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cAMP/PKA signaling balances respiratory activity with mitochondria dependent apoptosis via transcriptional regulation
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scholarly article
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PubMed
PubMed ID
21108829
retrieved
1 December 2016
title
cAMP/PKA signaling balances respiratory activity with mitochondria dependent apoptosis via transcriptional regulation
(English)
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PubMed
PubMed ID
21108829
retrieved
1 December 2016
main subject
cell biology
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apoptotic process
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Transcription factor HAP4 YKL109W
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GOA release 2020-03-11
mitochondrion
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inferred from title
author name string
Jane E Leadsham
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1
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Campbell W Gourlay
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2
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language of work or name
English
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publication date
25 November 2010
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published in
BMC Cell Biology
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PubMed
PubMed ID
21108829
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1 December 2016
volume
11
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page(s)
92
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issue
1
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copyright license
Creative Commons Attribution 2.0 Generic
start time
25 November 2010
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April 2022 Public Data File from Crossref
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copyrighted
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Europe PubMed Central
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22 June 2022
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https://europepmc.org/article/PMC/3001716
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CZ Software Mentions: A large dataset of software mentions in the biomedical literature
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https://scigraph.springernature.com/pub.10.1186/1471-2121-11-92
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Saccharomyces cerevisiae cAMP-dependent protein kinase controls entry into stationary phase through the Rim15p protein kinase
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SOK2 may regulate cyclic AMP-dependent protein kinase-stimulated growth and pseudohyphal development by repressing transcription
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The pde2 gene of Saccharomyces cerevisiae is allelic to rca1 and encodes a phosphodiesterase which protects the cell from extracellular cAMP.
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Cloning and characterization of BCY1, a locus encoding a regulatory subunit of the cyclic AMP-dependent protein kinase in Saccharomyces cerevisiae
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Three different genes in S. cerevisiae encode the catalytic subunits of the cAMP-dependent protein kinase
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Compromised respiratory adaptation and thermoregulation in aging and age-related diseases
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The role of mitochondria in apoptosis*.
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Cellular senescence: unravelling complexity
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Transcriptional control of mitochondrial biogenesis and function
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Gene expression signatures of cAMP/protein kinase A (PKA)-promoted, mitochondrial-dependent apoptosis. Comparative analysis of wild-type and cAMP-deathless S49 lymphoma cells
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The yeast A kinases differentially regulate iron uptake and respiratory function.
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Apoptosis induced by cAMP requires Smac/DIABLO transcriptional upregulation
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The Saccharomyces cerevisiae Sko1p transcription factor mediates HOG pathway-dependent osmotic regulation of a set of genes encoding enzymes implicated in protection from oxidative damage
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Cyclic AMP produced inside mitochondria regulates oxidative phosphorylation
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Elevated Ras/protein kinase A activity in Saccharomyces cerevisiae reduces proliferation rate and lifespan by two different reactive oxygen species-dependent routes.
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27 November 2018
The yeast cAMP protein kinase Tpk3p is involved in the regulation of mitochondrial enzymatic content during growth.
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27 November 2018
Sok2p transcription factor is involved in adaptive program relevant for long term survival of Saccharomyces cerevisiae colonies.
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27 November 2018
Overexpression of HAP4 in glucose-derepressed yeast cells reveals respiratory control of glucose-regulated genes
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27 November 2018
The yeast mitochondrial proteome, a study of fermentative and respiratory growth
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27 November 2018
The modulation of the biological activities of mitochondrial histone Abf2p by yeast PKA and its possible role in the regulation of mitochondrial DNA content during glucose repression
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27 November 2018
Mitochondria-targeted green fluorescent proteins: convenient tools for the study of organelle biogenesis in Saccharomyces cerevisiae.
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27 November 2018
Low activity of the yeast cAMP-dependent protein kinase catalytic subunit Tpk3 is due to the poor expression of the TPK3 gene
1 reference
stated in
PubMed
reference URL
https://pubmed.ncbi.nlm.nih.gov/21108829
retrieved
12 December 2020
based on heuristic
inferred from PubMed ID database lookup
Interactions between adenylyl cyclase, CAP and RAS from Saccharomyces cerevisiae
1 reference
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PubMed
reference URL
https://pubmed.ncbi.nlm.nih.gov/21108829
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12 December 2020
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inferred from PubMed ID database lookup
Mdm38 protein depletion causes loss of mitochondrial K+/H+ exchange activity, osmotic swelling and mitophagy
1 reference
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PubMed
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12 December 2020
based on heuristic
inferred from PubMed ID database lookup
Identifiers
DOI
10.1186/1471-2121-11-92
1 reference
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Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
3837986
Dimensions Publication ID
1002174402
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OpenCitations bibliographic resource ID
3837986
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
3837986
PMCID
3001716
0 references
PubMed ID
21108829
1 reference
stated in
PubMed
PubMed ID
21108829
retrieved
1 December 2016
ResearchGate publication ID
49636892
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