(Q37178911)

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Noncanonical NF-kappaB activation requires coordinated assembly of a regulatory complex of the adaptors cIAP1, cIAP2, TRAF2 and TRAF3 and the kinase NIK.

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scholarly article

1 reference

Europe PubMed Central

18 August 2017

Noncanonical NF-kappaB activation requires coordinated assembly of a regulatory complex of the adaptors cIAP1, cIAP2, TRAF2 and TRAF3 and the kinase NIK. (English)

1 reference

Europe PubMed Central

18 August 2017

1 reference

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object named as

Genhong Cheng

1 reference

Europe PubMed Central

18 August 2017

author name string

Brian J Zarnegar

1

1 reference

Europe PubMed Central

18 August 2017

Yaya Wang

2

1 reference

Europe PubMed Central

18 August 2017

Douglas J Mahoney

3

1 reference

Europe PubMed Central

18 August 2017

Paul W Dempsey

4

1 reference

Europe PubMed Central

18 August 2017

Herman H Cheung

5

1 reference

Europe PubMed Central

18 August 2017

Jeannie He

6

1 reference

Europe PubMed Central

18 August 2017

Travis Shiba

7

1 reference

Europe PubMed Central

18 August 2017

Xiaolu Yang

8

1 reference

Europe PubMed Central

18 August 2017

Wen-Chen Yeh

9

1 reference

Europe PubMed Central

18 August 2017

Tak W Mak

10

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Europe PubMed Central

18 August 2017

Robert G Korneluk

11

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Europe PubMed Central

18 August 2017

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publication date

9 November 2008

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Europe PubMed Central

18 August 2017

Nature Immunology

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Europe PubMed Central

18 August 2017

9

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Europe PubMed Central

18 August 2017

1371-1378

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Europe PubMed Central

18 August 2017

12

1 reference

Europe PubMed Central

18 August 2017

https://scigraph.springernature.com/pub.10.1038/ni.1676

0 references

TRAF2 and TRAF3 signal adapters act cooperatively to control the maturation and survival signals delivered to B cells by the BAFF receptor.

1 reference

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Control of canonical NF-kappaB activation through the NIK-IKK complex pathway

1 reference

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IAP antagonists target cIAP1 to induce TNFalpha-dependent apoptosis

1 reference

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IAP antagonists induce autoubiquitination of c-IAPs, NF-kappaB activation, and TNFalpha-dependent apoptosis

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Promiscuous mutations activate the noncanonical NF-kappaB pathway in multiple myeloma.

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Frequent engagement of the classical and alternative NF-kappaB pathways by diverse genetic abnormalities in multiple myeloma

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The inhibitors of apoptosis (IAPs) as cancer targets

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Specificity of TRAF3 in its negative regulation of the noncanonical NF-kappa B pathway

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Rescue of TRAF3-null mice by p100 NF-kappa B deficiency

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Regulation of antiviral responses by a direct and specific interaction between TRAF3 and Cardif

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Posttranscriptional downregulation of c-IAP2 by the ubiquitin protein ligase c-IAP1 in vivo

1 reference

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Activation of IKKalpha target genes depends on recognition of specific kappaB binding sites by RelB:p52 dimers

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A small molecule Smac mimic potentiates TRAIL- and TNFalpha-mediated cell death

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CD40 regulates the processing of NF-kappaB2 p100 to p52

1 reference

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Bridging the gap: composition, regulation, and physiological function of the IkappaB kinase complex

1 reference

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MAP3K-related kinase involved in NF-kappaB induction by TNF, CD95 and IL-1

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NF-kappa B: ten years after

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The TNFR2-TRAF signaling complex contains two novel proteins related to baculoviral inhibitor of apoptosis proteins

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Constitutive NF-kappa B activation, enhanced granulopoiesis, and neonatal lethality in I kappa B alpha-deficient mice

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Clonal lymphoid progenitor cell lines expressing the BCR/ABL oncogene retain full differentiative function

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A Smac mimetic rescue screen reveals roles for inhibitor of apoptosis proteins in tumor necrosis factor-alpha signaling

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Regulation of the NF-kappaB-inducing kinase by tumor necrosis factor receptor-associated factor 3-induced degradation

1 reference

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NF-kappa B and rel proteins in innate immunity

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Alymphoplasia is caused by a point mutation in the mouse gene encoding Nf-kappa b-inducing kinase

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Essential role of nuclear factor (NF)-kappaB-inducing kinase and inhibitor of kappaB (IkappaB) kinase alpha in NF-kappaB activation through lymphotoxin beta receptor, but not through tumor necrosis factor receptor I

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An intact zinc ring finger is required for tumor necrosis factor receptor-associated factor-mediated nuclear factor-kappaB activation but is dispensable for c-Jun N-terminal kinase signaling

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BAFF-induced NEMO-independent processing of NF-kappa B2 in maturing B cells.

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10.1038/NI.1676

1 reference

Europe PubMed Central

18 August 2017

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1 reference

Europe PubMed Central

18 August 2017

1 reference

Europe PubMed Central

18 August 2017

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