(Q38244750)

31 August 2017

title

Molecular identity of the mitochondrial permeability transition pore and its role in ischemia-reperfusion injury (English)

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31 August 2017

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8

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31 August 2017

Carlotta Giorgi

2

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Rita Pavasini

Rita Pavasini

5

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Gianluca Campo

Gianluca Campo

7

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Mariusz R Wieckowski

Mariusz R Wieckowski

6

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Giampaolo Morciano

1

Giampaolo Morciano

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31 August 2017

3

Massimo Bonora

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31 August 2017

4

Silvia Punzetti

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31 August 2017

publication date

27 August 2014

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Journal of Molecular and Cellular Cardiology

31 August 2017

published in

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31 August 2017

volume

78

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31 August 2017

page(s)

142-153

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The worldwide environment of cardiovascular disease: prevalence, diagnosis, therapy, and policy issues: a report from the American College of Cardiology

31 August 2017

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Cyclosporin A-sensitive and insensitive mechanisms produce the permeability transition in mitochondria

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Complexes between kinases, mitochondrial porin and adenylate translocator in rat brain resemble the permeability transition pore

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Complexes between porin, hexokinase, mitochondrial creatine kinase and adenylate translocator display properties of the permeability transition pore. Implication for regulation of permeability transition by the kinases

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Loss of cyclophilin D reveals a critical role for mitochondrial permeability transition in cell death

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Interactions of cyclophilin with the mitochondrial inner membrane and regulation of the permeability transition pore, and cyclosporin A-sensitive channel.

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The ADP/ATP translocator is not essential for the mitochondrial permeability transition pore

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Voltage-dependent anion channels are dispensable for mitochondrial-dependent cell death

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ATP synthesis and storage

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The mitochondrial phosphate carrier interacts with cyclophilin D and may play a key role in the permeability transition

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The roles of phosphate and the phosphate carrier in the mitochondrial permeability transition pore

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Genetic manipulation of the cardiac mitochondrial phosphate carrier does not affect permeability transition

7 January 2021

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Genetic deletion of the mitochondrial phosphate carrier desensitizes the mitochondrial permeability transition pore and causes cardiomyopathy

7 January 2021

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Isolation of the mitochondrial benzodiazepine receptor: association with the voltage-dependent anion channel and the adenine nucleotide carrier

7 January 2021

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7 January 2021

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Peripheral benzodiazepine receptor ligand, PK11195 induces mitochondria cytochrome c release and dissipation of mitochondria potential via induction of mitochondria permeability transition

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Resveratrol-induced apoptosis is enhanced in acute lymphoblastic leukemia cells by modulation of the mitochondrial permeability transition pore.

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7 January 2021

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Bax and Bak function as the outer membrane component of the mitochondrial permeability pore in regulating necrotic cell death in mice

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Bad targets the permeability transition pore independent of Bax or Bak to switch between Ca2+-dependent cell survival and death

7 January 2021

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Bcl-2 and Bax regulate the channel activity of the mitochondrial adenine nucleotide translocator

7 January 2021

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7 January 2021

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Glycogen synthase kinase-3beta mediates convergence of protection signaling to inhibit the mitochondrial permeability transition pore

7 January 2021

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p53 opens the mitochondrial permeability transition pore to trigger necrosis

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Mitochondria-ros crosstalk in the control of cell death and aging

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The role of oxidants and free radicals in reperfusion injury

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Ca2+ signaling, mitochondria and cell death

7 January 2021

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beta-Adrenergic pathway induces apoptosis through calcineurin activation in cardiac myocytes

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Mitochondrial permeability transition pore and calcium handling

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Role of the mitochondrial permeability transition in myocardial disease

7 January 2021

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The mitochondrial permeability transition pore and ischemia-reperfusion injury

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Mitochondrial permeability transition pore opening during myocardial reperfusion--a target for cardioprotection

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An update on cardioprotection: a review of the latest adjunctive therapies to limit myocardial infarction size in clinical trials

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Effect of cyclosporine on reperfusion injury in acute myocardial infarction

7 January 2021

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TRO40303, a new cardioprotective compound, inhibits mitochondrial permeability transition.

7 January 2021

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Rationale and Design of the MITOCARE Study: A Phase II, Multicenter, Randomized, Double-Blind, Placebo-Controlled Study to Assess the Safety and Efficacy of TRO40303 for the Reduction of Reperfusion Injury in Patients Undergoing Percutaneous Coro

7 January 2021

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Exenatide reduces final infarct size in patients with ST-segment-elevation myocardial infarction and short-duration of ischemia

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Atrial natriuretic peptide prevents the mitochondrial permeability transition pore opening by inactivating glycogen synthase kinase 3β via PKG and PI3K in cardiac H9c2 cells

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Efficacy of high-dose atorvastatin loading before primary percutaneous coronary intervention in ST-segment elevation myocardial infarction: the STATIN STEMI trial.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FJ.YJMCC.2014.08.015

10.1016/J.YJMCC.2014.08.015

7 January 2021

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DOI

1 reference

31 August 2017

1 reference