(Q80442353)

English

Impairment of Na(+),K(+)-ATPase in CD95(APO-1)-induced human T-cell leukemia cell apoptosis mediated by glutathione depletion and generation of hydrogen peroxide

scientific article published on 07 June 2007

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scholarly article

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Europe PubMed Central

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:17554377%20AND%20SRC:MED&resulttype=core&format=json

31 December 2019

Impairment of Na(+),K(+)-ATPase in CD95(APO-1)-induced human T-cell leukemia cell apoptosis mediated by glutathione depletion and generation of hydrogen peroxide (English)

1 reference

Europe PubMed Central

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31 December 2019

1 reference

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T-cell leukemia

1 reference

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author name string

Yin W

1

1 reference

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31 December 2019

Cheng W

2

1 reference

Europe PubMed Central

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31 December 2019

Shen W

3

1 reference

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31 December 2019

Shu L

4

1 reference

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31 December 2019

Zhao J

5

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31 December 2019

Zhang J

6

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31 December 2019

Hua ZC

7

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31 December 2019

publication date

7 June 2007

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31 December 2019

1 reference

Europe PubMed Central

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31 December 2019

21

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31 December 2019

8

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31 December 2019

1669-1678

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31 December 2019

https://scigraph.springernature.com/pub.10.1038/sj.leu.2404791

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Inhibition of Na+/K(+)-ATPase may be one mechanism contributing to potassium efflux and cell shrinkage in CD95-induced apoptosis

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Stimulation of Na,K-ATPase by low potassium is dependent on transferrin

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Fas(CD95)/FasL interactions required for programmed cell death after T-cell activation.

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Opposing effects of glutathione depletion and follicle-stimulating hormone on reactive oxygen species and apoptosis in cultured preovulatory rat follicles.

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Highly selective and prolonged depletion of mitochondrial glutathione in astrocytes markedly increases sensitivity to peroxynitrite.

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Role of glutathione depletion and reactive oxygen species generation in apoptotic signaling in a human B lymphoma cell line.

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Dopamine-induced exocytosis of Na,K-ATPase is dependent on activation of protein kinase C-epsilon and -delta

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Stimulation of Na,K-ATPase by low potassium requires reactive oxygen species

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