(Q37679695)

21 August 2017

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title

Paraquat and Parkinson's disease. (English)

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21 August 2017

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2

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21 August 2017

C Berry

1

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21 August 2017

P Nicotera

series ordinal

3

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21 August 2017

language of work or name

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22 January 2010

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Cell Death & Differentiation

21 August 2017

published in

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21 August 2017

volume

17

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21 August 2017

issue

7

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21 August 2017

page(s)

1115-1125

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Genetic and environmental factors in the cause of Parkinson's disease

21 August 2017

cites work

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.217

Neurobiology and treatment of Parkinson's disease

21 January 2018

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https://api.crossref.org/works/10.1038%2FCDD.2009.217

Evaluation of epidemiologic and animal data associating pesticides with Parkinson's disease

21 January 2018

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Pesticides and Parkinson's disease--is there a link?

21 January 2018

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Parkinsonism and occupational exposure to pesticides

21 January 2018

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Neurodegenerative diseases and exposure to pesticides in the elderly.

21 January 2018

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https://api.crossref.org/works/10.1038%2FCDD.2009.217

Chemical exposures and Parkinson's disease: a population-based case-control study

21 January 2018

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https://api.crossref.org/works/10.1038%2FCDD.2009.217

Pesticide exposure and risk for Parkinson's disease

21 January 2018

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https://api.crossref.org/works/10.1038%2FCDD.2009.217

Environmental risk factors for Parkinson's disease and parkinsonism: the Geoparkinson study

21 January 2018

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https://api.crossref.org/works/10.1038%2FCDD.2009.217

Possible environmental, occupational, and other etiologic factors for Parkinson's disease: a case-control study in Germany.

21 January 2018

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https://api.crossref.org/works/10.1038%2FCDD.2009.217

Environmental risk factors and Parkinson's disease: a case-control study in Taiwan

21 January 2018

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https://api.crossref.org/works/10.1038%2FCDD.2009.217

Environmental risk factors in Parkinson's disease

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.217

Pesticides and risk of Parkinson disease: a population-based case-control study

21 January 2018

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https://api.crossref.org/works/10.1038%2FCDD.2009.217

Pesticide exposure and self-reported Parkinson's disease in the agricultural health study.

21 January 2018

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https://api.crossref.org/works/10.1038%2FCDD.2009.217

Parkinson's disease: a case-control study of occupational and environmental risk factors.

21 January 2018

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https://api.crossref.org/works/10.1038%2FCDD.2009.217

A case-control study of Parkinson's disease in a horticultural region of British Columbia

21 January 2018

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https://api.crossref.org/works/10.1038%2FCDD.2009.217

Attenuation of paraquat-induced dopaminergic toxicity on the substantia nigra by (-)-deprenyl in vivo

21 January 2018

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https://api.crossref.org/works/10.1038%2FCDD.2009.217

Occupation and risk of parkinsonism: a multicenter case-control study

21 January 2018

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https://api.crossref.org/works/10.1038%2FCDD.2009.217

Parkinson's disease and residential exposure to maneb and paraquat from agricultural applications in the central valley of California

21 January 2018

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https://api.crossref.org/works/10.1038%2FCDD.2009.217

Dopamine transporter genetic variants and pesticides in Parkinson's disease

21 January 2018

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https://api.crossref.org/works/10.1038%2FCDD.2009.217

Implications of small effect sizes of individual genetic variants on the design and interpretation of genetic association studies of complex diseases.

21 January 2018

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https://api.crossref.org/works/10.1038%2FCDD.2009.217

Design and analysis of randomized clinical trials requiring prolonged observation of each patient. I. Introduction and design

21 January 2018

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Design and analysis of randomized clinical trials requiring prolonged observation of each patient. II. analysis and examples

21 January 2018

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https://api.crossref.org/works/10.1038%2FCDD.2009.217

Role of oxidative stress in paraquat-induced dopaminergic cell degeneration.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.217

Toxicity of redox cycling pesticides in primary mesencephalic cultures

21 January 2018

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https://api.crossref.org/works/10.1038%2FCDD.2009.217

Redox cycling of the herbicide paraquat in microglial cultures

21 January 2018

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https://api.crossref.org/works/10.1038%2FCDD.2009.217

Paraquat disposition in rats, guinea pigs and monkeys

21 January 2018

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https://api.crossref.org/works/10.1038%2FCDD.2009.217

No changes in behaviour, nigro-striatal system neurochemistry or neuronal cell death following toxic multiple oral paraquat administration to rats.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.217

Carrier-mediated processes in blood--brain barrier penetration and neural uptake of paraquat

21 January 2018

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https://api.crossref.org/works/10.1038%2FCDD.2009.217

Chronic Parkinsonism in humans due to a product of meperidine-analog synthesis

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.217

Degeneration of dopaminergic mesocortical neurons and activation of compensatory processes induced by a long-term paraquat administration in rats: implications for Parkinson's disease.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.217

Divergent mechanisms of paraquat, MPP+, and rotenone toxicity: oxidation of thioredoxin and caspase-3 activation

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.217

Paraquat neurotoxicity is distinct from that of MPTP and rotenone.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.217

Decreased susceptibility to oxidative stress underlies the resistance of specific dopaminergic cell populations to paraquat-induced degeneration

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.217

Complex I is the major site of mitochondrial superoxide production by paraquat

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.217

Mitochondrial complex I inhibition is not required for dopaminergic neuron death induced by rotenone, MPP+, or paraquat.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.217

Chemical genomic profiling for identifying intracellular targets of toxicants producing Parkinson's disease

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.217

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.217

Neuroprotection and dopamine agonists

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.217

Modulation of microglial pro-inflammatory and neurotoxic activity for the treatment of Parkinson's disease

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.217

The role of microglia in paraquat-induced dopaminergic neurotoxicity

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.217

Loss of function mutations in the gene encoding Omi/HtrA2 in Parkinson's disease

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.217

The mitochondrial protease HtrA2 is regulated by Parkinson's disease-associated kinase PINK1.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.217

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.217

The herbicide paraquat causes up-regulation and aggregation of alpha-synuclein in mice: paraquat and alpha-synuclein

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.217

Stress-induced alterations in parkin solubility promote parkin aggregation and compromise parkin's protective function

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.217

Pesticides directly accelerate the rate of alpha-synuclein fibril formation: a possible factor in Parkinson's disease

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.217

A fetal risk factor for Parkinson's disease

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.217

Developmental exposure to the pesticides paraquat and maneb and the Parkinson's disease phenotype

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.217

In utero bacterial endotoxin exposure causes loss of tyrosine hydroxylase neurons in the postnatal rat midbrain.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.217

Microglial activation as a priming event leading to paraquat-induced dopaminergic cell degeneration

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.217

Lipopolysaccharide-induced inflammation exacerbates tau pathology by a cyclin-dependent kinase 5-mediated pathway in a transgenic model of Alzheimer's disease.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.217

SIC Task Force appraisal of clinical diagnostic criteria for parkinsonian disorders

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.217

7 January 2021

Paraquat induces oxidative stress and neuronal cell death; neuroprotection by water-soluble Coenzyme Q10

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.217

7 January 2021

Further evidence that the blood/brain barrier impedes paraquat entry into the brain

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.217

7 January 2021

α-Synuclein Overexpression Protects against Paraquat-Induced Neurodegeneration

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.217

7 January 2021

Developmental pesticide models of the Parkinson disease phenotype

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2009.217

7 January 2021

Identifiers

DOI

10.1038/CDD.2009.217

1 reference

21 August 2017

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