxicity in Alzheimer's disease

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Non-enzymatically glycated tau in Alzheimer's disease induces neuronal oxidant stress resulting in cytokine gene expression and release of amyloid beta-peptide.

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