(Q80539473)

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Self-terminating AF depends on electrical remodeling while persistent AF depends on additional structural changes in a rapid atrially paced sheep model

scientific article published on 21 May 2007

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1 January 2020

Self-terminating AF depends on electrical remodeling while persistent AF depends on additional structural changes in a rapid atrially paced sheep model (English)

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Tania Roskams

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Wim Anné

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Patricia Holemans

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Frank Beckers

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Ilse Lenaerts

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Hugo Ector

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Hein Heidbüchel

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21 May 2007

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Journal of Molecular and Cellular Cardiology

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148-158

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1 January 2020

Atrial fibrillation begets atrial fibrillation. A study in awake chronically instrumented goats

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Promotion of atrial fibrillation by heart failure in dogs: atrial remodeling of a different sort

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Effects of angiotensin-converting enzyme inhibition on the development of the atrial fibrillation substrate in dogs with ventricular tachypacing-induced congestive heart failure

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Different patterns of angiotensin II and atrial natriuretic peptide secretion in a sheep model of atrial fibrillation

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Increased expression of extracellular signal-regulated kinase and angiotensin-converting enzyme in human atria during atrial fibrillation

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Angiotensin II antagonist prevents electrical remodeling in atrial fibrillation

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Atrial fibrillation and heart failure: natural history and pharmacological treatment

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Atrial fibrillation after radiofrequency ablation of atrial flutter: preventive effect of angiotensin converting enzyme inhibitors, angiotensin II receptor blockers, and diuretics

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Matrix metalloproteinases/tissue inhibitors of metalloproteinases: relationship between changes in proteolytic determinants of matrix composition and structural, functional, and clinical manifestations of hypertensive heart disease

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Effect of different pacing protocols on the induction of atrial fibrillation in a transvenously paced sheep model.

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Effect of short-term treatment with bumetanide, quinapril and low-sodium diet on dogs with moderate congestive heart failure

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Adding angiotensin II type 1 receptor blockade to angiotensin-converting enzyme inhibition limits myocyte remodeling after myocardial infarction

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Wavelength and atrial vulnerability: an endocavitary approach in humans.

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Matrix metalloproteinases and atrial remodeling in patients with mitral valve disease and atrial fibrillation

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Fibrosis of the left atria during progression of heart failure is associated with increased matrix metalloproteinases in the rat

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Effects of experimental heart failure on atrial cellular and ionic electrophysiology

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Consequences of atrial tachycardia-induced remodeling depend on the preexisting atrial substrate

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Electrical remodeling and atrial dilation during atrial tachycardia are influenced by ventricular rate: role of developing tachycardiomyopathy

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Impaired myofibrillar energetics and oxidative injury during human atrial fibrillation

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Angiotensin II, oxidative stress, and extracellular matrix degradation during transition to LV failure in rats with hypertension

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Activation of matrix metalloproteinases precedes left ventricular remodeling in hypertensive heart failure rats: its inhibition as a primary effect of Angiotensin-converting enzyme inhibitor

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7 January 2021

Effects of angiotensin II type 1 receptor antagonist on electrical and structural remodeling in atrial fibrillation

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Effects of inhibiting Na(+)/H(+)-exchange or angiotensin converting enzyme on atrial tachycardia-induced remodeling

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Atrial extracellular matrix remodeling and the maintenance of atrial fibrillation

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Atrial fibrillation: a perspective: thinking inside and outside the box.

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Electrical Remodeling of the Atria in Congestive Heart Failure

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Action potential remodeling in the human right atrium with chronic lone atrial fibrillation

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Therapy with angiotensin-converting enzyme inhibitors, angiotensin II receptor blockers, and statins: no effect on ablation outcome after ablation of atrial fibrillation

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Ventricular high-rate episodes in pacemaker diagnostics identify a high-risk subgroup of patients with tachy-brady syndrome.

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10.1016/J.YJMCC.2007.05.010

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1 January 2020

1 reference

Europe PubMed Central

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1 January 2020

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