(Q24296662)

DNA-binding transcription factor activity

GOA release 2020-03-11

1 reference

GOA release 2020-03-11

author

Damian Yap

5

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author name string

V Fogal

1

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N N Kartasheva

2

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G Trigiante

3

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S Llanos

4

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K H Vousden

6

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X Lu

Cell Death & Differentiation

7

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language of work or name

English

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publication date

April 2005

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published in

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volume

12

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issue

4

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page(s)

369-76

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cites work

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Characterization of structural p53 mutants which show selective defects in apoptosis but not cell cycle arrest

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Transcriptional activation by p53 correlates with suppression of growth but not transformation.

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p53: puzzle and paradigm

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Key roles for E2F1 in signaling p53-dependent apoptosis and in cell division within developing tumors

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Deregulated transcription factor E2F-1 expression leads to S-phase entry and p53-mediated apoptosis

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p53 and E2F-1 cooperate to mediate apoptosis

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p14ARF links the tumour suppressors RB and p53.

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Nucleolar Arf sequesters Mdm2 and activates p53

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p19(ARF) is dispensable for oncogenic stress-induced p53-mediated apoptosis and tumor suppression in vivo

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ARF is not required for apoptosis in Rb mutant mouse embryos

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ASPP proteins specifically stimulate the apoptotic function of p53

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iASPP oncoprotein is a key inhibitor of p53 conserved from worm to human.

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ASPP1 and ASPP2: common activators of p53 family members

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Gene expression changes in response to E2F1 activation.

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E2F-1 potentiates cell death by blocking antiapoptotic signaling pathways

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A common E2F-1 and p73 pathway mediates cell death induced by TCR activation.

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CDC25A phosphatase is a target of E2F and is required for efficient E2F-induced S phase

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Apaf-1 is a transcriptional target for E2F and p53

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Apaf-1 is a mediator of E2F-1-induced apoptosis

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Up-regulation of Bcl-2 homology 3 (BH3)-only proteins by E2F1 mediates apoptosis

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p53-dependent apoptosis modulates the cytotoxicity of anticancer agents

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Kinetics of p53 binding to promoter sites in vivo

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