(Q38151252)

29 August 2017

title

Mitochondrial reactive oxygen species as a mystery voice in hepatitis C. (English)

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29 August 2017

main subject

reactive oxygen species

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1

Keisuke Hino

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29 August 2017

3

Sohji Nishina

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29 August 2017

2

Yuichi Hara

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29 August 2017

publication date

8 January 2014

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29 August 2017

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29 August 2017

volume

44

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29 August 2017

issue

2

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29 August 2017

page(s)

123-132

1 reference

https://api.crossref.org/works/10.1111%2FHEPR.12247

29 August 2017

1 reference

Iron storage, lipid peroxidation and glutathione turnover in chronic anti-HCV positive hepatitis

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FHEPR.12247

Oxidative stress EPR measurement in human liver by radical-probe technique. Correlation with etiology, histology and cell proliferation.

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FHEPR.12247

In situ detection of oxidized n-3 polyunsaturated fatty acids in chronic hepatitis C: correlation with hepatic steatosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FHEPR.12247

Mitochondrial injury, oxidative stress, and antioxidant gene expression are induced by hepatitis C virus core protein

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FHEPR.12247

Hepatitis C virus core protein, cytochrome P450 2E1, and alcohol produce combined mitochondrial injury and cytotoxicity in hepatoma cells.

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FHEPR.12247

Hepatitis C virus core protein inhibits mitochondrial electron transport and increases reactive oxygen species (ROS) production

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FHEPR.12247

Mitochondrial electron transport inhibition in full genomic hepatitis C virus replicon cells is restored by reducing viral replication

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FHEPR.12247

Evidence for a link between hepatitis C virus infection and diabetes mellitus in a cirrhotic population

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FHEPR.12247

Further evidence for an association between non-insulin-dependent diabetes mellitus and chronic hepatitis C virus infection

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FHEPR.12247

Association of diabetes mellitus and chronic hepatitis C virus infection.

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FHEPR.12247

Hepatitis C virus core protein induces hepatic steatosis in transgenic mice

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FHEPR.12247

Hepatocyte steatosis is a cytopathic effect of hepatitis C virus genotype 3.

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FHEPR.12247

Diabetes increases the risk of hepatocellular carcinoma in the United States: a population based case control study

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FHEPR.12247

The core protein of hepatitis C virus induces hepatocellular carcinoma in transgenic mice

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FHEPR.12247

Steatosis and liver cancer in transgenic mice expressing the structural and nonstructural proteins of hepatitis C virus

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FHEPR.12247

Long-term phlebotomy with low-iron diet therapy lowers risk of development of hepatocellular carcinoma from chronic hepatitis C.

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FHEPR.12247

Linking mitochondrial bioenergetics to insulin resistance via redox biology

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FHEPR.12247

Targeting of hepatitis C virus core protein to mitochondria through a novel C-terminal localization motif

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FHEPR.12247

Persistent expression of hepatitis C virus non-structural proteins leads to increased autophagy and mitochondrial injury in human hepatoma cells

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FHEPR.12247

Role of Hepatitis C virus core protein in viral-induced mitochondrial dysfunction

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FHEPR.12247

Subcellular localization of hepatitis C virus structural proteins in a cell culture system that efficiently replicates the virus

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FHEPR.12247

Hepatitis C virus-induced mitochondrial dysfunctions

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FHEPR.12247

Proteomics analysis of mitochondrial proteins reveals overexpression of a mitochondrial protein chaperon, prohibitin, in cells expressing hepatitis C virus core protein

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FHEPR.12247

Hepatitis C virus-core and non structural proteins lead to different effects on cellular antioxidant defenses

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FHEPR.12247

Expression of gastrointestinal glutathione peroxidase is inversely correlated to the presence of hepatitis C virus subgenomic RNA in human liver cells.

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FHEPR.12247

Hepatitis C Virus Triggers Mitochondrial Permeability Transition with Production of Reactive Oxygen Species, Leading to DNA Damage and STAT3 Activation

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FHEPR.12247

Cellular response to conditional expression of hepatitis C virus core protein in Huh7 cultured human hepatoma cells

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FHEPR.12247

Hepatitis C Virus (HCV) Proteins Induce NADPH Oxidase 4 Expression in a Transforming Growth Factor -Dependent Manner: a New Contributor to HCV-Induced Oxidative Stress

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FHEPR.12247

Hepatocyte NAD(P)H oxidases as an endogenous source of reactive oxygen species during hepatitis C virus infection

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FHEPR.12247

Cardiac energy metabolism homeostasis: role of cytosolic calcium

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FHEPR.12247

Human hepatitis C virus NS5A protein alters intracellular calcium levels, induces oxidative stress, and activates STAT-3 and NF-kappa B.

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FHEPR.12247

Hepatitis C virus core protein increases mitochondrial ROS production by stimulation of Ca2+ uniporter activity.

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FHEPR.12247

Hepatitis C virus protein expression causes calcium-mediated mitochondrial bioenergetic dysfunction and nitro-oxidative stress

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FHEPR.12247

Redox control of neural function: background, mechanisms, and significance

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FHEPR.12247

Parkin is recruited selectively to impaired mitochondria and promotes their autophagy

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FHEPR.12247

21 January 2018

Mechanisms of mitophagy

1 reference

https://api.crossref.org/works/10.1111%2FHEPR.12247

Mitophagy plays an essential role in reducing mitochondrial production of reactive oxygen species and mutation of mitochondrial DNA by maintaining mitochondrial quantity and quality in yeast

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FHEPR.12247

PINK1/Parkin-mediated mitophagy is dependent on VDAC1 and p62/SQSTM1

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FHEPR.12247

Hepatitis C virus induces the mitochondrial translocation of Parkin and subsequent mitophagy

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FHEPR.12247

Insulin resistance is associated with chronic hepatitis C virus infection and fibrosis progression [corrected].

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FHEPR.12247

Impaired IRS-1/PI3-kinase signaling in patients with HCV: a mechanism for increased prevalence of type 2 diabetes

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FHEPR.12247

Hepatitis C virus infection and diabetes: direct involvement of the virus in the development of insulin resistance

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FHEPR.12247

Hepatitis C Virus Down-Regulates Insulin Receptor Substrates 1 and 2 through Up-Regulation of Suppressor of Cytokine Signaling 3

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FHEPR.12247

The metabolic regulator PGC-1α links hepatitis C virus infection to hepatic insulin resistance.

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FHEPR.12247

SOCS-3 is an insulin-induced negative regulator of insulin signaling

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FHEPR.12247

Suppressor of cytokine signaling 1 (SOCS-1) and SOCS-3 cause insulin resistance through inhibition of tyrosine phosphorylation of insulin receptor substrate proteins by discrete mechanisms

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FHEPR.12247

IRS-1-mediated inhibition of insulin receptor tyrosine kinase activity in TNF-alpha- and obesity-induced insulin resistance

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FHEPR.12247

Phosphorylation of Ser307 in insulin receptor substrate-1 blocks interactions with the insulin receptor and inhibits insulin action

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FHEPR.12247

Irs1 serine 307 promotes insulin sensitivity in mice

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FHEPR.12247

PTPs versus PTKs: the redox side of the coin

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FHEPR.12247

Redox redux: revisiting PTPs and the control of cell signaling

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FHEPR.12247

Phosphorylation of IRS proteins, insulin action, and insulin resistance

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FHEPR.12247

Reactive oxygen species have a causal role in multiple forms of insulin resistance

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FHEPR.12247

Hepatitis C virus core protein inhibits microsomal triglyceride transfer protein activity and very low density lipoprotein secretion: a model of viral-related steatosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FHEPR.12247

Critical role of PA28 in hepatitis C virus-associated steatogenesis and hepatocarcinogenesis

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FHEPR.12247

Hepatic iron overload induces hepatocellular carcinoma in transgenic mice expressing the hepatitis C virus polyprotein

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FHEPR.12247

Insertion of specific bases during DNA synthesis past the oxidation-damaged base 8-oxodG

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FHEPR.12247

Hepatic oxidative DNA damage correlates with iron overload in chronic hepatitis C patients.

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FHEPR.12247

Balancing acts: molecular control of mammalian iron metabolism

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FHEPR.12247

LEAP-1, a novel highly disulfide-bonded human peptide, exhibits antimicrobial activity

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FHEPR.12247

Hepcidin, a urinary antimicrobial peptide synthesized in the liver

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FHEPR.12247

Hepcidin expression in the liver: relatively low level in patients with chronic hepatitis C

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FHEPR.12247

Reduced serum hepcidin levels in patients with chronic hepatitis C.

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FHEPR.12247

Hepatitis C virus-induced reactive oxygen species raise hepatic iron level in mice by reducing hepcidin transcription

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FHEPR.12247

Expression and function of C/EBP homology protein (GADD153) in podocytes

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FHEPR.12247

Hepatitis C virus-induced oxidative stress suppresses hepcidin expression through increased histone deacetylase activity

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FHEPR.12247

Retraction statement: Disruption of cyclophilin D-mediated calcium transfer from the endoplasmic reticulum to mitochondria contributes to hepatic endoplasmic reticulum stress and insulin resistance

21 January 2018

1 reference

3 January 2024

https://opencitations.net/index/api/v1/citations/10.1002/HEP.26189

Identifiers

DOI

10.1111/HEPR.12247

1 reference

29 August 2017

1 reference