(Q34620765)

3 August 2017

1 reference

title

Mitochondrial kinase signalling pathways in myocardial protection from ischaemia/reperfusion-induced necrosis (English)

1 reference

3 August 2017

Tetsuji Miura

1

1 reference

3 August 2017

Masaya Tanno

series ordinal

2

1 reference

3 August 2017

Tatsuya Sato

series ordinal

3

1 reference

3 August 2017

language of work or name

1 reference

18 June 2010

1 reference

3 August 2017

Cardiovascular Research

1 reference

3 August 2017

88

1 reference

3 August 2017

7-15

1 reference

3 August 2017

1

1 reference

Mitochondrial membrane permeabilization in cell death

3 August 2017

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

Regulation and pharmacology of the mitochondrial permeability transition pore

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

The role of mitochondria in apoptosis*.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

The cell biology of acute myocardial ischemia

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

Mitochondrial dysfunction in cardiac disease: ischemia--reperfusion, aging, and heart failure

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

Effect of inhibition of Na(+)/Ca(2+) exchanger at the time of myocardial reperfusion on hypercontracture and cell death

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

Ion transport and energetics during cell death and protection

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

Mitochondrial formation of reactive oxygen species

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

The role of oxidants and free radicals in reperfusion injury

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

Sanglifehrin A acts as a potent inhibitor of the mitochondrial permeability transition and reperfusion injury of the heart by binding to cyclophilin-D at a different site from cyclosporin A.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

Specific inhibition of the mitochondrial permeability transition prevents lethal reperfusion injury

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

Inhibition of GSK3beta by postconditioning is required to prevent opening of the mitochondrial permeability transition pore during reperfusion

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

Protection of ischemic myocardium in dogs using intracoronary 2,3-butanedione monoxime (BDM).

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

The ADP/ATP translocator is not essential for the mitochondrial permeability transition pore

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

Voltage-dependent anion channels are dispensable for mitochondrial-dependent cell death

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

The mitochondrial phosphate carrier interacts with cyclophilin D and may play a key role in the permeability transition

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

Cyclophilin-D promotes the mitochondrial permeability transition but has opposite effects on apoptosis and necrosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

Cyclophilin D-dependent mitochondrial permeability transition regulates some necrotic but not apoptotic cell death

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

Loss of cyclophilin D reveals a critical role for mitochondrial permeability transition in cell death

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

What is the mitochondrial permeability transition pore?

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

Mitochondrial non-specific pores remain closed during cardiac ischaemia, but open upon reperfusion

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

Signaling pathways in ischemic preconditioning

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

GSK-3beta, a therapeutic target for cardiomyocyte protection

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

Limitation of infarct size by erythropoietin is associated with translocation of Akt to the mitochondria after reperfusion.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

Convergent signal transduction pathways controlling cardiomyocyte survival and function: the role of PI 3-kinase and Akt.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

Rapid accumulation of Akt in mitochondria following phosphatidylinositol 3-kinase activation

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

Akt mediates mitochondrial protection in cardiomyocytes through phosphorylation of mitochondrial hexokinase-II.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

3'Phosphoinositide-dependent kinase-1 is essential for ischemic preconditioning of the myocardium.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

Phenotypic spectrum caused by transgenic overexpression of activated Akt in the heart

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

Sex differences in the mechanism of Met5-enkephalin-induced cardioprotection: role of PI3K/Akt

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

Preconditioning-mimetics bradykinin and DADLE activate PI3-kinase through divergent pathways

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

Localizing extracellular signal-regulated kinase (ERK) in pharmacological preconditioning's trigger pathway

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

Endogenous adenosine protects preconditioned heart during early minutes of reperfusion by activating Akt.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

Infarct limitation by a protein kinase G activator at reperfusion in rabbit hearts is dependent on sensitizing the heart to A2b agonists by protein kinase C

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

Ischemic preconditioning, insulin, and morphine all cause hexokinase redistribution

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

Inhibiting mitochondrial permeability transition pore opening: a new paradigm for myocardial preconditioning?

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

Erythropoietin, modified to not stimulate red blood cell production, retains its cardioprotective properties.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

Protein kinase C: poised to signal

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

Opening of mitochondrial K(ATP) channel occurs downstream of PKC-epsilon activation in the mechanism of preconditioning

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

Targeted disruption of the protein kinase C epsilon gene abolishes the infarct size reduction that follows ischaemic preconditioning of isolated buffer-perfused mouse hearts

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

Sustained in vivo cardiac protection by a rationally designed peptide that causes epsilon protein kinase C translocation

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

Functional proteomic analysis of a three-tier PKCepsilon-Akt-eNOS signaling module in cardiac protection

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

Protein kinase C protects preconditioned rabbit hearts by increasing sensitivity of adenosine A2b-dependent signaling during early reperfusion

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

Mitochondrial PKCepsilon and MAPK form signaling modules in the murine heart: enhanced mitochondrial PKCepsilon-MAPK interactions and differential MAPK activation in PKCepsilon-induced cardioprotection

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

Protein kinase Cepsilon interacts with and inhibits the permeability transition pore in cardiac mitochondria

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

Activation of mitochondrial ERK protects cancer cells from death through inhibition of the permeability transition

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

Protein kinase G transmits the cardioprotective signal from cytosol to mitochondria

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

Mitochondrial aldehyde dehydrogenase and cardiac diseases

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

Expression of activated PKC epsilon (PKC epsilon) protects the ischemic heart, without attenuating ischemic H(+) production

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

PKCepsilon activation augments cardiac mitochondrial respiratory post-anoxic reserve--a putative mechanism in PKCepsilon cardioprotection.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

Forward operation of adenine nucleotide translocase during F0F1-ATPase reversal: critical role of matrix substrate-level phosphorylation

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

Sex differences in the phosphorylation of mitochondrial proteins result in reduced production of reactive oxygen species and cardioprotection in females

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

Reperfusion-induced translocation of deltaPKC to cardiac mitochondria prevents pyruvate dehydrogenase reactivation

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

Translocation of deltaPKC to mitochondria during cardiac reperfusion enhances superoxide anion production and induces loss in mitochondrial function.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

Inhibition of delta-protein kinase C protects against reperfusion injury of the ischemic heart in vivo

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

Acute PKCdelta inhibition limits ischaemia-reperfusion injury in the aged rat heart: role of GSK-3beta

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

Proteomic and metabolomic analysis of cardioprotection: Interplay between protein kinase C epsilon and delta in regulating glucose metabolism of murine hearts

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

Ischaemic preconditioning improves proteasomal activity and increases the degradation of deltaPKC during reperfusion

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

DeltaPKC-mediated activation of epsilonPKC in ethanol-induced cardiac protection from ischemia

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

Regulation of cardiac hypertrophy by intracellular signalling pathways

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

Tumor cell phenotype is sustained by selective MAPK oxidation in mitochondria

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

Mitochondrially localized ERK2 regulates mitophagy and autophagic cell stress: implications for Parkinson's disease

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

ERK phosphorylation mediates sildenafil-induced myocardial protection against ischemia-reperfusion injury in mice.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

Multiple, brief coronary occlusions during early reperfusion protect rabbit hearts by targeting cell signaling pathways.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

Role of the gap junction in ischemic preconditioning in the heart

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

A novel estrogen receptor GPER inhibits mitochondria permeability transition pore opening and protects the heart against ischemia-reperfusion injury

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

Glycogen synthase kinase-3 (GSK3): inflammation, diseases, and therapeutics

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

Further evidence that the tyrosine phosphorylation of glycogen synthase kinase-3 (GSK3) in mammalian cells is an autophosphorylation event

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

Ser9 phosphorylation of mitochondrial GSK-3beta is a primary mechanism of cardiomyocyte protection by erythropoietin against oxidant-induced apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

Phosphorylation of glycogen synthase kinase-3beta during preconditioning through a phosphatidylinositol-3-kinase--dependent pathway is cardioprotective

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

Endoplasmic reticulum stress in diabetic hearts abolishes erythropoietin-induced myocardial protection by impairment of phospho-glycogen synthase kinase-3beta-mediated suppression of mitochondrial permeability transition

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

Angiotensin II type 1 receptor-mediated upregulation of calcineurin activity underlies impairment of cardioprotective signaling in diabetic hearts

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

Glycogen synthase kinase 3 inhibition slows mitochondrial adenine nucleotide transport and regulates voltage-dependent anion channel phosphorylation

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

Glycogen Synthase Kinase-3β (GSK3β) Binds to and Promotes the Actions of p53

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

Inhibitory phosphorylation of glycogen synthase kinase-3 (GSK-3) in response to lithium. Evidence for autoregulation of GSK-3.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

In self-defence: hexokinase promotes voltage-dependent anion channel closure and prevents mitochondria-mediated apoptotic cell death

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

Regulation of hexokinase binding to VDAC

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

Hexokinase II detachment from mitochondria triggers apoptosis through the permeability transition pore independent of voltage-dependent anion channels

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

Glucose phosphorylation and mitochondrial binding are required for the protective effects of hexokinases I and II.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

Mitochondrial hexokinase II promotes neuronal survival and acts downstream of glycogen synthase kinase-3.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

Hexokinase-mitochondria interaction mediated by Akt is required to inhibit apoptosis in the presence or absence of Bax and Bak

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

Akt inhibits apoptosis downstream of BID cleavage via a glucose-dependent mechanism involving mitochondrial hexokinases

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

A reevaluation of the roles of hexokinase I and II in the heart.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

Inhibition of mitochondrial permeability transition pore opening by ischemic preconditioning is probably mediated by reduction of oxidative stress rather than mitochondrial protein phosphorylation.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

Ischemic postconditioning in pigs: no causal role for RISK activation.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

Glycogen synthase kinase-3 inactivation is not required for ischemic preconditioning or postconditioning in the mouse

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

Interaction of cardiovascular risk factors with myocardial ischemia/reperfusion injury, preconditioning, and postconditioning.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

Glycogen synthase kinase-3β mediates convergence of protection signaling to inhibit the mitochondrial permeability transition pore

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVQ206

21 January 2018

Identifiers

DOI

10.1093/CVR/CVQ206

1 reference

release_vq7y3vgk6fhidatlwfjf3uyh6e

3 August 2017

1 reference

Fatcat

https://api.fatcat.wiki/v0/release/vq7y3vgk6fhidatlwfjf3uyh6e

mapped directly with Wikidata item

24 November 2022

based on heuristic

1 reference