(Q38274962)

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:25446182%20AND%20SRC:MED&resulttype=core&format=json

23 February 2020

review article

1 reference

title

The mitochondrial permeability transition pore and its role in myocardial ischemia reperfusion injury (English)

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:25446182%20AND%20SRC:MED&resulttype=core&format=json

23 February 2020

1 reference

1 reference

1

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:25446182%20AND%20SRC:MED&resulttype=core&format=json

23 February 2020

Derek Hausenloy

series ordinal

4

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:25446182%20AND%20SRC:MED&resulttype=core&format=json

23 February 2020

author name string

Parisa Samangouei

series ordinal

2

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:25446182%20AND%20SRC:MED&resulttype=core&format=json

23 February 2020

Siavash Beikoghli Kalkhoran

series ordinal

3

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:25446182%20AND%20SRC:MED&resulttype=core&format=json

23 February 2020

publication date

14 November 2014

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:25446182%20AND%20SRC:MED&resulttype=core&format=json

Journal of Molecular and Cellular Cardiology

23 February 2020

published in

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:25446182%20AND%20SRC:MED&resulttype=core&format=json

23 February 2020

volume

78

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:25446182%20AND%20SRC:MED&resulttype=core&format=json

23 February 2020

page(s)

23-34

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:25446182%20AND%20SRC:MED&resulttype=core&format=json

National trends in heart failure hospitalization after acute myocardial infarction for Medicare beneficiaries: 1998-2010.

23 February 2020

cites work

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Myocardial reperfusion injury.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Myocardial ischemia-reperfusion injury: a neglected therapeutic target.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

The mitochondrial permeability transition pore: its fundamental role in mediating cell death during ischaemia and reperfusion

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

The mitochondrial permeability transition pore as a target for preconditioning and postconditioning.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

The mitochondrial permeability transition pore and cyclophilin D in cardioprotection

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Cyclosporin A and cardioprotection: from investigative tool to therapeutic agent.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

The mitochondrial permeability transition: a current perspective on its identity and role in ischaemia/reperfusion injury.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

The Ca2+-induced membrane transition in mitochondria. I. The protective mechanisms

7 January 2021

2 references

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

7 January 2021

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

inferred from DOI database lookup

7 January 2021

based on heuristic

The Ca2+-induced membrane transition in mitochondria. III. Transitional Ca2+ release

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

The Ca2+-induced membrane transition in mitochondria

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

The reversible Ca2+-induced permeabilization of rat liver mitochondria

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Evidence for the presence of a reversible Ca2+-dependent pore activated by oxidative stress in heart mitochondria

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Kinetic evidence for a heart mitochondrial pore activated by Ca2+, inorganic phosphate and oxidative stress. A potential mechanism for mitochondrial dysfunction during cellular Ca2+ overload

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

The mitochondrial permeability transition pore

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Mitochondrial non-specific pores remain closed during cardiac ischaemia, but open upon reperfusion

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Inhibition of anoxia-induced injury in heart myocytes by cyclosporin A

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Protection by Cyclosporin A of ischemia/reperfusion-induced damage in isolated rat hearts.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Opening of the mitochondrial permeability transition pore causes depletion of mitochondrial and cytosolic NAD+ and is a causative event in the death of myocytes in postischemic reperfusion of the heart.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Mitochondrial ATP-sensitive potassium channels attenuate matrix Ca(2+) overload during simulated ischemia and reperfusion: possible mechanism of cardioprotection

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Real-time 2-photon imaging of mitochondrial function in perfused rat hearts subjected to ischemia/reperfusion

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Inhibiting mitochondrial permeability transition pore opening: a new paradigm for myocardial preconditioning?

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Inhibiting mitochondrial permeability transition pore opening at reperfusion protects against ischaemia-reperfusion injury

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Inhibition of mitochondrial permeability transition improves functional recovery and reduces mortality following acute myocardial infarction in mice

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Properties of the permeability transition pore in mitochondria devoid of Cyclophilin D.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Loss of cyclophilin D reveals a critical role for mitochondrial permeability transition in cell death

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Cyclophilin D-dependent mitochondrial permeability transition regulates some necrotic but not apoptotic cell death

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

The role of mitochondrial permeability transition in reperfusion-induced cardiomyocyte death depends on the duration of ischemia

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Translating novel strategies for cardioprotection: the Hatter Workshop Recommendations

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Translating cardioprotection for patient benefit: position paper from the Working Group of Cellular Biology of the Heart of the European Society of Cardiology.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

The ADP/ATP translocator is not essential for the mitochondrial permeability transition pore

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

The mitochondrial permeability transition pore may comprise VDAC molecules

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

The mitochondrial permeability transition pore may comprise VDAC molecules

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Voltage-dependent anion channels are dispensable for mitochondrial-dependent cell death

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Inhibition of Ca2(+)-induced large-amplitude swelling of liver and heart mitochondria by cyclosporin is probably caused by the inhibitor binding to mitochondrial-matrix peptidyl-prolyl cis-trans isomerase and preventing it interacting with the adeni

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Further evidence that cyclosporin A protects mitochondria from calcium overload by inhibiting a matrix peptidyl-prolyl cis-trans isomerase. Implications for the immunosuppressive and toxic effects of cyclosporin

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

In Saccharomyces cerevisiae, the phosphate carrier is a component of the mitochondrial unselective channel

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Genetic manipulation of the cardiac mitochondrial phosphate carrier does not affect permeability transition

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

The roles of phosphate and the phosphate carrier in the mitochondrial permeability transition pore

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Genetic deletion of the mitochondrial phosphate carrier desensitizes the mitochondrial permeability transition pore and causes cardiomyopathy

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Cyclophilin D modulates mitochondrial F0F1-ATP synthase by interacting with the lateral stalk of the complex

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Role of the c subunit of the FO ATP synthase in mitochondrial permeability transition

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Dimers of mitochondrial ATP synthase form the permeability transition pore

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Channel formation by yeast F-ATP synthase and the role of dimerization in the mitochondrial permeability transition

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

An uncoupling channel within the c-subunit ring of the F1FO ATP synthase is the mitochondrial permeability transition pore

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Bax and adenine nucleotide translocator cooperate in the mitochondrial control of apoptosis

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Bax interacts with the permeability transition pore to induce permeability transition and cytochrome c release in isolated mitochondria

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Bcl-2 family proteins regulate the release of apoptogenic cytochrome c by the mitochondrial channel VDAC

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Bax and Bak function as the outer membrane component of the mitochondrial permeability pore in regulating necrotic cell death in mice

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Cardioprotection techniques: preconditioning, postconditioning and remote conditioning (basic science).

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Regional ischemic 'preconditioning' protects remote virgin myocardium from subsequent sustained coronary occlusion

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Remote ischaemic preconditioning: underlying mechanisms and clinical application.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Preconditioning with ischemia: a delay of lethal cell injury in ischemic myocardium

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Ischaemic preconditioning inhibits opening of mitochondrial permeability transition pores in the reperfused rat heart

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Preconditioning protects by inhibiting the mitochondrial permeability transition

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Inhibition of myocardial injury by ischemic postconditioning during reperfusion: comparison with ischemic preconditioning

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Postconditioning inhibits mitochondrial permeability transition

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Mitochondrial Ca2+ uptake during simulated ischemia does not affect permeability transition pore opening upon simulated reperfusion

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Role of sarcoplasmic reticulum in mitochondrial permeability transition and cardiomyocyte death during reperfusion

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Opening of potassium channels protects mitochondrial function from calcium overload

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Preconditioning limits mitochondrial Ca(2+) during ischemia in rat hearts: role of K(ATP) channels

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Opening mitochondrial K(ATP) in the heart--what happens, and what does not happen

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

K(ATP) channels and preconditioning: a re-examination of the role of mitochondrial K(ATP) channels and an overview of alternative mechanisms

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Mitochondrial K(ATP) channels in cell survival and death

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Reactive oxygen species, but not Ca2+ overloading, trigger pH- and mitochondrial permeability transition-dependent death of adult rat myocytes after ischemia-reperfusion

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

The physiological role of mitochondrial calcium revealed by mice lacking the mitochondrial calcium uniporter.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Depressing mitochondria-reticulum interactions protects cardiomyocytes from lethal hypoxia-reoxygenation injury.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Ischemic preconditioning decreases oxidative stress during reperfusion: a chemiluminescence study

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Preconditioning in cardiomyocytes protects by attenuating oxidant stress at reperfusion

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Ischemic preconditioning decreases the reperfusion-related formation of hydroxyl radicals in a rabbit model of regional myocardial ischemia and reperfusion: the role of K(ATP) channels

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Inhibition of mitochondrial permeability transition pore opening by ischemic preconditioning is probably mediated by reduction of oxidative stress rather than mitochondrial protein phosphorylation.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Hypoxic postconditioning reduces cardiomyocyte loss by inhibiting ROS generation and intracellular Ca2+ overload

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

The role of oxidized cytochrome c in regulating mitochondrial reactive oxygen species production and its perturbation in ischaemia

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Ischaemic accumulation of succinate controls reperfusion injury through mitochondrial ROS.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Ischemic preconditioning slows energy metabolism and delays ultrastructural damage during a sustained ischemic episode

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Ischemic preconditioning preserves creatine phosphate and intracellular pH

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Effect of ischemic preconditioning on mitochondrial oxidative phosphorylation and high energy phosphates in rat hearts

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Preconditioning preserves mitochondrial function and glycolytic flux during an early period of reperfusion in perfused rat hearts

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Ischemic preconditioning in rats: role of mitochondrial K(ATP) channel in preservation of mitochondrial function

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Cardioprotection by ischemic preconditioning preserves mitochondrial function and functional coupling between adenine nucleotide translocase and creatine kinase.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Did a classic preconditioning study provide a clue to the identity of the mitochondrial permeability transition pore?

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

The pH hypothesis of postconditioning: staccato reperfusion reintroduces oxygen and perpetuates myocardial acidosis

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Mechanism of preconditioning. Ionic alterations

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Loss of myocardial protection after preconditioning correlates with the time course of glycogen recovery within the preconditioned segment.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Role of Na(+)/H(+) exchanger during ischemia and preconditioning in the isolated rat heart

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Activity of the Na(+)/H(+) exchanger is critical to reperfusion damage and preconditioning in the isolated rat heart

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Na+/H+ exchange in ischemia, reperfusion and preconditioning

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Protein kinase B/Akt phosphorylates and inhibits the cardiac Na+/H+ exchanger NHE1.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Staged reperfusion attenuates myocardial stunning in dogs. Role of transient acidosis during early reperfusion

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Prolonged transient acidosis during early reperfusion contributes to the cardioprotective effects of postconditioning

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

New directions for protecting the heart against ischaemia-reperfusion injury: targeting the Reperfusion Injury Salvage Kinase (RISK)-pathway

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Reperfusion injury salvage kinase signalling: taking a RISK for cardioprotection.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Activation of the protective Survivor Activating Factor Enhancement (SAFE) pathway against reperfusion injury: Does it go beyond the RISK pathway?

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Reperfusion injury salvage kinase and survivor activating factor enhancement prosurvival signaling pathways in ischemic postconditioning: two sides of the same coin

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

The reperfusion injury salvage kinase pathway: a common target for both ischemic preconditioning and postconditioning.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Remote postconditioning by humoral factors in effluent from ischemic preconditioned rat hearts is mediated via PI3K/Akt-dependent cell-survival signaling at reperfusion

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

PI 3-kinase regulates the mitochondrial transition pore in controlled reperfusion and postconditioning

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Signalling via the reperfusion injury signalling kinase (RISK) pathway links closure of the mitochondrial permeability transition pore to cardioprotection

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Rapid accumulation of Akt in mitochondria following phosphatidylinositol 3-kinase activation

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Mitochondrial PKCepsilon and MAPK form signaling modules in the murine heart: enhanced mitochondrial PKCepsilon-MAPK interactions and differential MAPK activation in PKCepsilon-induced cardioprotection

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Protein kinase G transmits the cardioprotective signal from cytosol to mitochondria

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

7 January 2021

The renaissance of GSK3

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Phosphorylation of glycogen synthase kinase-3beta during preconditioning through a phosphatidylinositol-3-kinase--dependent pathway is cardioprotective

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Inhibition of GSK3beta by postconditioning is required to prevent opening of the mitochondrial permeability transition pore during reperfusion

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Glycogen synthase kinase-3 inactivation is not required for ischemic preconditioning or postconditioning in the mouse

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Glycogen synthase kinase-3beta mediates convergence of protection signaling to inhibit the mitochondrial permeability transition pore

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Modulation of the mitochondrial permeability transition pore complex in GSK-3beta-mediated myocardial protection

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Glycogen synthase kinase 3 inhibition slows mitochondrial adenine nucleotide transport and regulates voltage-dependent anion channel phosphorylation

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Translocation of glycogen synthase kinase-3β (GSK-3β), a trigger of permeability transition, is kinase activity-dependent and mediated by interaction with voltage-dependent anion channel 2 (VDAC2).

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Mitochondrial hexokinase and cardioprotection of the intact heart

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Targeting hexokinase II to mitochondria to modulate energy metabolism and reduce ischaemia-reperfusion injury in heart

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

TNFalpha is required to confer protection in an in vivo model of classical ischaemic preconditioning

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Dual activation of STAT-3 and Akt is required during the trigger phase of ischaemic preconditioning

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Ischaemic postconditioning protects against reperfusion injury via the SAFE pathway

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Leptin, the obesity-associated hormone, exhibits direct cardioprotective effects

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Inhibition of permeability transition pore opening by mitochondrial STAT3 and its role in myocardial ischemia/reperfusion

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Function of mitochondrial Stat3 in cellular respiration

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Mitochondrial morphology and cardiovascular disease

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Mitochondrial dynamics in cardiovascular health and disease

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

The shape of things to come: mitochondrial fusion and fission in the adult heart

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Mitochondrial fusion and fission proteins: novel therapeutic targets for combating cardiovascular disease.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Inhibiting mitochondrial fission protects the heart against ischemia/reperfusion injury

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Reversible phosphorylation of Drp1 by cyclic AMP-dependent protein kinase and calcineurin regulates mitochondrial fission and cell death

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Cyclic AMP-dependent protein kinase phosphorylation of Drp1 regulates its GTPase activity and mitochondrial morphology.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Insulin stimulates mitochondrial fusion and function in cardiomyocytes via the Akt-mTOR-NFκB-Opa-1 signaling pathway

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Nitrite activates protein kinase A in normoxia to mediate mitochondrial fusion and tolerance to ischaemia/reperfusion.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Preconditioning tachycardia decreases the activity of the mitochondrial permeability transition pore in the dog heart

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Transient mitochondrial permeability transition pore opening mediates preconditioning-induced protection

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Mitochondria are excitable organelles capable of generating and conveying electrical and calcium signals.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

From calcium signaling to cell death: two conformations for the mitochondrial permeability transition pore. Switching from low- to high-conductance state

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Electrical coupling and plasticity of the mitochondrial network

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Transient and long-lasting openings of the mitochondrial permeability transition pore can be monitored directly in intact cells by changes in mitochondrial calcein fluorescence

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Diazoxide opens the mitochondrial permeability transition pore and alters Ca2+ transients in rat ventricular myocytes

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Transient opening of mitochondrial permeability transition pore by reactive oxygen species protects myocardium from ischemia-reperfusion injury

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Protective role of transient pore openings in calcium handling by cardiac mitochondria

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Cyclophilin D controls mitochondrial pore-dependent Ca(2+) exchange, metabolic flexibility, and propensity for heart failure in mice

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

CypD(-/-) hearts have altered levels of proteins involved in Krebs cycle, branch chain amino acid degradation and pyruvate metabolism

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Cyclophilin D modulates mitochondrial acetylome

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Enhancement of anxiety, facilitation of avoidance behavior, and occurrence of adult-onset obesity in mice lacking mitochondrial cyclophilin D

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Mitochondria-associated endoplasmic reticulum membrane (MAM) integrity is required for insulin signaling and is implicated in hepatic insulin resistance

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Interaction of cardiovascular risk factors with myocardial ischemia/reperfusion injury, preconditioning, and postconditioning.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Interaction of risk factors, comorbidities, and comedications with ischemia/reperfusion injury and cardioprotection by preconditioning, postconditioning, and remote conditioning

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Cyclosporine A administered during reperfusion fails to restore cardioprotection in prediabetic Zucker obese rats in vivo

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Dysfunctional survival-signaling and stress-intolerance in aged murine and human myocardium.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Protection of myocardium by cyclosporin a and insulin: in vitro simulated ischemia study in human myocardium

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Mitochondrial permeability transition pore as a target for cardioprotection in the human heart

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

The mitochondrial permeability transition pore as a target for cardioprotection in hypertrophic cardiomyopathy.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Effect of cyclosporine on reperfusion injury in acute myocardial infarction

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Effect of cyclosporine on left ventricular remodeling after reperfused myocardial infarction

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Postconditioning and protection from reperfusion injury: where do we stand? Position paper from the Working Group of Cellular Biology of the Heart of the European Society of Cardiology

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

The effect of prethrombolytic cyclosporine-A injection on clinical outcome of acute anterior ST-elevation myocardial infarction

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

The effect of cyclosporin-A on peri-operative myocardial injury in adult patients undergoing coronary artery bypass graft surgery: a randomised controlled clinical trial

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Cyclosporine protects the heart during aortic valve surgery.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Inhibition of mitochondrial permeability transition to prevent the post-cardiac arrest syndrome: a pre-clinical study

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Cyclosporine treatment improves cardiac function and systemic hemodynamics during resuscitation in a newborn piglet model of asphyxia: a dose-response study.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Preconditioning delays Ca2+-induced mitochondrial permeability transition

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Specific inhibition of the mitochondrial permeability transition prevents lethal reperfusion injury

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

Mitochondrial targeting of cyclosporin A enables selective inhibition of cyclophilin-D and enhanced cytoprotection after glucose and oxygen deprivation

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005

TRO40303, a new cardioprotective compound, inhibits mitochondrial permeability transition.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.11.005