(Q82577221)

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Blockade of complement activation product C5a activity using specific antibody attenuates intestinal damage in trinitrobenzene sulfonic acid induced model of colitis

scientific article published on 22 November 2010

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scholarly article

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16 January 2020

Blockade of complement activation product C5a activity using specific antibody attenuates intestinal damage in trinitrobenzene sulfonic acid induced model of colitis (English)

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16 January 2020

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Renfeng Guo

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Beifen Shen

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Guojiang Chen

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Gencheng Han

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16 January 2020

author name string

Yuemei Yang

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Xudong Gao

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Yan Dou

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Huihui Wang

5

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Renxi Wang

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Jianan Wang

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Liyan Wang

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Xinying Li

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He Xiao

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Yan Li

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publication date

22 November 2010

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Laboratory Investigation

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91

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472-483

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16 January 2020

https://scigraph.springernature.com/pub.10.1038/labinvest.2010.183

0 references

Inflammatory bowel disease: etiology and pathogenesis

1 reference

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Experimental models of inflammatory bowel disease

1 reference

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The immunology of mucosal models of inflammation

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The IL23 axis plays a key role in the pathogenesis of IBD.

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Interleukin-23 drives innate and T cell-mediated intestinal inflammation

1 reference

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Unravelling the pathogenesis of inflammatory bowel disease

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Importance of innate immunity and collagen binding integrin alpha1beta1 in TNBS-induced colitis

1 reference

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Differential alteration in intestinal epithelial cell expression of toll-like receptor 3 (TLR3) and TLR4 in inflammatory bowel disease

1 reference

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Epithelial NEMO links innate immunity to chronic intestinal inflammation

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Role of C5a in inflammatory responses

1 reference

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Complement activation and inhibition in experimental models of arthritis

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The role of the complement system in ischemia-reperfusion injury

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The anaphylatoxins bridge innate and adaptive immune responses in allergic asthma.

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Expression and function of the C5a receptor in rat alveolar epithelial cells

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Expression and function of C5a receptor in mouse microvascular endothelial cells

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Functions of anaphylatoxin C5a in rat liver: direct and indirect actions on nonparenchymal and parenchymal cells

1 reference

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Extrahepatic synthesis of complement proteins in inflammation

1 reference

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Disruption of T helper 2-immune responses in Epstein-Barr virus-induced gene 3-deficient mice

1 reference

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The transcription factor T-bet regulates mucosal T cell activation in experimental colitis and Crohn's disease

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Complement C5a regulates IL-17 by affecting the crosstalk between DC and gammadelta T cells in CLP-induced sepsis

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Improved procedure for the isolation of functionally active lymphoid cells from the murine intestine

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The C terminus of the human C5a receptor (CD88) is required for normal ligand-dependent receptor internalization

1 reference

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Resveratrol attenuates C5a-induced inflammatory responses in vitro and in vivo by inhibiting phospholipase D and sphingosine kinase activities

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Neutrophil C5a receptor and the outcome in a rat model of sepsis

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Distinct cytokine patterns identified from multiplex profiles of murine DSS and TNBS-induced colitis

1 reference

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Immune modulatory treatment of trinitrobenzene sulfonic acid colitis with calcitriol is associated with a change of a T helper (Th) 1/Th17 to a Th2 and regulatory T cell profile

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Expression of cell membrane complement regulatory glycoproteins along the normal and diseased human gastrointestinal tract

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Intestinal trefoil factor induces decay-accelerating factor expression and enhances the protective activities against complement activation in intestinal epithelial cells

1 reference

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Complement-targeted therapeutics

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Complement. First of two parts

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