(Q28202364)

4

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Jean-François Tanti

object named as

Jean-François Tanti

6

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author name string

Teresa Gonzalez

2

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Thierry Grémeaux

3

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Yannick Le Marchand-Brustel

Journal of Biological Chemistry

5

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language of work or name

English

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publication date

18 July 2003

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published in

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volume

278

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page(s)

26550-7

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issue

29

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cites work

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APS facilitates c-Cbl tyrosine phosphorylation and GLUT4 translocation in response to insulin in 3T3-L1 adipocytes

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Osmotic shock stimulates GLUT4 translocation in 3T3L1 adipocytes by a novel tyrosine kinase pathway

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The osmotic shock-induced glucose transport pathway in 3T3-L1 adipocytes is mediated by gab-1 and requires Gab-1-associated phosphatidylinositol 3-kinase activity for full activation

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A Crk-II/TC10 signaling pathway is required for osmotic shock-stimulated glucose transport

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Detrimental effect of hyperosmolality on insulin-stimulated glucose metabolism in adipose and muscle tissue in vitro

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Osmotic shock inhibits insulin signaling by maintaining Akt/protein kinase B in an inactive dephosphorylated state

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Insulin resistance and a diabetes mellitus-like syndrome in mice lacking the protein kinase Akt2 (PKB beta)

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Requirement for activation of the serine-threonine kinase Akt (protein kinase B) in insulin stimulation of protein synthesis but not of glucose transport

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Phosphorylation of Ser307 in insulin receptor substrate-1 blocks interactions with the insulin receptor and inhibits insulin action

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Defect in skeletal muscle phosphatidylinositol-3-kinase in obese insulin-resistant mice

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Activated phosphatidylinositol 3-kinase is sufficient to mediate actin rearrangement and GLUT4 translocation in 3T3-L1 adipocytes

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Phosphatidylinositol 3-kinase is necessary and sufficient for insulin-stimulated stress fiber breakdown

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The small guanosine triphosphate-binding protein Rab4 is involved in insulin-induced GLUT4 translocation and actin filament rearrangement in 3T3-L1 cells

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Mammalian target of rapamycin pathway regulates insulin signaling via subcellular redistribution of insulin receptor substrate 1 and integrates nutritional signals and metabolic signals of insulin

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Serine/threonine phosphorylation of IRS-1 triggers its degradation: possible regulation by tyrosine phosphorylation

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Osmotic stress regulates the stability of cyclin D1 in a p38SAPK2-dependent manner

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Rapamycin partially prevents insulin resistance induced by chronic insulin treatment

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FKBP12-rapamycin-associated protein associates with mitochondria and senses osmotic stress via mitochondrial dysfunction

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