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GDF11 does not rescue aging-related pathological hypertrophy
scientific article
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scholarly article
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title
GDF11 does not rescue aging-related pathological hypertrophy
(English)
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author
Maria Myzithras
object named as
Maria Myzithras
series ordinal
10
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Xiongwen Chen
object named as
Xiongwen Chen
series ordinal
17
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Steven R Houser
series ordinal
19
object named as
Steven R Houser
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author name string
Shavonn C Smith
series ordinal
1
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Xiaoxiao Zhang
series ordinal
2
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Xiaoying Zhang
series ordinal
3
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Polina Gross
series ordinal
4
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Timothy Starosta
series ordinal
5
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Sadia Mohsin
series ordinal
6
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Michael Franti
series ordinal
7
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Priyanka Gupta
series ordinal
8
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David Hayes
series ordinal
9
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Julius Kahn
series ordinal
11
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James Tanner
series ordinal
12
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Steven M Weldon
series ordinal
13
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Ashraf Khalil
series ordinal
14
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Xinji Guo
series ordinal
15
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Abdelkarim Sabri
series ordinal
16
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Scott MacDonnell
series ordinal
18
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language of work or name
English
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publication date
6 November 2015
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published in
Circulation Research
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volume
117
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issue
11
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page(s)
926-32
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cites work
GDF11 Increases with Age and Inhibits Skeletal Muscle Regeneration
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PubMed Central
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Vascular and neurogenic rejuvenation of the aging mouse brain by young systemic factors
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Restoring systemic GDF11 levels reverses age-related dysfunction in mouse skeletal muscle
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PubMed Central
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20 March 2017
Growth differentiation factor 11 is a circulating factor that reverses age-related cardiac hypertrophy
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PubMed Central
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20 March 2017
Genetic deletion of myostatin from the heart prevents skeletal muscle atrophy in heart failure
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A mutation in the myostatin gene increases muscle mass and enhances racing performance in heterozygote dogs
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Regulation of skeletal muscle mass in mice by a new TGF-beta superfamily member
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Rejuvenation of aged progenitor cells by exposure to a young systemic environment
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Mouse strain determines cardiac growth potential
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An inhibitor of transforming growth factor beta type I receptor ameliorates muscle atrophy in a mouse model of caveolin 3-deficient muscular dystrophy
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Rejuvenation of regeneration in the aging central nervous system
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29 September 2017
Aging-related defects are associated with adverse cardiac remodeling in a mouse model of reperfused myocardial infarction
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Transforming growth factor-beta and myostatin signaling in skeletal muscle
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Myostatin does not regulate cardiac hypertrophy or fibrosis
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Fibrosis, not cell size, delineates beta-myosin heavy chain reexpression during cardiac hypertrophy and normal aging in vivo
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Age-dependent effect of myostatin blockade on disease severity in a murine model of limb-girdle muscular dystrophy
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The old-age heart: normal aging changes which can produce or mimic cardiac disease
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Smaller cardiac cell size and reduced extra-cellular collagen might be beneficial for hearts of Ames dwarf mice
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Consequences of weight cycling in obese spontaneously hypertensive rats
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2 June 2018
Diabetic type of cardiomyopathy in food-restricted rats
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2 June 2018
GDF11 and the Mythical Fountain of Youth.
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27 November 2018
Combined loss of neuronal and endothelial nitric oxide synthase causes premature mortality and age-related hypertrophic cardiac remodeling in mice.
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27 November 2018
Analysis of myocardial response to significant weight loss in obese rats.
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27 November 2018
Model of functional cardiac aging: young adult mice with mild overexpression of serum response factor
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PubMed
reference URL
https://pubmed.ncbi.nlm.nih.gov/26383970
retrieved
12 December 2020
based on heuristic
inferred from PubMed ID database lookup
Calcineurin Expression, Activation, and Function in Cardiac Pressure-Overload Hypertrophy
1 reference
stated in
PubMed
reference URL
https://pubmed.ncbi.nlm.nih.gov/26383970
retrieved
12 December 2020
based on heuristic
inferred from PubMed ID database lookup
Identifiers
DOI
10.1161/CIRCRESAHA.115.307527
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
1117140
OpenCitations bibliographic resource ID
1117140
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
1117140
PMCID
4636963
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
1117140
PubMed ID
26383970
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
1117140
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