(Q37608457)

English

Receptor-interacting protein 1 increases chemoresistance by maintaining inhibitor of apoptosis protein levels and reducing reactive oxygen species through a microRNA-146a-mediated catalase pathway

scientific article published on 14 January 2014

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scholarly article

1 reference

Europe PubMed Central

21 August 2017

Receptor-interacting protein 1 increases chemoresistance by maintaining inhibitor of apoptosis protein levels and reducing reactive oxygen species through a microRNA-146a-mediated catalase pathway (English)

1 reference

Europe PubMed Central

21 August 2017

apoptotic process

0 references

1 reference

based on heuristic

inferred from title

reactive oxygen species

1 reference

based on heuristic

inferred from title

author name string

Qiong Wang

1

1 reference

Europe PubMed Central

21 August 2017

Wenshu Chen

2

1 reference

Europe PubMed Central

21 August 2017

Lang Bai

3

1 reference

Europe PubMed Central

21 August 2017

Wenjie Chen

4

1 reference

Europe PubMed Central

21 August 2017

Mabel T Padilla

5

1 reference

Europe PubMed Central

21 August 2017

Amy S Lin

6

1 reference

Europe PubMed Central

21 August 2017

Shaoqing Shi

7

1 reference

Europe PubMed Central

21 August 2017

Xia Wang

8

1 reference

Europe PubMed Central

21 August 2017

Yong Lin

9

1 reference

Europe PubMed Central

21 August 2017

language of work or name

0 references

publication date

14 January 2014

1 reference

Europe PubMed Central

21 August 2017

Journal of Biological Chemistry

1 reference

Europe PubMed Central

21 August 2017

289

1 reference

Europe PubMed Central

21 August 2017

9

1 reference

Europe PubMed Central

21 August 2017

5654-5663

1 reference

Europe PubMed Central

21 August 2017

RIP1 potentiates BPDE-induced transformation in human bronchial epithelial cells through catalase-mediated suppression of excessive reactive oxygen species

1 reference

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ARD1 binding to RIP1 mediates doxorubicin-induced NF-κB activation

1 reference

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1 reference

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RIP1 is required for IAP inhibitor-mediated sensitization of childhood acute leukemia cells to chemotherapy-induced apoptosis

1 reference

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Many stimuli pull the necrotic trigger, an overview

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1 reference

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Hallmarks of Cancer: The Next Generation

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Advances in chemotherapy in advanced non-small-cell lung cancer

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1 reference

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The role of mitochondria in reactive oxygen species metabolism and signaling

1 reference

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1 reference

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1 reference

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IAP-targeted therapies for cancer

1 reference

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c-IAP1 and c-IAP2 are critical mediators of tumor necrosis factor alpha (TNFalpha)-induced NF-kappaB activation

1 reference

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TNF-alpha induces two distinct caspase-8 activation pathways

1 reference

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RIP1, a kinase on the crossroads of a cell's decision to live or die

1 reference

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Apoptosis induced by the toll-like receptor adaptor TRIF is dependent on its receptor interacting protein homotypic interaction motif

1 reference

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The RIP kinases: crucial integrators of cellular stress

1 reference

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The death domain kinase RIP has an essential role in DNA damage-induced NF-kappa B activation

1 reference

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Fas triggers an alternative, caspase-8-independent cell death pathway using the kinase RIP as effector molecule

1 reference

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The death domain kinase RIP is essential for TRAIL (Apo2L)-induced activation of IkappaB kinase and c-Jun N-terminal kinase

1 reference

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The death domain kinase RIP mediates the TNF-induced NF-kappaB signal

1 reference

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The novel SMAC mimetic birinapant exhibits potent activity against human melanoma cells

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Wogonin enhances antitumor activity of tumor necrosis factor-related apoptosis-inducing ligand in vivo through ROS-mediated downregulation of cFLIPL and IAP proteins

1 reference

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Attenuation of TNFSF10/TRAIL-induced apoptosis by an autophagic survival pathway involving TRAF2- and RIPK1/RIP1-mediated MAPK8/JNK activation

1 reference

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Redox modulation of the DNA damage response

1 reference

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Tumor cell-protective catalase as a novel target for rational therapeutic approaches based on specific intercellular ROS signaling

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Chemotherapy, chemoresistance and the changing treatment landscape for NSCLC.

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Sensitization of TNF-induced cytotoxicity in lung cancer cells by concurrent suppression of the NF-kappaB and Akt pathways

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A critical role of luteolin-induced reactive oxygen species in blockage of tumor necrosis factor-activated nuclear factor-kappaB pathway and sensitization of apoptosis in lung cancer cells

1 reference

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1 reference

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RIP mediates tumor necrosis factor receptor 1 activation of NF-kappaB but not Fas/APO-1-initiated apoptosis

1 reference

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1 reference

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1 reference

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1 reference

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Identifiers

10.1074/JBC.M113.526152

1 reference

Europe PubMed Central

21 August 2017

1 reference

Europe PubMed Central

21 August 2017

1 reference

Europe PubMed Central

21 August 2017

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