(Q38261194)

1 September 2017

1 reference

title

IL-1β dependent cerebellar synaptopathy in a mouse mode of multiple sclerosis (English)

1 reference

1 September 2017

0 references

Antonietta Gentile

2

0 references

Georgia Mandolesi

object named as

Georgia Mandolesi

series ordinal

1

0 references

Diego Centonze

object named as

Diego Centonze

series ordinal

4

0 references

Alessandra Musella

object named as

Alessandra Musella

series ordinal

3

0 references

publication date

1 February 2015

1 reference

1 September 2017

1 reference

1 September 2017

volume

14

1 reference

1 September 2017

issue

1

1 reference

1 September 2017

page(s)

19-22

1 reference

https://scigraph.springernature.com/pub.10.1007/s12311-014-0613-0

1 September 2017

exact match

0 references

1 reference

https://api.crossref.org/works/10.1007%2FS12311-014-0613-0

Measurement and clinical effect of grey matter pathology in multiple sclerosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS12311-014-0613-0

The clinical impact of cerebellar grey matter pathology in multiple sclerosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS12311-014-0613-0

A channelopathy contributes to cerebellar dysfunction in a model of multiple sclerosis.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS12311-014-0613-0

An electrophysiologic approach to quantify impaired synaptic transmission and plasticity in experimental autoimmune encephalomyelitis.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS12311-014-0613-0

GABAergic signaling and connectivity on Purkinje cells are impaired in experimental autoimmune encephalomyelitis.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS12311-014-0613-0

Interleukin-1β alters glutamate transmission at purkinje cell synapses in a mouse model of multiple sclerosis.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS12311-014-0613-0

Inflammation subverts hippocampal synaptic plasticity in experimental multiple sclerosis.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS12311-014-0613-0

Inflammation triggers synaptic alteration and degeneration in experimental autoimmune encephalomyelitis

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS12311-014-0613-0

Interleukin-1β causes synaptic hyperexcitability in multiple sclerosis.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS12311-014-0613-0

Abnormal NMDA receptor function exacerbates experimental autoimmune encephalomyelitis

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS12311-014-0613-0

The link between inflammation, synaptic transmission and neurodegeneration in multiple sclerosis.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS12311-014-0613-0

Interleukin-1β promotes long-term potentiation in patients with multiple sclerosis.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS12311-014-0613-0

Impaired striatal GABA transmission in experimental autoimmune encephalomyelitis

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS12311-014-0613-0

Molecular mechanisms linking neuroinflammation and neurodegeneration in MS.

21 January 2018

1 reference

PubMed

https://pubmed.ncbi.nlm.nih.gov/25326653

inferred from PubMed ID database lookup

12 December 2020

based on heuristic

Identifiers

DOI

10.1007/S12311-014-0613-0

1 reference

Dimensions Publication ID

1 September 2017

1033297707

0 references

1 reference