(Q28544797)

English

IFI16 restricts HSV-1 replication by accumulating on the hsv-1 genome, repressing HSV-1 gene expression, and directly or indirectly modulating histone modifications

scientific article (publication date: November 2014)

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scholarly article

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IFI16 restricts HSV-1 replication by accumulating on the hsv-1 genome, repressing HSV-1 gene expression, and directly or indirectly modulating histone modifications (English)

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Herpes simplex virus type 1

1 reference

based on heuristic

inferred from title

author name string

Karen E Johnson

1

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Virginie Bottero

2

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Stephanie Flaherty

3

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Sujoy Dutta

4

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Vivek Vikram Singh

5

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Bala Chandran

6

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language of work or name

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publication date

November 2014

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10

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11

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e1004503

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copyright license

Creative Commons Attribution 4.0 International

6 November 2014

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April 2022 Public Data File from Crossref

copyright status

0 references

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Sp1 binds to promoter sequences and activates herpes simplex virus 'immediate-early' gene transcription in vitro

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4223080

3 June 2018

Interferon inhibits herpes simplex virus-specific translation: a reinvestigation

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4223080

3 June 2018

Interferon treatment inhibits onset of herpes simplex virus immediate-early transcription

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4223080

3 June 2018

The nuclear location of PML, a cellular member of the C3HC4 zinc-binding domain protein family, is rearranged during herpes simplex virus infection by the C3HC4 viral protein ICP0.

1 reference

https://api.crossref.org/works/10.1371%2FJOURNAL.PPAT.1004503

21 January 2018

Herpes simplex virus-1 infection causes the secretion of a type I interferon-antagonizing protein and inhibits signaling at or before Jak-1 activation

1 reference

https://api.crossref.org/works/10.1371%2FJOURNAL.PPAT.1004503

21 January 2018

Binding of ICP4, TATA-binding protein, and RNA polymerase II to herpes simplex virus type 1 immediate-early, early, and late promoters in virus-infected cells

1 reference

https://api.crossref.org/works/10.1371%2FJOURNAL.PPAT.1004503

21 January 2018

STING-dependent signaling.

1 reference

https://api.crossref.org/works/10.1371%2FJOURNAL.PPAT.1004503

21 January 2018

The herpes simplex virus 1 Us11 protein cooperates with suboptimal amounts of human immunodeficiency virus type 1 (HIV-1) Rev protein to rescue HIV-1 production

1 reference

https://api.crossref.org/works/10.1371%2FJOURNAL.PPAT.1004503

21 January 2018

Production and purification of lentiviral vectors

1 reference

https://api.crossref.org/works/10.1371%2FJOURNAL.PPAT.1004503

21 January 2018

Dissimilar DNA binding by p53 in normal and tumor-derived cells

1 reference

https://api.crossref.org/works/10.1371%2FJOURNAL.PPAT.1004503

21 January 2018

Herpes simplex virus type 1 suppresses the interferon signaling pathway by inhibiting phosphorylation of STATs and janus kinases during an early infection stage

1 reference

https://pubmed.ncbi.nlm.nih.gov/25375629

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

The VP16 activation domain interacts with multiple transcriptional components as determined by protein-protein cross-linking in vivo

1 reference

https://pubmed.ncbi.nlm.nih.gov/25375629

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

IFN-gamma inhibits human airway smooth muscle cell proliferation by modulating the E2F-1/Rb pathway

1 reference

https://pubmed.ncbi.nlm.nih.gov/25375629

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Identifiers

10.1371/JOURNAL.PPAT.1004503

1 reference

Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

OpenCitations bibliographic resource ID

1 reference

Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

PMC publication ID

1 reference

Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

PubMed publication ID

1 reference

Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

ResearchGate publication ID

0 references

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