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The Next Immune-Checkpoint Inhibitors: PD-1/PD-L1 Blockade in Melanoma
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PubMed
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title
The Next Immune-Checkpoint Inhibitors: PD-1/PD-L1 Blockade in Melanoma
(English)
1 reference
stated in
PubMed
author
Gordon J. Freeman
series ordinal
2
object named as
Gordon J. Freeman
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stated in
Crossref
author name string
Kathleen M. Mahoney
series ordinal
1
1 reference
stated in
Crossref
David F. McDermott
series ordinal
3
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stated in
Crossref
language of work or name
English
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stated in
PubMed
publication date
1 April 2015
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stated in
PubMed
published in
Clinical Therapeutics
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stated in
PubMed
volume
37
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stated in
PubMed
issue
4
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stated in
PubMed
page(s)
764-82
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PubMed
cites work
Immune Checkpoint Blockade in Cancer Therapy
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stated in
PubMed Central
reference URL
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Tuning of antigen sensitivity by T cell receptor-dependent negative feedback controls T cell effector function in inflamed tissues
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Fc-dependent depletion of tumor-infiltrating regulatory T cells co-defines the efficacy of anti-CTLA-4 therapy against melanoma
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PubMed Central
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Nivolumab plus ipilimumab in advanced melanoma
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PubMed Central
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20 March 2017
Safety and Tumor Responses with Lambrolizumab (Anti–PD-1) in Melanoma
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PubMed Central
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20 March 2017
Safety and Activity of Anti–PD-L1 Antibody in Patients with Advanced Cancer
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Safety, activity, and immune correlates of anti-PD-1 antibody in cancer
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Intratumor Heterogeneity and Branched Evolution Revealed by Multiregion Sequencing
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Improved Survival with Ipilimumab in Patients with Metastatic Melanoma
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CTLA-4 and PD-1 receptors inhibit T-cell activation by distinct mechanisms
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Costimulatory B7-H1 in renal cell carcinoma patients: Indicator of tumor aggressiveness and potential therapeutic target
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Blockade of programmed death-1 ligands on dendritic cells enhances T cell activation and cytokine production
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Engagement of the PD-1 immunoinhibitory receptor by a novel B7 family member leads to negative regulation of lymphocyte activation
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Induced expression of PD-1, a novel member of the immunoglobulin gene superfamily, upon programmed cell death
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PD-1 and its ligands in tolerance and immunity
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Differential binding properties of B7-H1 and B7-DC to programmed death-1
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PD-L2 is a second ligand for PD-1 and inhibits T cell activation
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Development of lupus-like autoimmune diseases by disruption of the PD-1 gene encoding an ITIM motif-carrying immunoreceptor
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Guidelines for the evaluation of immune therapy activity in solid tumors: immune-related response criteria
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Alternative splice variants of the human PD-1 gene
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Predictive correlates of response to the anti-PD-L1 antibody MPDL3280A in cancer patients
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MPDL3280A (anti-PD-L1) treatment leads to clinical activity in metastatic bladder cancer
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Survival, durable tumor remission, and long-term safety in patients with advanced melanoma receiving nivolumab
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Colocalization of inflammatory response with B7-h1 expression in human melanocytic lesions supports an adaptive resistance mechanism of immune escape
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Phase I study of single-agent anti-programmed death-1 (MDX-1106) in refractory solid tumors: safety, clinical activity, pharmacodynamics, and immunologic correlates
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PD-1 and CTLA-4 combination blockade expands infiltrating T cells and reduces regulatory T and myeloid cells within B16 melanoma tumors
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Pooled Analysis of Long-Term Survival Data From Phase II and Phase III Trials of Ipilimumab in Unresectable or Metastatic Melanoma
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Integrative analysis reveals selective 9p24.1 amplification, increased PD-1 ligand expression, and further induction via JAK2 in nodular sclerosing Hodgkin lymphoma and primary mediastinal large B-cell lymphoma.
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Identifiers
DOI
10.1016/J.CLINTHERA.2015.02.018
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Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
1458568
OpenCitations bibliographic resource ID
1458568
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stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
1458568
PMCID
4497957
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
1458568
PubMed ID
25823918
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
1458568
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