(Q35058606)

6 August 2017

1 reference

title

Mitochondria, calcium and pro-apoptotic proteins as mediators in cell death signaling (English)

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6 August 2017

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S S Smaili

1

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6 August 2017

Y-T Hsu

2

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6 August 2017

A C P Carvalho

3

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6 August 2017

T R Rosenstock

4

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6 August 2017

J C Sharpe

5

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6 August 2017

R J Youle

6

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6 August 2017

language of work or name

English

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publication date

29 January 2003

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Brazilian Journal of Medical and Biological Research

6 August 2017

published in

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6 August 2017

volume

36

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6 August 2017

issue

2

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6 August 2017

page(s)

183-190

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Mitochondria and apoptosis

6 August 2017

cites work

1 reference

https://api.crossref.org/works/10.1590%2FS0100-879X2003000200004

Mitochondria in Ca2+ signaling and apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1590%2FS0100-879X2003000200004

The mitochondrial death/life regulator in apoptosis and necrosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1590%2FS0100-879X2003000200004

Smac, a mitochondrial protein that promotes cytochrome c-dependent caspase activation by eliminating IAP inhibition

21 January 2018

1 reference

https://api.crossref.org/works/10.1590%2FS0100-879X2003000200004

Bax and adenine nucleotide translocator cooperate in the mitochondrial control of apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1590%2FS0100-879X2003000200004

Calcium ion-dependent signalling and mitochondrial dysfunction: mitochondrial calcium uptake during hormonal stimulation in intact liver cells and its implication for the mitochondrial permeability transition

21 January 2018

1 reference

https://api.crossref.org/works/10.1590%2FS0100-879X2003000200004

Regulation of Ca 2+ release from mitochondria by the oxidation-reduction state of pyridine nucleotides

21 January 2018

1 reference

https://api.crossref.org/works/10.1590%2FS0100-879X2003000200004

Oxidative stress, thiol reagents, and membrane potential modulate the mitochondrial permeability transition by affecting nucleotide binding to the adenine nucleotide translocase

21 January 2018

1 reference

https://api.crossref.org/works/10.1590%2FS0100-879X2003000200004

Complexes between porin, hexokinase, mitochondrial creatine kinase and adenylate translocator display properties of the permeability transition pore. Implication for regulation of permeability transition by the kinases

21 January 2018

1 reference

https://api.crossref.org/works/10.1590%2FS0100-879X2003000200004

Mitochondrial permeability transition and oxidative stress

21 January 2018

1 reference

https://api.crossref.org/works/10.1590%2FS0100-879X2003000200004

The role of reactive oxygen species in mitochondrial permeability transition

21 January 2018

1 reference

https://api.crossref.org/works/10.1590%2FS0100-879X2003000200004

Cyclosporin A inhibits inositol 1,4,5-trisphosphate-dependent Ca2+ signals by enhancing Ca2+ uptake into the endoplasmic reticulum and mitochondria

21 January 2018

1 reference

https://api.crossref.org/works/10.1590%2FS0100-879X2003000200004

Membrane-linked systems preventing superoxide formation

21 January 2018

1 reference

https://api.crossref.org/works/10.1590%2FS0100-879X2003000200004

The Bcl-2 protein family: arbiters of cell survival

21 January 2018

1 reference

https://api.crossref.org/works/10.1590%2FS0100-879X2003000200004

Cytosol-to-membrane redistribution of Bax and Bcl-X(L) during apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1590%2FS0100-879X2003000200004

Cleavage of BID by caspase 8 mediates the mitochondrial damage in the Fas pathway of apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1590%2FS0100-879X2003000200004

X-ray and NMR structure of human Bcl-xL, an inhibitor of programmed cell death

21 January 2018

1 reference

https://api.crossref.org/works/10.1590%2FS0100-879X2003000200004

Diphtheria toxin fragment forms large pores in phospholipid bilayer membranes

21 January 2018

1 reference

https://api.crossref.org/works/10.1590%2FS0100-879X2003000200004

Comparison of the ion channel characteristics of proapoptotic BAX and antiapoptotic BCL-2.

21 January 2018

1 reference

https://api.crossref.org/works/10.1590%2FS0100-879X2003000200004

Bax directly induces release of cytochrome c from isolated mitochondria

21 January 2018

1 reference

https://api.crossref.org/works/10.1590%2FS0100-879X2003000200004

Apaf-1, a human protein homologous to C. elegans CED-4, participates in cytochrome c-dependent activation of caspase-3

21 January 2018

1 reference

https://api.crossref.org/works/10.1590%2FS0100-879X2003000200004

Cytochrome c and dATP-dependent formation of Apaf-1/caspase-9 complex initiates an apoptotic protease cascade

21 January 2018

1 reference

https://api.crossref.org/works/10.1590%2FS0100-879X2003000200004

Bax interacts with the permeability transition pore to induce permeability transition and cytochrome c release in isolated mitochondria

21 January 2018

1 reference

https://api.crossref.org/works/10.1590%2FS0100-879X2003000200004

The coordinate release of cytochrome c during apoptosis is rapid, complete and kinetically invariant

21 January 2018

1 reference

https://api.crossref.org/works/10.1590%2FS0100-879X2003000200004

Bax-induced cytochrome C release from mitochondria is independent of the permeability transition pore but highly dependent on Mg2+ ions

21 January 2018

1 reference

https://api.crossref.org/works/10.1590%2FS0100-879X2003000200004

Bcl-2 heterodimerizes in vivo with a conserved homolog, Bax, that accelerates programmed cell death

21 January 2018

1 reference

https://api.crossref.org/works/10.1590%2FS0100-879X2003000200004

Bax-deficient mice with lymphoid hyperplasia and male germ cell death

21 January 2018

1 reference

https://api.crossref.org/works/10.1590%2FS0100-879X2003000200004

Tumor-cell resistance to death receptor--induced apoptosis through mutational inactivation of the proapoptotic Bcl-2 homolog Bax.

21 January 2018

1 reference

https://api.crossref.org/works/10.1590%2FS0100-879X2003000200004

Structure-function comparisons of the proapoptotic protein Bax in yeast and mammalian cells

21 January 2018

1 reference

https://api.crossref.org/works/10.1590%2FS0100-879X2003000200004

Structure of Bax: coregulation of dimer formation and intracellular localization

21 January 2018

1 reference

https://api.crossref.org/works/10.1590%2FS0100-879X2003000200004

Bax translocation to mitochondria subsequent to a rapid loss of mitochondrial membrane potential

21 January 2018

1 reference

https://api.crossref.org/works/10.1590%2FS0100-879X2003000200004

Conformation of the Bax C-terminus regulates subcellular location and cell death

21 January 2018

1 reference

https://api.crossref.org/works/10.1590%2FS0100-879X2003000200004

Bax and Bak coalesce into novel mitochondria-associated clusters during apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1590%2FS0100-879X2003000200004

Bax oligomerization is required for channel-forming activity in liposomes and to trigger cytochrome c release from mitochondria

21 January 2018

1 reference

https://api.crossref.org/works/10.1590%2FS0100-879X2003000200004

Bcl-xL regulates the membrane potential and volume homeostasis of mitochondria

21 January 2018

1 reference

https://api.crossref.org/works/10.1590%2FS0100-879X2003000200004

Caspase cleaved BID targets mitochondria and is required for cytochrome c release, while BCL-XL prevents this release but not tumor necrosis factor-R1/Fas death

21 January 2018

1 reference

https://api.crossref.org/works/10.1590%2FS0100-879X2003000200004

Pro-apoptotic cascade activates BID, which oligomerizes BAK or BAX into pores that result in the release of cytochrome c.

21 January 2018

1 reference

https://api.crossref.org/works/10.1590%2FS0100-879X2003000200004

Proapoptotic BAX and BAK: a requisite gateway to mitochondrial dysfunction and death

21 January 2018

1 reference

https://api.crossref.org/works/10.1590%2FS0100-879X2003000200004

A reversible component of mitochondrial respiratory dysfunction in apoptosis can be rescued by exogenous cytochrome c.

21 January 2018

1 reference

https://api.crossref.org/works/10.1590%2FS0100-879X2003000200004

Mechanisms of neuronal damage in brain hypoxia/ischemia: focus on the role of mitochondrial calcium accumulation.

21 January 2018

1 reference

https://api.crossref.org/works/10.1590%2FS0100-879X2003000200004

The role of calcium in the regulation of apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1590%2FS0100-879X2003000200004

Mitochondrial ca(2+) signaling and cardiac apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1590%2FS0100-879X2003000200004

Synergistic movements of Ca(2+) and Bax in cells undergoing apoptosis.

21 January 2018

1 reference

https://api.crossref.org/works/10.1590%2FS0100-879X2003000200004

Caffeine sensitizes human H358 cell line to p53-mediated apoptosis by inducing mitochondrial translocation and conformational change of BAX protein.

21 January 2018

1 reference

https://api.crossref.org/works/10.1590%2FS0100-879X2003000200004

tcBid promotes Ca(2+) signal propagation to the mitochondria: control of Ca(2+) permeation through the outer mitochondrial membrane

21 January 2018

1 reference

https://api.crossref.org/works/10.1590%2FS0100-879X2003000200004

A distinct pathway remodels mitochondrial cristae and mobilizes cytochrome c during apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1590%2FS0100-879X2003000200004

10.1590/S0100-879X2003000200004

21 January 2018

Identifiers

DOI

1 reference

release_53hhcmfz6bdn7pc4piah4gzapq

6 August 2017

Fatcat ID

1 reference

Fatcat

https://api.fatcat.wiki/v0/release/53hhcmfz6bdn7pc4piah4gzapq

mapped directly with Wikidata item

24 November 2022

based on heuristic

1 reference