(Q38295649)

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Altered myocardial gene expression reveals possible maladaptive processes in heterozygous and homozygous cardiac myosin-binding protein C knockout mice

scientific article published on 03 December 2007

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Europe PubMed Central

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1 September 2017

Altered myocardial gene expression reveals possible maladaptive processes in heterozygous and homozygous cardiac myosin-binding protein C knockout mice (English)

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PubMed publication ID

1 September 2017

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Eijssen LM

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van den Bosch BJ

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Vignier N

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1 September 2017

Lindsey PJ

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1 September 2017

van den Burg CM

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Carrier L

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Doevendans PA

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van der Vusse GJ

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Smeets HJ

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3 December 2007

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1 September 2017

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1

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52-60

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1 September 2017

Molecular genetics of familial hypertrophic cardiomyopathy (FHC).

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https://api.crossref.org/works/10.1016%2FJ.YGENO.2007.09.005

7 January 2021

Mutations in the gene for cardiac myosin-binding protein C and late-onset familial hypertrophic cardiomyopathy

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Familial hypertrophic cardiomyopathy: from mutations to functional defects

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The 2373insG mutation in the MYBPC3 gene is a founder mutation, which accounts for nearly one-fourth of the HCM cases in the Netherlands

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Hypertrophic cardiomyopathy: distribution of disease genes, spectrum of mutations, and implications for a molecular diagnosis strategy

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Myosin binding protein C, a potential regulator of cardiac contractility

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Cardiac myosin binding protein C: its role in physiology and disease

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Novel splice donor site mutation in the cardiac myosin-binding protein-C gene in familial hypertrophic cardiomyopathy. Characterization Of cardiac transcript and protein

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A newly created splice donor site in exon 25 of the MyBP-C gene is responsible for inherited hypertrophic cardiomyopathy with incomplete disease penetrance

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Asymmetric septal hypertrophy in heterozygous cMyBP-C null mice

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Hypertrophic cardiomyopathy in cardiac myosin binding protein-C knockout mice

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Dilated cardiomyopathy in homozygous myosin-binding protein-C mutant mice

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Comparison of two murine models of familial hypertrophic cardiomyopathy

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A mouse model of myosin binding protein C human familial hypertrophic cardiomyopathy

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Phenotypic deficits in mice expressing a myosin binding protein C lacking the titin and myosin binding domains

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In vivo modeling of myosin binding protein C familial hypertrophic cardiomyopathy

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Length and protein kinase A modulations of myocytes in cardiac myosin binding protein C-deficient mice

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Genome-wide expression profiling of mid-gestation placenta and embryo using a 15,000 mouse developmental cDNA microarray

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GenMAPP, a new tool for viewing and analyzing microarray data on biological pathways

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MAPPFinder: using Gene Ontology and GenMAPP to create a global gene-expression profile from microarray data

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PubMatrix: a tool for multiplex literature mining

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Early and transient gene expression changes in pressure overload-induced cardiac hypertrophy in mice

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Fatty acid homeostasis in the normoxic and ischemic heart

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Cardiac hypertrophy: the good, the bad, and the ugly

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Energy metabolism in the normal and failing heart: potential for therapeutic interventions

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Metabolic remodelling of the failing heart: the cardiac burn-out syndrome?

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The molecular basis of myocardial hypertrophy and heart failure

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Molecular interplay between mechanical and humoral signalling in cardiac hypertrophy

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Four and a half LIM protein 1 binds myosin-binding protein C and regulates myosin filament formation and sarcomere assembly

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Mixed messages: transcription patterns in failing and recovering human myocardium.

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Molecular determinants of myocardial hypertrophy and failure: alternative pathways for beneficial and maladaptive hypertrophy

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Protein turnover in cardiac cell growth and survival

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Impairment of the ubiquitin-proteasome system by truncated cardiac myosin binding protein C mutants

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Organization and regulation of mitogen-activated protein kinase signaling pathways

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Adaptive and maladaptive hypertrophic pathways: points of convergence and divergence

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Myocyte hypertrophy and apoptosis: a balancing act.

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Genotype–phenotype correlations in familial hypertrophic cardiomyopathy A comparison between mutations in the cardiac protein-C and the beta-myosin heavy chain genes

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Clinical features and prognostic implications of familial hypertrophic cardiomyopathy related to the cardiac myosin-binding protein C gene

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Minimum information about a microarray experiment (MIAME)-toward standards for microarray data

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Effects of atmospheric ozone on microarray data quality

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7 January 2021

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10.1016/J.YGENO.2007.09.005

1 reference

Europe PubMed Central

PubMed publication ID

1 September 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

1 September 2017

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