(Q24305971)

inferred from title

superoxide dismutase 1

1 reference

GOA release 2020-03-11

Valeria Crippa

8

0 references

Paola Rusmini

Paola Rusmini

11

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Patrizia Riso

Patrizia Riso

4

0 references

Marisa Porrini

Marisa Porrini

6

0 references

Caterina Bendotti

Caterina Bendotti

13

0 references

Angelo Poletti

Angelo Poletti

14

0 references

author name string

Daniela Sau

1

0 references

Silvia De Biasi

2

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Laura Vitellaro-Zuccarello

3

0 references

Serena Guarnieri

5

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Silvia Simeoni

7

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Elisa Onesto

9

0 references

Isabella Palazzolo

10

0 references

Elena Bolzoni

Lessons from models of SOD1-linked familial ALS.

12

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language of work or name

English

0 references

publication date

1 July 2007

0 references

published in

Human Molecular Genetics

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volume

16

0 references

issue

13

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page(s)

1604-18

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cites work

1 reference

https://api.crossref.org/works/10.1093%2FHMG%2FDDM110

ALS: a disease of motor neurons and their nonneuronal neighbors

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FHMG%2FDDM110

Molecular biology of amyotrophic lateral sclerosis: insights from genetics

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FHMG%2FDDM110

Mutations in Cu/Zn superoxide dismutase gene are associated with familial amyotrophic lateral sclerosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FHMG%2FDDM110

Motor neuron degeneration in mice that express a human Cu,Zn superoxide dismutase mutation

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FHMG%2FDDM110

Copper-zinc superoxide dismutase and amyotrophic lateral sclerosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FHMG%2FDDM110

Amyotrophic lateral sclerosis with neuronal intranuclear protein inclusions

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FHMG%2FDDM110

Increased oxidative damage to DNA in an animal model of amyotrophic lateral sclerosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FHMG%2FDDM110

Oxidative stress in ALS: a mechanism of neurodegeneration and a therapeutic target

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FHMG%2FDDM110

Early vacuolization and mitochondrial damage in motor neurons of FALS mice are not associated with apoptosis or with changes in cytochrome oxidase histochemical reactivity.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FHMG%2FDDM110

An adverse property of a familial ALS-linked SOD1 mutation causes motor neuron disease characterized by vacuolar degeneration of mitochondria

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FHMG%2FDDM110

Neuroblastoma x spinal cord (NSC) hybrid cell lines resemble developing motor neurons

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FHMG%2FDDM110

Androgen-induced neurite outgrowth is mediated by neuritin in motor neurones.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FHMG%2FDDM110

Motoneuronal cell death is not correlated with aggregate formation of androgen receptors containing an elongated polyglutamine tract

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FHMG%2FDDM110

Aggregation and proteasome: the case of elongated polyglutamine aggregation in spinal and bulbar muscular atrophy.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FHMG%2FDDM110

Androgen receptor gene mutations in X-linked spinal and bulbar muscular atrophy

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FHMG%2FDDM110

The polyglutamine tract of androgen receptor: from functions to dysfunctions in motor neurons.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FHMG%2FDDM110

Membrane filter assay for detection of amyloid-like polyglutamine-containing protein aggregates.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FHMG%2FDDM110

Impairment of the ubiquitin-proteasome system by protein aggregation

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FHMG%2FDDM110

Global impairment of the ubiquitin-proteasome system by nuclear or cytoplasmic protein aggregates precedes inclusion body formation.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FHMG%2FDDM110

Conversion to the amyotrophic lateral sclerosis phenotype is associated with intermolecular linked insoluble aggregates of SOD1 in mitochondria

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FHMG%2FDDM110

Radio-sensitivity of the cells from amyotrophic lateral sclerosis model mice transfected with human mutant SOD1.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FHMG%2FDDM110

Motor neurons in Cu/Zn superoxide dismutase-deficient mice develop normally but exhibit enhanced cell death after axonal injury

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FHMG%2FDDM110

Superoxide-mediated cytotoxicity in superoxide dismutase-deficient fetal fibroblasts

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FHMG%2FDDM110

Transgenic mouse model for familial amyotrophic lateral sclerosis with superoxide dismutase-1 mutation

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FHMG%2FDDM110

Mutation of superoxide dismutase elevates reactive species: comparison of nitration and oxidation of proteins in different brain regions of transgenic mice with amyotrophic lateral sclerosis.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FHMG%2FDDM110

Aggregation and Motor Neuron Toxicity of an ALS-Linked SOD1 Mutant Independent from Wild-Type SOD1

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FHMG%2FDDM110

Alterations in G(1) to S phase cell-cycle regulators during amyotrophic lateral sclerosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FHMG%2FDDM110

Histological evidence of redox system breakdown caused by superoxide dismutase 1 (SOD1) aggregation is common to SOD1-mutated motor neurons in humans and animal models

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FHMG%2FDDM110

Identification of a signal for rapid export of proteins from the nucleus

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FHMG%2FDDM110

Nuclear import can be separated into distinct steps in vitro: nuclear pore binding and translocation

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FHMG%2FDDM110

Alteration of familial ALS-linked mutant SOD1 solubility with disease progression: Its modulation by the proteasome and Hsp70

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FHMG%2FDDM110

Sequence and structural determinants of Cu, Zn superoxide dismutase aggregation

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FHMG%2FDDM110

Increased oxidative damage to DNA in ALS patients

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FHMG%2FDDM110

7 January 2021

Superoxide dismutase concentration and activity in familial amyotrophic lateral sclerosis

1 reference

https://api.crossref.org/works/10.1093%2FHMG%2FDDM110

7 January 2021

Mitochondrial localization of mutant superoxide dismutase 1 triggers caspase-dependent cell death in a cellular model of familial amyotrophic lateral sclerosis

1 reference

https://api.crossref.org/works/10.1093%2FHMG%2FDDM110

7 January 2021

Does tomato consumption effectively increase the resistance of lymphocyte DNA to oxidative damage?

1 reference

https://api.crossref.org/works/10.1093%2FHMG%2FDDM110

7 January 2021

Androgen receptor with elongated polyglutamine tract forms aggregates that alter axonal trafficking and mitochondrial distribution in motor neuronal processes

1 reference

https://api.crossref.org/works/10.1093%2FHMG%2FDDM110

7 January 2021

Reduction of CuZn-superoxide dismutase activity exacerbates neuronal cell injury and edema formation after transient focal cerebral ischemia

1 reference

https://api.crossref.org/works/10.1093%2FHMG%2FDDM110

7 January 2021

Massive mitochondrial degeneration in motor neurons triggers the onset of amyotrophic lateral sclerosis in mice expressing a mutant SOD1

1 reference

https://api.crossref.org/works/10.1093%2FHMG%2FDDM110

7 January 2021