(Q33407898)

26 July 2017

Complement activation by heme as a secondary hit for atypical hemolytic uremic syndrome (English)

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atypical hemolytic uremic syndrome

26 July 2017

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based on heuristic

inferred from title

author

Véronique Frémeaux-Bacchi

6

Veronique Fremeaux-Bacchi

1 reference

26 July 2017

Lise Halbwachs-Mecarelli

5

Lise Halbwachs-Mecarelli

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Marie Frimat

Marie Frimat

1

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Jordan D Dimitrov

Jordan D Dimitrov

3

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Lubka T. Roumenina

Lubka T Roumenina

7

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author name string

Fanny Tabarin

2

1 reference

26 July 2017

Caroline Poitou

4

1 reference

26 July 2017

Lise Halbwachs-Mecarelli

5

1 reference

26 July 2017

language of work or name

English

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publication date

21 May 2013

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26 July 2017

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26 July 2017

122

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26 July 2017

2

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26 July 2017

282-292

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Atypical hemolytic-uremic syndrome

26 July 2017

1 reference

https://api.crossref.org/works/10.1182/BLOOD-2013-03-489245

Alternative complement pathway assessment in patients with atypical HUS.

25 October 2020

1 reference

https://api.crossref.org/works/10.1182/BLOOD-2013-03-489245

A prevalent C3 mutation in aHUS patients causes a direct C3 convertase gain of function

25 October 2020

1 reference

https://api.crossref.org/works/10.1182/BLOOD-2013-03-489245

Pregnancy-associated hemolytic uremic syndrome revisited in the era of complement gene mutations

25 October 2020

1 reference

https://api.crossref.org/works/10.1182/BLOOD-2013-03-489245

Heme, heme oxygenase, and ferritin: how the vascular endothelium survives (and dies) in an iron-rich environment

25 October 2020

1 reference

https://api.crossref.org/works/10.1182/BLOOD-2013-03-489245

Free heme toxicity and its detoxification systems in human

25 October 2020

1 reference

https://api.crossref.org/works/10.1182/BLOOD-2013-03-489245

Heme degradation and vascular injury.

25 October 2020

1 reference

https://api.crossref.org/works/10.1182/BLOOD-2013-03-489245

Coagulation in the pathophysiology of hemolytic anemias

25 October 2020

1 reference

https://api.crossref.org/works/10.1182/BLOOD-2013-03-489245

The clinical sequelae of intravascular hemolysis and extracellular plasma hemoglobin: a novel mechanism of human disease

25 October 2020

1 reference

https://api.crossref.org/works/10.1182/BLOOD-2013-03-489245

Hematin promotes complement alternative pathway-mediated deposition of C3 activation fragments on human erythrocytes: potential implications for the pathogenesis of anemia in malaria

25 October 2020

1 reference

https://api.crossref.org/works/10.1182/BLOOD-2013-03-489245

Severe malarial anemia of low parasite burden in rodent models results from accelerated clearance of uninfected erythrocytes

25 October 2020

1 reference

https://api.crossref.org/works/10.1182/BLOOD-2013-03-489245

Complement activation during painful crisis in sickle cell anemia

25 October 2020

1 reference

https://api.crossref.org/works/10.1182/BLOOD-2013-03-489245

Complement activation: the missing link between ADAMTS-13 deficiency and microvascular thrombosis of thrombotic microangiopathies

25 October 2020

1 reference

https://api.crossref.org/works/10.1182/BLOOD-2013-03-489245

Hyperfunctional C3 convertase leads to complement deposition on endothelial cells and contributes to atypical hemolytic uremic syndrome

25 October 2020

1 reference

https://api.crossref.org/works/10.1182/BLOOD-2013-03-489245

Genetics and outcome of atypical hemolytic uremic syndrome: a nationwide French series comparing children and adults

25 October 2020

1 reference

https://api.crossref.org/works/10.1182/BLOOD-2013-03-489245

Mutations in complement C3 predispose to development of atypical hemolytic uremic syndrome

25 October 2020

1 reference

https://api.crossref.org/works/10.1182/BLOOD-2013-03-489245

Conditionally immortalized human glomerular endothelial cells expressing fenestrations in response to VEGF.

25 October 2020

1 reference

https://api.crossref.org/works/10.1182/BLOOD-2013-03-489245

Mutation and ADAMTS13-dependent modulation of disease severity in a mouse model for von Willebrand disease type 2B.

25 October 2020

1 reference

https://api.crossref.org/works/10.1182/BLOOD-2013-03-489245

Heme interacts with c1q and inhibits the classical complement pathway

25 October 2020

1 reference

https://api.crossref.org/works/10.1182/BLOOD-2013-03-489245

Structures of complement component C3 provide insights into the function and evolution of immunity

25 October 2020

1 reference

https://api.crossref.org/works/10.1182/BLOOD-2013-03-489245

Anti factor H autoantibodies block C-terminal recognition function of factor H in hemolytic uremic syndrome

25 October 2020

1 reference

https://api.crossref.org/works/10.1182/BLOOD-2013-03-489245

Overall neutralization of complement factor H by autoantibodies in the acute phase of the autoimmune form of atypical hemolytic uremic syndrome

25 October 2020

1 reference

https://api.crossref.org/works/10.1182/BLOOD-2013-03-489245

C3 adsorbed to a polymer surface can form an initiating alternative pathway convertase

25 October 2020

1 reference

https://api.crossref.org/works/10.1182/BLOOD-2013-03-489245

Mutations of factor H impair regulation of surface-bound C3b by three mechanisms in atypical hemolytic uremic syndrome

25 October 2020

1 reference

https://api.crossref.org/works/10.1182/BLOOD-2013-03-489245

Platelet activation leads to activation and propagation of the complement system

25 October 2020

1 reference

https://api.crossref.org/works/10.1182/BLOOD-2013-03-489245

Structure/function of C5 convertases of complement

25 October 2020

1 reference

https://api.crossref.org/works/10.1182/BLOOD-2013-03-489245

Activation of complement in normal serum by hydrogen peroxide and hydrogen peroxide-related oxygen radicals produced by activated neutrophils

25 October 2020

1 reference

https://api.crossref.org/works/10.1182/BLOOD-2013-03-489245

Cross-talk between the complement system and endothelial cells in physiologic conditions and in vascular diseases

25 October 2020

1 reference

https://api.crossref.org/works/10.1182/BLOOD-2013-03-489245

Oxidative stress renders retinal pigment epithelial cells susceptible to complement-mediated injury.

25 October 2020

1 reference

https://api.crossref.org/works/10.1182/BLOOD-2013-03-489245

Dynamic control of the complement system by modulated expression of regulatory proteins

25 October 2020

1 reference

https://api.crossref.org/works/10.1182/BLOOD-2013-03-489245

C4b-binding Protein and Factor H Compensate for the Loss of Membrane-bound Complement Inhibitors to Protect Apoptotic Cells against Excessive Complement Attack

25 October 2020

1 reference

https://api.crossref.org/works/10.1182/BLOOD-2013-03-489245

Clinical features of anti-factor H autoantibody-associated hemolytic uremic syndrome

25 October 2020

1 reference

https://api.crossref.org/works/10.1182/BLOOD-2013-03-489245

Complement factor H binds malondialdehyde epitopes and protects from oxidative stress

25 October 2020

1 reference

https://api.crossref.org/works/10.1182/BLOOD-2013-03-489245

Endothelial-cell heme uptake from heme proteins: induction of sensitization and desensitization to oxidant damage

25 October 2020

1 reference

https://api.crossref.org/works/10.1182/BLOOD-2013-03-489245

Oxygen radicals induce human endothelial cells to express GMP-140 and bind neutrophils

25 October 2020

1 reference

https://api.crossref.org/works/10.1182/BLOOD-2013-03-489245

Alternative pathway activation of complement by Shiga toxin promotes exuberant C3a formation that triggers microvascular thrombosis

25 October 2020

1 reference

https://api.crossref.org/works/10.1182/BLOOD-2013-03-489245

Interactions of hemin, antimalarial drugs and hemin-antimalarial complexes with phospholipid monolayers

25 October 2020

1 reference

https://api.crossref.org/works/10.1182/BLOOD-2013-03-489245

Brief Report: Plasma Concentrations of Hemopexin, Haptoglobin and Heme in Patients with Various Hemolytic Diseases

25 October 2020

1 reference

https://api.crossref.org/works/10.1182/BLOOD-2013-03-489245

10.1182/BLOOD-2013-03-489245

25 October 2020

Identifiers

DOI

1 reference

26 July 2017

1 reference