(Q35032088)

5 August 2017

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title

Ubiquitin and synaptic dysfunction: ataxic mice highlight new common themes in neurological disease (English)

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5 August 2017

Michael D Ehlers

1

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5 August 2017

publication date

1 January 2003

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5 August 2017

published in

Trends in Neurosciences

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5 August 2017

volume

26

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5 August 2017

issue

1

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5 August 2017

page(s)

4-7

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Naturally secreted oligomers of amyloid beta protein potently inhibit hippocampal long-term potentiation in vivo

5 August 2017

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7 January 2021

Deubiquitinating enzymes: their diversity and emerging roles

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7 January 2021

The ubiquitin-proteasome proteolytic pathway: destruction for the sake of construction

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7 January 2021

Protein regulation by monoubiquitin

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7 January 2021

Regulation of ubiquitin-dependent processes by deubiquitinating enzymes

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7 January 2021

Nonhydrolyzable diubiquitin analogues are inhibitors of ubiquitin conjugation and deconjugation

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Morphologic abnormalities in the postnatal differentiation of CA1 pyramidal cells and granule cells in the hippocampal formation of the ataxic mouse

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7 January 2021

New roles for ubiquitin in the assembly and function of neuronal circuits

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7 January 2021

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Use-dependent effects of amyloidogenic fragments of (beta)-amyloid precursor protein on synaptic plasticity in rat hippocampus in vivo.

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7 January 2021

Generation of aggregated beta-amyloid in the rat hippocampus impairs synaptic transmission and plasticity and causes memory deficits

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7 January 2021

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7 January 2021

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7 January 2021

Failure of the ubiquitin-proteasome system in Parkinson's disease

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7 January 2021

Mutation of the Angelman ubiquitin ligase in mice causes increased cytoplasmic p53 and deficits of contextual learning and long-term potentiation.

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7 January 2021

A role of the ubiquitin-proteasome system in neuropathic pain

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7 January 2021

Parkin and CASK/LIN-2 associate via a PDZ-mediated interaction and are co-localized in lipid rafts and postsynaptic densities in brain

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7 January 2021

Parkin functions as an E2-dependent ubiquitin- protein ligase and promotes the degradation of the synaptic vesicle-associated protein, CDCrel-1

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

Expression of Alzheimer's disease-associated presenilin-1 is controlled by proteolytic degradation and complex formation

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7 January 2021

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Identifiers

DOI

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5 August 2017

1 reference