(Q30502486)

English

Enhanced lysosomal activity is involved in Bax inhibitor-1-induced regulation of the endoplasmic reticulum (ER) stress response and cell death against ER stress: involvement of vacuolar H+-ATPase (V-ATPase).

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Europe PubMed Central

PMC publication ID

13 October 2017

Enhanced lysosomal activity is involved in Bax inhibitor-1-induced regulation of the endoplasmic reticulum (ER) stress response and cell death against ER stress: involvement of vacuolar H+-ATPase (V-ATPase). (English)

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Europe PubMed Central

PMC publication ID

13 October 2017

endoplasmic reticulum

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Chin-Ha Chung

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Europe PubMed Central

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13 October 2017

author name string

Geum-Hwa Lee

1

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Europe PubMed Central

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13 October 2017

Do-Sung Kim

2

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Europe PubMed Central

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13 October 2017

Hyung-Tae Kim

3

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13 October 2017

Jung-Wook Lee

4

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13 October 2017

Taeho Ahn

6

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Europe PubMed Central

PMC publication ID

13 October 2017

Jung Min Lim

7

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Europe PubMed Central

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13 October 2017

In-Ki Kim

8

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13 October 2017

Han-Jung Chae

9

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13 October 2017

Hyung-Ryong Kim

10

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13 October 2017

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publication date

17 May 2011

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Europe PubMed Central

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13 October 2017

Journal of Biological Chemistry

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Europe PubMed Central

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13 October 2017

286

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Europe PubMed Central

PMC publication ID

13 October 2017

28

1 reference

Europe PubMed Central

PMC publication ID

13 October 2017

24743-24753

1 reference

Europe PubMed Central

PMC publication ID

13 October 2017

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Lectin-like ERAD players in ER and cytosol

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1 reference

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Bax Inhibitor-1 Is a pH-dependent regulator of Ca2+ channel activity in the endoplasmic reticulum

1 reference

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Lysosomes in iron metabolism, ageing and apoptosis

1 reference

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1 reference

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1 reference

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Bax inhibitor-1 regulates endoplasmic reticulum stress-associated reactive oxygen species and heme oxygenase-1 expression

1 reference

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1 reference

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Effects of cellular cholesterol loading on macrophage foam cell lysosome acidification

1 reference

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Mediators of endoplasmic reticulum stress-induced apoptosis

1 reference

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Suppression of basal autophagy in neural cells causes neurodegenerative disease in mice

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Cytoprotective gene bi-1 is required for intrinsic protection from endoplasmic reticulum stress and ischemia-reperfusion injury

1 reference

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Role of the unfolded protein response in cell death

1 reference

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TRB3, a novel ER stress-inducible gene, is induced via ATF4-CHOP pathway and is involved in cell death

1 reference

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14 July 2018

BI-1 regulates an apoptosis pathway linked to endoplasmic reticulum stress

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3137050

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Dissection of Arabidopsis Bax inhibitor-1 suppressing Bax-, hydrogen peroxide-, and salicylic acid-induced cell death

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3137050

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Control of PERK eIF2alpha kinase activity by the endoplasmic reticulum stress-induced molecular chaperone P58IPK.

1 reference

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Selective inhibition of osteoclast vacuolar H(+)-ATPase

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1 reference

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1 reference

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1 reference

https://pubmed.ncbi.nlm.nih.gov/21586565

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[Unfolded protein response: its role in physiology and physiopathology]

1 reference

https://pubmed.ncbi.nlm.nih.gov/21586565

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Identifiers

10.1074/JBC.M110.167734

1 reference

Europe PubMed Central

PMC publication ID

13 October 2017

PMC publication ID

1 reference

Europe PubMed Central

PMC publication ID

13 October 2017

PubMed publication ID

1 reference

Europe PubMed Central

PMC publication ID

13 October 2017

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