(Q28307859)

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HIV-1 proviral DNA excision using an evolved recombinase

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HIV-1 proviral DNA excision using an evolved recombinase (English)

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4

Frank Buchholz

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author name string

Indrani Sarkar

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1

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Ilona Hauber

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2

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Joachim Hauber

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3

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language of work or name

English

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publication date

29 June 2007

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published in

Science

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volume

316

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issue

5833

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page(s)

1912-5

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cites work

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In vitro evolution of beta-glucuronidase into a beta-galactosidase proceeds through non-specific intermediates

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Treatment with indinavir, zidovudine, and lamivudine in adults with human immunodeficiency virus infection and prior antiretroviral therapy

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AMD3100, a small molecule inhibitor of HIV-1 entry via the CXCR4 co-receptor.

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Cellular APOBEC3G restricts HIV-1 infection in resting CD4+ T cells

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Integrase inhibitors and cellular immunity suppress retroviral replication in rhesus macaques

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Antiretroviral-drug resistance among patients recently infected with HIV.

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Site-specific DNA recombinases as instruments for genomic surgery

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Mechanisms of site-specific recombination.

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Current issues in mouse genome engineering

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Alteration of Cre recombinase site specificity by substrate-linked protein evolution.

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Directed evolution of the site specificity of Cre recombinase

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Non-contact positions impose site selectivity on Cre recombinase

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Conditional control of gene expression in the mouse

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Site-specific recombination of asymmetric lox sites mediated by a heterotetrameric Cre recombinase complex

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Transmission of human immunodeficiency type 1 viruses with intersubtype recombinant long terminal repeat sequences

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Evolving strategies for enzyme engineering

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HIV-1 regulatory/accessory genes: keys to unraveling viral and host cell biology

(Q28307859)

HIV-1 proviral DNA excision using an evolved recombinase

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