(Q24307612)

negative regulation of apoptotic process

GOA release 2020-03-11

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Journal of Biological Chemistry

GOA release 2020-03-11

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James F Curtin

1

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Thomas G Cotter

series ordinal

2

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language of work or name

English

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publication date

23 April 2004

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published in

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volume

279

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issue

17

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page(s)

17090-100

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cites work

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Molecular ordering of the initial signaling events of CD95.

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Fas-mediated apoptosis in seven human prostate cancer cell lines: correlation with tumor stage

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Constitutive JNK activation in NIH 3T3 fibroblasts induces a partially transformed phenotype

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Raf-independent deregulation of p38 and JNK mitogen-activated protein kinases are critical for Ras transformation

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The Bcr-Abl leukemia oncogene activates Jun kinase and requires Jun for transformation

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Anisomycin activates JNK and sensitises DU 145 prostate carcinoma cells to Fas mediated apoptosis.

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A novel assay for apoptosis. Flow cytometric detection of phosphatidylserine expression on early apoptotic cells using fluorescein labelled Annexin V

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JC-1, but not DiOC6(3) or rhodamine 123, is a reliable fluorescent probe to assess delta psi changes in intact cells: implications for studies on mitochondrial functionality during apoptosis

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The Jun kinase/stress-activated protein kinase pathway functions to regulate DNA repair and inhibition of the pathway sensitizes tumor cells to cisplatin

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Differential gene regulation by specific gain-of-function JNK1 proteins expressed in Swiss 3T3 fibroblasts

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Protein kinase C isoenzyme patterns characteristically modulated in early prostate cancer

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Identification and sequence of human PKY, a putative kinase with increased expression in multidrug-resistant cells, with homology to yeast protein kinase Yak1

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Identification of a 170 kDa membrane kinase with increased activity in KB‐V1 multidrug resistant cells

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Homeodomain-interacting protein kinase 1 modulates Daxx localization, phosphorylation, and transcriptional activity

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The serine/threonine kinase HIPK2 interacts with TRADD, but not with CD95 or TNF-R1 in 293T cells.

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Sensitization to death receptor cytotoxicity by inhibition of fas-associated death domain protein (FADD)/caspase signaling. Requirement of cell cycle progression

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Hyperosmolarity and CD95L trigger CD95/EGF receptor association and tyrosine phosphorylation of CD95 as prerequisites for CD95 membrane trafficking and DISC formation

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Immune privilege or inflammation? Insights into the Fas ligand enigma

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The PEA3 Ets transcription factor is a downstream target of the HER2/Neu receptor tyrosine kinase

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Identifiers

DOI

10.1074/JBC.M307629200

1 reference

Consolidated OpenCitations Corpus – April 2017

1 reference

Consolidated OpenCitations Corpus – April 2017

PubMed publication ID

14766760

1 reference

Consolidated OpenCitations Corpus – April 2017