(Q24676207)

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Aurora B expression correlates with aggressive behaviour in glioblastoma multiforme

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Aurora B expression correlates with aggressive behaviour in glioblastoma multiforme (English)

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Weifen F Zeng

1

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Kapila Navaratne

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2

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Richard A Prayson

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3

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Robert J Weil

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4

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language of work or name

English

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publication date

February 2007

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published in

Journal of Clinical Pathology

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volume

60

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issue

2

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page(s)

218-21

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cites work

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Aurora B overexpression associates with the thyroid carcinoma undifferentiated phenotype and is required for thyroid carcinoma cell proliferation

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Aurora kinases, aneuploidy and cancer, a coincidence or a real link?

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Aurora-B dysfunction of multinucleated giant cells in glioma detected by site-specific phosphorylated antibodies.

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High expression of Aurora-B/Aurora and Ipll-like midbody-associated protein (AIM-1) in astrocytomas

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VX-680, a potent and selective small-molecule inhibitor of the Aurora kinases, suppresses tumor growth in vivo

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Aurora B expression in normal testis and seminomas.

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Genetic aberrations defined by comparative genomic hybridization distinguish long-term from typical survivors of glioblastoma.

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Constitutional von Hippel-Lindau (VHL) gene deletions detected in VHL families by fluorescence in situ hybridization

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Identifiers

DOI

10.1136/JCP.2006.036806

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Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

241608

OpenCitations bibliographic resource ID

241608

1 reference

stated in

Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

1 reference

Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

241608

PubMed publication ID

17264249

1 reference

stated in

Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

241608

ResearchGate publication ID

6540806

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(Q24676207)

Aurora B expression correlates with aggressive behaviour in glioblastoma multiforme

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