(Q24302352)

membrane depolarization during atrial cardiac muscle cell action potential

GOA release 2020-03-11

1 reference

GOA release 2020-03-11

1 reference

6

Hans R Figulla

Hans R

Figulla

1 reference

9 December 2023

Ralf Surber

1

Ralf

Surber

1 reference

9 December 2023

Sabine Hensellek

2

Sabine

Hensellek

1 reference

9 December 2023

Dirk Prochnau

3

Dirk

Prochnau

1 reference

9 December 2023

Gerald S Werner

4

Gerald S

Werner

1 reference

9 December 2023

Klaus Benndorf

5

Klaus

Benndorf

1 reference

9 December 2023

Thomas Zimmer

7

Thomas

Zimmer

1 reference

Genomic organization of the human SCN5A gene encoding the cardiac sodium channel

9 December 2023

language of work or name

English

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publication date

1 March 2008

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published in

Cardiovascular Research

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volume

77

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issue

4

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page(s)

740-8

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cites work

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVM096

7 January 2021

The b1 Subunit but not the b2 Subunit Colocalizes with the Human Heart Na+ Channel (hH1) already within the Endoplasmic Reticulum

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVM096

7 January 2021

Sea anemone toxin (ATX II) modulation of heart and skeletal muscle sodium channel alpha-subunits expressed in tsA201 cells

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVM096

7 January 2021

SCN5A mutations associated with an inherited cardiac arrhythmia, long QT syndrome

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVM096

7 January 2021

A novel SCN5A mutation manifests as a malignant form of long QT syndrome with perinatal onset of tachycardia/bradycardia

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVM096

7 January 2021

Spectrum and prevalence of cardiac sodium channel variants among black, white, Asian, and Hispanic individuals: implications for arrhythmogenic susceptibility and Brugada/long QT syndrome genetic testing

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVM096

7 January 2021

Site-3 toxins and cardiac sodium channels

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVM096

7 January 2021

A mutation in the human cardiac sodium channel (E161K) contributes to sick sinus syndrome, conduction disease and Brugada syndrome in two families

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVM096

7 January 2021

Molecular biology of sodium channels and their role in cardiac arrhythmias

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVM096

7 January 2021

A novel SCN5A arrhythmia mutation, M1766L, with expression defect rescued by mexiletine

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVM096

7 January 2021

A de novo missense mutation of human cardiac Na+ channel exhibiting novel molecular mechanisms of long QT syndrome.

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVM096

7 January 2021

The ajmaline challenge in Brugada syndrome: diagnostic impact, safety, and recommended protocol

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVM096

7 January 2021

Cardiac conduction defects associate with mutations in SCN5A

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVM096

7 January 2021

Genetic basis and molecular mechanism for idiopathic ventricular fibrillation

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVM096

7 January 2021

A sodium-channel mutation causes isolated cardiac conduction disease

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVM096

7 January 2021

Molecular mechanism for an inherited cardiac arrhythmia

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVM096

7 January 2021

Molecular determinants of high affinity binding of alpha-scorpion toxin and sea anemone toxin in the S3-S4 extracellular loop in domain IV of the Na+ channel alpha subunit

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVM096

7 January 2021

Inherited Brugada and long QT-3 syndrome mutations of a single residue of the cardiac sodium channel confer distinct channel and clinical phenotypes

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVM096

7 January 2021

Voltage-dependent open-state inactivation of cardiac sodium channels: gating current studies with Anthopleurin-A toxin

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVM096

7 January 2021

Tracking voltage-dependent conformational changes in skeletal muscle sodium channel during activation.

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVM096

7 January 2021

Further characterization of the syndrome of right bundle branch block, ST segment elevation, and sudden cardiac death

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVM096

7 January 2021

Reduced voltage dependence of inactivation in the SCN5A sodium channel mutation delF1617.

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVM096

7 January 2021

A novel SCN5A mutation associated with long QT-3: altered inactivation kinetics and channel dysfunction.

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVM096

7 January 2021

Images in cardiovascular medicine. Life-threatening neonatal arrhythmia: successful treatment and confirmation of clinically suspected extreme long QT-syndrome-3.

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVM096

7 January 2021

SCN5A mutation associated with dilated cardiomyopathy, conduction disorder, and arrhythmia

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVM096

7 January 2021

Arrhythmogenic mechanism of an LQT-3 mutation of the human heart Na(+) channel alpha-subunit: A computational analysis

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVM096

7 January 2021

Right bundle-branch block and ST-segment elevation in leads V1 through V3: a marker for sudden death in patients without demonstrable structural heart disease

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVM096

7 January 2021

Phenotypic characterization of a novel long-QT syndrome mutation (R1623Q) in the cardiac sodium channel.

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVM096

7 January 2021

A single Na(+) channel mutation causing both long-QT and Brugada syndromes

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVM096

7 January 2021

Two distinct congenital arrhythmias evoked by a multidysfunctional Na(+) channel

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVM096

7 January 2021

Clinical, genetic, and biophysical characterization of SCN5A mutations associated with atrioventricular conduction block

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVM096

7 January 2021

De novo mutation in the SCN5A gene associated with early onset of sudden infant death

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVM096

7 January 2021

Novel SCN5A mutation leading either to isolated cardiac conduction defect or Brugada syndrome in a large French family

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVM096

7 January 2021

Novel arrhythmogenic mechanism revealed by a long-QT syndrome mutation in the cardiac Na(+) channel.

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVM096

7 January 2021

Homozygous SCN5A mutation in long-QT syndrome with functional two-to-one atrioventricular block

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVM096

7 January 2021

Long QT syndrome, Brugada syndrome, and conduction system disease are linked to a single sodium channel mutation

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVM096

7 January 2021

The Brugada syndrome--an update

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVM096

7 January 2021

release_2iipigcknrbubnbtj425r534bm

Identifiers

DOI

10.1093/CVR/CVM096

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Fatcat ID

0 references