(Q38737214)

6 September 2017

title

Mitochondrial involvement in myocyte death and heart failure (English)

1 reference

6 September 2017

Michael J Goldenthal

1

1 reference

6 September 2017

publication date

17 February 2016

1 reference

6 September 2017

published in

Heart Failure Reviews

1 reference

6 September 2017

volume

21

1 reference

6 September 2017

issue

2

1 reference

6 September 2017

page(s)

137-155

1 reference

https://scigraph.springernature.com/pub.10.1007/s10741-016-9531-1

6 September 2017

exact match

0 references

cites work

Mechanisms of myocardial regeneration

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

A mechanistic role for cardiac myocyte apoptosis in heart failure

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Ca2+- and mitochondrial-dependent cardiomyocyte necrosis as a primary mediator of heart failure

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Striking a balance: autophagy, apoptosis, and necrosis in a normal and failing heart.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Autophagy: an affair of the heart

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Cell biology. Metabolic control of cell death

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Mechanisms of cell death in heart disease

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Bax and Bak function as the outer membrane component of the mitochondrial permeability pore in regulating necrotic cell death in mice

21 January 2018

2 references

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

21 January 2018

12 December 2020

A time to reap, a time to sow: mitophagy and biogenesis in cardiac pathophysiology

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Cell death in the myocardium: my heart won't go on.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Death receptors: signaling and modulation

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Fas preassociation required for apoptosis signaling and dominant inhibition by pathogenic mutations

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

A novel protein that interacts with the death domain of Fas/APO1 contains a sequence motif related to the death domain

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Involvement of MACH, a novel MORT1/FADD-interacting protease, in Fas/APO-1- and TNF receptor-induced cell death

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

An induced proximity model for caspase-8 activation.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Pro-caspase-3 is a major physiologic target of caspase-8.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Bioenergetics and cell death.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Endonuclease G is an apoptotic DNase when released from mitochondria

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Molecular characterization of mitochondrial apoptosis-inducing factor

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Three-dimensional structure of the apoptosome: implications for assembly, procaspase-9 binding, and activation.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Induction of apoptotic program in cell-free extracts: requirement for dATP and cytochrome c

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Cell death: critical control points

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Mitochondrial regulation of cell death.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

X-ray and NMR structure of human Bcl-xL, an inhibitor of programmed cell death

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Inhibition of Bax channel-forming activity by Bcl-2

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Bax oligomerization is required for channel-forming activity in liposomes and to trigger cytochrome c release from mitochondria

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

How do BCL-2 proteins induce mitochondrial outer membrane permeabilization?

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Bax activation initiates the assembly of a multimeric catalyst that facilitates Bax pore formation in mitochondrial outer membranes

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

A new view of the lethal apoptotic pore

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Proapoptotic BAX and BAK: a requisite gateway to mitochondrial dysfunction and death

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Involvement of cardiolipin in tBID-induced activation of BAX during apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

BAD: undertaker by night, candyman by day.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Direct activation of Bax by p53 mediates mitochondrial membrane permeabilization and apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

The coordinate release of cytochrome c during apoptosis is rapid, complete and kinetically invariant

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

An intracellular wave of cytochrome c propagates and precedes Bax redistribution during apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Spatial and temporal dynamics of mitochondrial membrane permeability waves during apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Cell death by necrosis, a regulated way to go.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

A heart mitochondrial Ca2(+)-dependent pore of possible relevance to re-perfusion-induced injury. Evidence that ADP facilitates pore interconversion between the closed and open states

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

The role of reactive oxygen species in mitochondrial permeability transition

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

The mitochondrial permeability transition in cell death: a common mechanism in necrosis, apoptosis and autophagy

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Mitochondrial boundary membrane contact sites in brain: points of hexokinase and creatine kinase location, and control of Ca2+ transport

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Mitochondrial function as a determinant of recovery or death in cell response to injury.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Pore formation and uncoupling initiate a Ca2+-independent degradation of mitochondrial phospholipids

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Mitochondrial function in ischaemia and reperfusion of the ageing heart

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

The Ca2+-induced membrane transition in mitochondria

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Further evidence that cyclosporin A protects mitochondria from calcium overload by inhibiting a matrix peptidyl-prolyl cis-trans isomerase. Implications for the immunosuppressive and toxic effects of cyclosporin

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Recruitment of mitochondrial cyclophilin to the mitochondrial inner membrane under conditions of oxidative stress that enhance the opening of a calcium-sensitive non-specific channel

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Mitochondrial non-specific pores remain closed during cardiac ischaemia, but open upon reperfusion

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

The Ca2+-induced membrane transition in mitochondria. III. Transitional Ca2+ release

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Inhibition of Ca2(+)-induced large-amplitude swelling of liver and heart mitochondria by cyclosporin is probably caused by the inhibitor binding to mitochondrial-matrix peptidyl-prolyl cis-trans isomerase and preventing it interacting with the adeni

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Electrical currents associated with nucleotide transport by the reconstituted mitochondrial ADP/ATP carrier

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Recent progress in elucidating the molecular mechanism of the mitochondrial permeability transition pore.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Mitochondrial ATP synthasome: three-dimensional structure by electron microscopy of the ATP synthase in complex formation with carriers for Pi and ADP/ATP.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

The mitochondrial permeability transition pore may comprise VDAC molecules

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Hexokinase II and reperfusion injury: TAT-HK2 peptide impairs vascular function in Langendorff-perfused rat hearts.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Complexes between porin, hexokinase, mitochondrial creatine kinase and adenylate translocator display properties of the permeability transition pore. Implication for regulation of permeability transition by the kinases

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

The ADP/ATP translocator is not essential for the mitochondrial permeability transition pore

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Voltage-dependent anion channels are dispensable for mitochondrial-dependent cell death

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Properties of the permeability transition in VDAC1(-/-) mitochondria

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Regulation of the mitochondrial permeability transition pore by the outer membrane does not involve the peripheral benzodiazepine receptor (Translocator Protein of 18 kDa (TSPO)).

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Patch clamp investigation into the phosphate carrier from Saccharomyces cerevisiae mitochondria.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Genetic deletion of the mitochondrial phosphate carrier desensitizes the mitochondrial permeability transition pore and causes cardiomyopathy

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Genetic manipulation of the cardiac mitochondrial phosphate carrier does not affect permeability transition

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Loss of cyclophilin D reveals a critical role for mitochondrial permeability transition in cell death

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Properties of the permeability transition pore in mitochondria devoid of Cyclophilin D.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Cyclophilin D is a component of mitochondrial permeability transition and mediates neuronal cell death after focal cerebral ischemia

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Activation of mitochondrial ERK protects cancer cells from death through inhibition of the permeability transition

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Cysteine 203 of cyclophilin D is critical for cyclophilin D activation of the mitochondrial permeability transition pore

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Cyclophilin D interacts with Bcl2 and exerts an anti-apoptotic effect

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Cyclophilin D modulates mitochondrial F0F1-ATP synthase by interacting with the lateral stalk of the complex

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

The oligomycin-sensitivity conferring protein of mitochondrial ATP synthase: emerging new roles in mitochondrial pathophysiology

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Dimers of mitochondrial ATP synthase form the permeability transition pore

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Role of the c subunit of the FO ATP synthase in mitochondrial permeability transition

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

An uncoupling channel within the c-subunit ring of the F1FO ATP synthase is the mitochondrial permeability transition pore

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Channel formation by yeast F-ATP synthase and the role of dimerization in the mitochondrial permeability transition

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Phosphorylation of a peptide related to subunit c of the F0F1-ATPase/ATP synthase and relationship to permeability transition pore opening in mitochondria

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Cell death disguised: The mitochondrial permeability transition pore as the c-subunit of the F(1)F(O) ATP synthase

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Molecular mechanisms of cell death: central implication of ATP synthase in mitochondrial permeability transition

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

What is the mitochondrial permeability transition pore?

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

The C Ring of the F1Fo ATP Synthase Forms the Mitochondrial Permeability Transition Pore: A Critical Appraisal

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

From ATP to PTP and Back: A Dual Function for the Mitochondrial ATP Synthase

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

The mitochondrial permeability transition pore: a mystery solved?

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

The mitochondrial permeability transition from in vitro artifact to disease target

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Regulation of the permeability transition pore in skeletal muscle mitochondria. Modulation By electron flow through the respiratory chain complex i.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Regulation of the inner membrane mitochondrial permeability transition by the outer membrane translocator protein (peripheral benzodiazepine receptor)

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Shedding light on the mitochondrial permeability transition.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Bax interacts with the permeability transition pore to induce permeability transition and cytochrome c release in isolated mitochondria

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Bax and adenine nucleotide translocator cooperate in the mitochondrial control of apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Bax regulates primary necrosis through mitochondrial dynamics

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Bad targets the permeability transition pore independent of Bax or Bak to switch between Ca2+-dependent cell survival and death

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Bcl-2 and Bax regulate the channel activity of the mitochondrial adenine nucleotide translocator

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Mediation of the antiapoptotic activity of Bcl-xL protein upon interaction with VDAC1 protein

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Bcl-xL regulates metabolic efficiency of neurons through interaction with the mitochondrial F1FO ATP synthase.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Bcl-xL regulates mitochondrial energetics by stabilizing the inner membrane potential

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

The role of hexokinase in cardioprotection - mechanism and potential for translation

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Cyclophilin D-dependent mitochondrial permeability transition regulates some necrotic but not apoptotic cell death

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Bcl-xL regulates the membrane potential and volume homeostasis of mitochondria

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Cytochrome c is released in a single step during apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Cytochrome c maintains mitochondrial transmembrane potential and ATP generation after outer mitochondrial membrane permeabilization during the apoptotic process

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Preservation of mitochondrial structure and function after Bid- or Bax-mediated cytochrome c release

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Apoptosis predominates in nonmyocytes in heart failure

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Cardiomyocyte apoptosis in the failing heart--a critical review from definition and classification of cell death

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Molecular mechanisms that differentiate apoptosis from programmed necrosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Mitochondrial morphology in mitophagy and macroautophagy

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Bcl-2 family members: dual regulators of apoptosis and autophagy

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Mitochondrial pruning by Nix and BNip3: an essential function for cardiac-expressed death factors

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Inhibition of ischemic cardiomyocyte apoptosis through targeted ablation of Bnip3 restrains postinfarction remodeling in mice

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Myocytes die by multiple mechanisms in failing human hearts

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

On the nature of cell death during remodeling of hypertrophied human myocardium.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Autophagy in chronically ischemic myocardium.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Autophagic cardiomyocyte death in cardiomyopathic hamsters and its prevention by granulocyte colony-stimulating factor

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Degradation systems in heart failure

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Autophagy in load-induced heart disease

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

The origin of the autophagosomal membrane

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Regulation of autophagy by ROS: physiology and pathology

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

The AMPK signalling pathway coordinates cell growth, autophagy and metabolism

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Phosphorylation of ULK1 (hATG1) by AMP-activated protein kinase connects energy sensing to mitophagy

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Preconditioning involves selective mitophagy mediated by Parkin and p62/SQSTM1.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Distinct roles of autophagy in the heart during ischemia and reperfusion: roles of AMP-activated protein kinase and Beclin 1 in mediating autophagy.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

The interplay between cell death signaling pathways in the heart

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Proteasome and p97 mediate mitophagy and degradation of mitofusins induced by Parkin

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Fission and selective fusion govern mitochondrial segregation and elimination by autophagy

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

The mitochondrial protein hFis1 regulates mitochondrial fission in mammalian cells through an interaction with the dynamin-like protein DLP1

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Impaired OMA1-dependent cleavage of OPA1 and reduced DRP1 fission activity combine to prevent mitophagy in cells that are dependent on oxidative phosphorylation

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Oma1, a novel membrane-bound metallopeptidase in mitochondria with activities overlapping with the m-AAA protease

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Mitochondrial function: OMA1 and OPA1, the grandmasters of mitochondrial health

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

How mitochondrial dynamism orchestrates mitophagy

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Molecular mechanisms mediating mitochondrial dynamics and mitophagy and their functional roles in the cardiovascular system

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Mitochondrial quality control: Easy come, easy go

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

The failing heart--an engine out of fuel

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Mitochondrial dysfunction in heart failure

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Bioenergetics of the failing heart

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Mitochondrial centrality in heart failure

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Harnessing the Power of Integrated Mitochondrial Biology and Physiology: A Special Report on the NHLBI Mitochondria in Heart Diseases Initiative

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Energy metabolic reprogramming in the hypertrophied and early stage failing heart: a multisystems approach

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Maintaining ancient organelles: mitochondrial biogenesis and maturation

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Myocardial energetics in heart failure

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Matrix revisited: mechanisms linking energy substrate metabolism to the function of the heart

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Rethinking cardiac metabolism: metabolic cycles to refuel and rebuild the failing heart

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Mitochondrial protein phosphorylation as a regulatory modality: implications for mitochondrial dysfunction in heart failure.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Exogenous NAD blocks cardiac hypertrophic response via activation of the SIRT3-LKB1-AMP-activated kinase pathway

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Mitochondrial complex I deficiency increases protein acetylation and accelerates heart failure

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Mitochondrial outer membrane permeabilization during apoptosis: the innocent bystander scenario

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Non-apoptotic functions of apoptosis-regulatory proteins

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Cytochrome c deficiency causes embryonic lethality and attenuates stress-induced apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Loss of Aif function causes cell death in the mouse embryo, but the temporal progression of patterning is normal

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

AIF deficiency compromises oxidative phosphorylation

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Muscle-specific loss of apoptosis-inducing factor leads to mitochondrial dysfunction, skeletal muscle atrophy, and dilated cardiomyopathy

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Dissociating the dual roles of apoptosis-inducing factor in maintaining mitochondrial structure and apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Cristae formation-linking ultrastructure and function of mitochondria

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Macromolecular organization of ATP synthase and complex I in whole mitochondria.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Is there a relationship between the supramolecular organization of the mitochondrial ATP synthase and the formation of cristae?

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

The ATP synthase is involved in generating mitochondrial cristae morphology

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Human F1F0 ATP synthase, mitochondrial ultrastructure and OXPHOS impairment: a (super-)complex matter?

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Mitochondrial calcium and the regulation of metabolism in the heart

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

The ins and outs of mitochondrial calcium.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

The Mitochondrial Permeability Transition Pore: Channel Formation by F-ATP Synthase, Integration in Signal Transduction, and Role in Pathophysiology.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Mitochondrial permeability transition pore and calcium handling

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Molecular identity of the mitochondrial permeability transition pore and its role in ischemia-reperfusion injury

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Role of the mitochondrial permeability transition in myocardial disease

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

A forty-kilodalton protein of the inner membrane is the mitochondrial calcium uniporter

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Integrative genomics identifies MCU as an essential component of the mitochondrial calcium uniporter

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

The physiological role of mitochondrial calcium revealed by mice lacking the mitochondrial calcium uniporter.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

The mitochondrial permeability transition pore is a dispensable element for mitochondrial calcium efflux

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Clinical applications of coenzyme Q10.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Mitochondria-targeted agents: Future perspectives of mitochondrial pharmaceutics in cardiovascular diseases

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Coenzyme Q10 treatment of cardiovascular disorders of ageing including heart failure, hypertension and endothelial dysfunction

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Coenzyme Q10 for the treatment of heart failure: a review of the literature

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

The effect of coenzyme Q10 on morbidity and mortality in chronic heart failure: results from Q-SYMBIO: a randomized double-blind trial

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Three-year survival of patients with heart failure caused by dilated cardiomyopathy and L-carnitine administration

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

L-carnitine treatment in patients with mild diastolic heart failure is associated with improvement in diastolic function and symptoms

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

A modern approach to the treatment of mitochondrial disease

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Targeting antioxidants to mitochondria by conjugation to lipophilic cations.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Selective targeting of a redox-active ubiquinone to mitochondria within cells: antioxidant and antiapoptotic properties

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Delivery of bioactive molecules to mitochondria in vivo.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Mitochondria-targeted antioxidants

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Mitochondria-Targeted Antioxidant MitoQ 10 Improves Endothelial Function and Attenuates Cardiac Hypertrophy

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Animal and human studies with the mitochondria-targeted antioxidant MitoQ.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Serendipity and the discovery of novel compounds that restore mitochondrial plasticity

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Mitochondrial permeability transition pore opening as a promising therapeutic target in cardiac diseases

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Identification of a novel compound that inhibits both mitochondria-mediated necrosis and apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Protection of hearts from reperfusion injury by propofol is associated with inhibition of the mitochondrial permeability transition.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Antioxidant MCI-186 inhibits mitochondrial permeability transition pore and upregulates Bcl-2 expression

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Preconditioning and postconditioning: the essential role of the mitochondrial permeability transition pore

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Preconditioning delays Ca2+-induced mitochondrial permeability transition

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Inhibiting mitochondrial permeability transition pore opening: a new paradigm for myocardial preconditioning?

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Targeting mitochondria for cardioprotection: examining the benefit for patients.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Anesthetic cardioprotection in relation to mitochondria: basic science

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Multiplicity of effectors of the cardioprotective agent, diazoxide

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Antiapoptotic effect of nicorandil mediated by mitochondrial atp-sensitive potassium channels in cultured cardiac myocytes

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Effect of nicorandil on coronary events in patients with stable angina: the Impact Of Nicorandil in Angina (IONA) randomised trial

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Effect of nicorandil in patients with heart failure: a systematic review and meta-analysis

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Effects of oral nicorandil therapy on sympathetic nerve activity and cardiac events in patients with chronic heart failure: subanalysis of our previous report using propensity score matching

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Essential versus accessory aspects of cell death: recommendations of the NCCD 2015.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

The mitochondrial permeability transition pore may comprise VDAC molecules

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10741-016-9531-1

Crucial role of sulfhydryl groups in the mitochondrial inner membrane structure

21 January 2018

1 reference

12 December 2020

Cytotoxicity-dependent APO-1 (Fas/CD95)-associated proteins form a death-inducing signaling complex (DISC) with the receptor

1 reference

12 December 2020

Antioxidant supplements for prevention of mortality in healthy participants and patients with various diseases

1 reference

12 December 2020

Kinetic evidence for a heart mitochondrial pore activated by Ca2+, inorganic phosphate and oxidative stress. A potential mechanism for mitochondrial dysfunction during cellular Ca2+ overload

1 reference

12 December 2020

Apoptosis in the failing human heart

1 reference

12 December 2020

Effects of intravenous nicorandil on the mid-term prognosis of patients with acute heart failure syndrome

1 reference

12 December 2020

Progression from compensated hypertrophy to failure in the pressure-overloaded human heart: structural deterioration and compensatory mechanisms

1 reference

12 December 2020

Regulation of mitochondrial ATP synthase in cardiac pathophysiology

1 reference

12 December 2020

The mitochondrial permeability transition pore and its role in cell death

1 reference

12 December 2020

Oxidation of pyridine nucleotides and depletion of ATP and ADP during calcium- and inorganic phosphate-induced mitochondrial permeability transition

1 reference

12 December 2020

Reversal of permeability transition during recovery of hearts from ischemia and its enhancement by pyruvate

1 reference

12 December 2020

The Ca2+-induced membrane transition in mitochondria. II. Nature of the Ca2+ trigger site

1 reference

12 December 2020

Apoptosis in myocytes in end-stage heart failure

1 reference

12 December 2020

Cardiomyocyte apoptosis and progression of heart failure to transplantation

1 reference

12 December 2020

BH3 death domain peptide induces cell type-selective mitochondrial outer membrane permeability

1 reference

12 December 2020

Apoptotic versus autophagic cell death in heart failure

1 reference

12 December 2020

Coenzyme Q10 for heart failure

1 reference

12 December 2020

The mitochondrial permeability transition pore may comprise VDAC molecules. I. Binary structure and voltage dependence of the pore

1 reference

12 December 2020

Myocyte death in the failing human heart is gender dependent

1 reference

12 December 2020

Can antioxidants be effective therapeutics?

1 reference

12 December 2020

Targeting an antioxidant to mitochondria decreases cardiac ischemia-reperfusion injury

1 reference

12 December 2020

10.1007/S10741-016-9531-1

Identifiers

DOI

1 reference

6 September 2017

1 reference