(Q24291932)

GOA release 2020-03-11

1 reference

S C Masters

1

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H Yang

2

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S R Datta

3

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M E Greenberg

4

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H Fu

Cdi1, a human G1 and S phase protein phosphatase that associates with Cdk2

5

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language of work or name

English

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publication date

December 2001

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published in

Molecular Pharmacology

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volume

60

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page(s)

1325-31

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issue

6

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cites work

1 reference

https://api.crossref.org/works/10.1124%2FMOL.60.6.1325

7 January 2021

Transient transfection assay of cell death genes

1 reference

https://api.crossref.org/works/10.1124%2FMOL.60.6.1325

7 January 2021

Akt phosphorylation of BAD couples survival signals to the cell-intrinsic death machinery

1 reference

https://api.crossref.org/works/10.1124%2FMOL.60.6.1325

7 January 2021

The structural basis for 14-3-3:phosphopeptide binding specificity

1 reference

https://api.crossref.org/works/10.1124%2FMOL.60.6.1325

7 January 2021

Akt promotes cell survival by phosphorylating and inhibiting a Forkhead transcription factor

1 reference

https://api.crossref.org/works/10.1124%2FMOL.60.6.1325

7 January 2021

Interaction of 14-3-3 with signaling proteins is mediated by the recognition of phosphoserine

1 reference

https://api.crossref.org/works/10.1124%2FMOL.60.6.1325

7 January 2021

Serine phosphorylation of death agonist BAD in response to survival factor results in binding to 14-3-3 not BCL-X(L)

1 reference

https://api.crossref.org/works/10.1124%2FMOL.60.6.1325

7 January 2021

Rsk1 mediates a MEK-MAP kinase cell survival signal

1 reference

https://api.crossref.org/works/10.1124%2FMOL.60.6.1325

7 January 2021

Phosphorylation of the pro-apoptotic protein BAD on serine 155, a novel site, contributes to cell survival

1 reference

https://api.crossref.org/works/10.1124%2FMOL.60.6.1325

7 January 2021

The kit receptor promotes cell survival via activation of PI 3-kinase and subsequent Akt-mediated phosphorylation of Bad on Ser136

1 reference

https://api.crossref.org/works/10.1124%2FMOL.60.6.1325

7 January 2021

14-3-3 proteins and survival kinases cooperate to inactivate BAD by BH3 domain phosphorylation

1 reference

https://api.crossref.org/works/10.1124%2FMOL.60.6.1325

7 January 2021

Phosphorylation and inactivation of BAD by mitochondria-anchored protein kinase A

1 reference

https://api.crossref.org/works/10.1124%2FMOL.60.6.1325

7 January 2021

Isolation of high-affinity peptide antagonists of 14-3-3 proteins by phage display

1 reference

https://api.crossref.org/works/10.1124%2FMOL.60.6.1325

7 January 2021

Regulation of BAD by cAMP-dependent protein kinase is mediated via phosphorylation of a novel site, Ser155

1 reference

https://api.crossref.org/works/10.1124%2FMOL.60.6.1325

7 January 2021

The eukaryotic host factor that activates exoenzyme S of Pseudomonas aeruginosa is a member of the 14-3-3 protein family

1 reference

https://api.crossref.org/works/10.1124%2FMOL.60.6.1325

7 January 2021

Suppression of apoptosis signal-regulating kinase 1-induced cell death by 14-3-3 proteins

1 reference

https://api.crossref.org/works/10.1124%2FMOL.60.6.1325

7 January 2021

14-3-3 proteins associate with A20 in an isoform-specific manner and function both as chaperone and adapter molecules

1 reference

https://api.crossref.org/works/10.1124%2FMOL.60.6.1325

7 January 2021

Raf-1 kinase and exoenzyme S interact with 14-3-3zeta through a common site involving lysine 49.

1 reference

https://api.crossref.org/works/10.1124%2FMOL.60.6.1325

7 January 2021

Mutations in the hydrophobic surface of an amphipathic groove of 14-3-3zeta disrupt its interaction with Raf-1 kinase.

1 reference

https://api.crossref.org/works/10.1124%2FMOL.60.6.1325

7 January 2021

14-3-3zeta binds a phosphorylated Raf peptide and an unphosphorylated peptide via its conserved amphipathic groove

1 reference

https://api.crossref.org/works/10.1124%2FMOL.60.6.1325

7 January 2021

p90(RSK) blocks bad-mediated cell death via a protein kinase C-dependent pathway

1 reference

https://api.crossref.org/works/10.1124%2FMOL.60.6.1325

7 January 2021

The Akt proto-oncogene links Ras to Pak and cell survival signals

1 reference

https://api.crossref.org/works/10.1124%2FMOL.60.6.1325

7 January 2021

Growth factors inactivate the cell death promoter BAD by phosphorylation of its BH3 domain on Ser155.

1 reference

https://api.crossref.org/works/10.1124%2FMOL.60.6.1325

7 January 2021

BAD Ser-155 phosphorylation regulates BAD/Bcl-XL interaction and cell survival

1 reference

https://api.crossref.org/works/10.1124%2FMOL.60.6.1325

7 January 2021

14-3-3 proteins block apoptosis and differentially regulate MAPK cascades

1 reference

https://api.crossref.org/works/10.1124%2FMOL.60.6.1325

7 January 2021

Interleukin-3-induced phosphorylation of BAD through the protein kinase Akt

1 reference

https://api.crossref.org/works/10.1124%2FMOL.60.6.1325

7 January 2021

Cell survival promoted by the Ras-MAPK signaling pathway by transcription-dependent and -independent mechanisms

1 reference

https://api.crossref.org/works/10.1124%2FMOL.60.6.1325

7 January 2021

p21-activated kinase 1 phosphorylates the death agonist bad and protects cells from apoptosis

1 reference

https://api.crossref.org/works/10.1124%2FMOL.60.6.1325

7 January 2021

High-efficiency transformation of mammalian cells by plasmid DNA

1 reference

https://api.crossref.org/works/10.1124%2FMOL.60.6.1325

7 January 2021

14-3-3 proteins: structure, function, and regulation

1 reference

https://api.crossref.org/works/10.1124%2FMOL.60.6.1325

7 January 2021

Interference of BAD (Bcl-xL/Bcl-2-Associated Death Promoter)-Induced Apoptosis in Mammalian Cells by 14-3-3 Isoforms and P11

1 reference

https://api.crossref.org/works/10.1124%2FMOL.60.6.1325

7 January 2021