(Q36093080)

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The Selective Degradation of Synaptic Connexin 43 Protein by Hypoxia-induced Autophagy Impairs Natural Killer Cell-mediated Tumor Cell Killing

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Europe PubMed Central

PMC publication ID

12 August 2017

The Selective Degradation of Synaptic Connexin 43 Protein by Hypoxia-induced Autophagy Impairs Natural Killer Cell-mediated Tumor Cell Killing (English)

1 reference

Europe PubMed Central

PMC publication ID

12 August 2017

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Salem Chouaib

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Europe PubMed Central

PMC publication ID

12 August 2017

author name string

Andrés Tittarelli

1

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Europe PubMed Central

PMC publication ID

12 August 2017

Bassam Janji

2

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Europe PubMed Central

PMC publication ID

12 August 2017

Kris Van Moer

3

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Europe PubMed Central

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12 August 2017

Muhammad Zaeem Noman

4

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12 August 2017

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publication date

28 July 2015

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Europe PubMed Central

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12 August 2017

Journal of Biological Chemistry

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Europe PubMed Central

PMC publication ID

12 August 2017

290

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Europe PubMed Central

PMC publication ID

12 August 2017

39

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Europe PubMed Central

PMC publication ID

12 August 2017

23670-23679

1 reference

Europe PubMed Central

PMC publication ID

12 August 2017

describes a project that uses

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Europe PubMed Central

11 June 2022

https://www.ebi.ac.uk/europepmc/webservices/rest/PMC4583044/fullTextXML

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Tumor plasticity interferes with anti-tumor immunity

1 reference

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Protein kinase C (PKC) mediated interaction between conexin43 (Cx43) and K(+)(ATP) channel subunit (Kir6.1) in cardiomyocyte mitochondria: Implications in cytoprotection against hypoxia induced cell apoptosis

1 reference

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Probenecid blocks human P2X7 receptor-induced dye uptake via a pannexin-1 independent mechanism.

1 reference

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Interactions between autophagy receptors and ubiquitin-like proteins form the molecular basis for selective autophagy.

1 reference

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Cell biological steps and checkpoints in accessing NK cell cytotoxicity

1 reference

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Gap junction intercellular communications regulate NK cell activation and modulate NK cytotoxic capacity

1 reference

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Connexin a check-point component of cell apoptosis in normal and physiopathological conditions

1 reference

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Granzyme B degradation by autophagy decreases tumor cell susceptibility to natural killer-mediated lysis under hypoxia

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Targeting autophagy as a potential therapeutic approach for melanoma therapy

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Gap junctional communication promotes apoptosis in a connexin-type-dependent manner

1 reference

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1 reference

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Primary tumor hypoxia recruits CD11b+/Ly6Cmed/Ly6G+ immune suppressor cells and compromises NK cell cytotoxicity in the premetastatic niche.

1 reference

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Internalized gap junctions are degraded by autophagy

1 reference

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Autophagy modulates dynamics of connexins at the plasma membrane in a ubiquitin-dependent manner

1 reference

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Tumor microenvironment complexity: emerging roles in cancer therapy.

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Connexin43 acts as a colorectal cancer tumor suppressor and predicts disease outcome

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Functional gap junctions accumulate at the immunological synapse and contribute to T cell activation

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Blocking hypoxia-induced autophagy in tumors restores cytotoxic T-cell activity and promotes regression.

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The gap junction protein Cx43 regulates B-lymphocyte spreading and adhesion.

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ATP and glutamate released via astroglial connexin 43 hemichannels mediate neuronal death through activation of pannexin 1 hemichannels

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New views on natural killer cell-based immunotherapy for melanoma treatment.

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2 October 2017

A novel role of gap junction connexin46 protein to protect breast tumors from hypoxia

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HIF-1: upstream and downstream of cancer metabolism

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2 October 2017

The cooperative induction of hypoxia-inducible factor-1 alpha and STAT3 during hypoxia induced an impairment of tumor susceptibility to CTL-mediated cell lysis.

1 reference

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p62/SQSTM1 binds directly to Atg8/LC3 to facilitate degradation of ubiquitinated protein aggregates by autophagy

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2 October 2017

Gap junction-mediated intercellular communication in the immune system

1 reference

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Genistein and quercetin increase connexin43 and suppress growth of breast cancer cells

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2 October 2017

Zonula occludens-1 alters connexin43 gap junction size and organization by influencing channel accretion

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The role of altered cell-cell communication in melanoma progression

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Chromosome 9p deletions in cutaneous malignant melanoma tumors: the minimal deleted region involves markers outside the p16 (CDKN2) gene.

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Gap junction intercellular communication mediated by connexin43 in astrocytes is essential for their resistance to oxidative stress

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Bacteria-induced gap junctions in tumors favor antigen cross-presentation and antitumor immunity.

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Implications and challenges of connexin connections to cancer.

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Gap junctions at the dendritic cell-T cell interface are key elements for antigen-dependent T cell activation.

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1 reference

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https://pubmed.ncbi.nlm.nih.gov/26221040

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1 reference

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1 reference

https://pubmed.ncbi.nlm.nih.gov/26221040

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based on heuristic

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Identifiers

10.1074/JBC.M115.651547

1 reference

Europe PubMed Central

PMC publication ID

12 August 2017

PMC publication ID

1 reference

Europe PubMed Central

PMC publication ID

12 August 2017

PubMed publication ID

1 reference

Europe PubMed Central

PMC publication ID

12 August 2017

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