(Q84705537)

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Liver mitochondrial function and redox status in an experimental model of non-alcoholic fatty liver disease induced by monosodium L-glutamate in rats

scientific article published on 29 July 2011

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scholarly article

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9 February 2020

Liver mitochondrial function and redox status in an experimental model of non-alcoholic fatty liver disease induced by monosodium L-glutamate in rats (English)

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9 February 2020

metabolic dysfunction–associated steatotic liver disease

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based on heuristic

inferred from title

non-alcoholic fatty liver

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based on heuristic

inferred from title

Emy Luiza Ishii Iwamoto

2

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author name string

Murilo de Oliveira Lazarin

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Nair Seiko Yamamoto

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Rodrigo Polimeni Constantin

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Rosângela Fernandes Garcia

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Cecília E Mareze da Costa

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Adriana de Souza Vitoriano

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Monique Cristine de Oliveira

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Clairce L Salgueiro-Pagadigorria

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29 July 2011

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Experimental and Molecular Pathology

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91

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3

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687-694

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9 February 2020

Recent concepts in non-alcoholic fatty liver disease

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Nonalcoholic fatty liver disease

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Obesity-related fatty liver is unchanged in mice deficient for mitochondrial uncoupling protein 2.

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Long-chain fatty acids regulate liver carnitine palmitoyltransferase I gene (L-CPT I) expression through a peroxisome-proliferator-activated receptor alpha (PPARalpha)-independent pathway

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10.1016/J.YEXMP.2011.07.003

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9 February 2020

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