(Q38968529)

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Docosahexanoic acid antagonizes TNF-α-induced necroptosis by attenuating oxidative stress, ceramide production, lysosomal dysfunction, and autophagic features

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scholarly article

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Europe PubMed Central

PMC publication ID

8 September 2017

Docosahexanoic acid antagonizes TNF-α-induced necroptosis by attenuating oxidative stress, ceramide production, lysosomal dysfunction, and autophagic features (English)

1 reference

Europe PubMed Central

PMC publication ID

8 September 2017

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based on heuristic

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object named as

Marino De Leon

9

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author name string

Fabio J Pacheco

1

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Europe PubMed Central

PMC publication ID

8 September 2017

Frankis G Almaguel

2

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Europe PubMed Central

PMC publication ID

8 September 2017

Whitney Evans

3

1 reference

Europe PubMed Central

PMC publication ID

8 September 2017

Leslimar Rios-Colon

4

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Europe PubMed Central

PMC publication ID

8 September 2017

Valery Filippov

5

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Europe PubMed Central

PMC publication ID

8 September 2017

Lai S Leoh

6

1 reference

Europe PubMed Central

PMC publication ID

8 September 2017

Elizabeth Rook-Arena

7

1 reference

Europe PubMed Central

PMC publication ID

8 September 2017

Melanie Mediavilla-Varela

8

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Europe PubMed Central

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8 September 2017

Carlos A Casiano

10

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Europe PubMed Central

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8 September 2017

publication date

6 August 2014

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Europe PubMed Central

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8 September 2017

Inflammation Research

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Europe PubMed Central

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8 September 2017

63

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Europe PubMed Central

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8 September 2017

10

1 reference

Europe PubMed Central

PMC publication ID

8 September 2017

859-871

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Europe PubMed Central

PMC publication ID

8 September 2017

Activity of protein kinase RIPK3 determines whether cells die by necroptosis or apoptosis

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4205108

9 September 2017

Depletion of RIPK3 or MLKL blocks TNF-driven necroptosis and switches towards a delayed RIPK1 kinase-dependent apoptosis

1 reference

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Control of life-or-death decisions by RIP1 kinase

1 reference

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Omega-3 polyunsaturated fatty acids and cancer

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RIP3: a molecular switch for necrosis and inflammation

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Dietary omega-3 fatty acid deficiency and high fructose intake in the development of metabolic syndrome, brain metabolic abnormalities, and non-alcoholic fatty liver disease

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Plasma phospholipid fatty acids and prostate cancer risk in the SELECT trial

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Role of omega-3 fatty acids in obesity, metabolic syndrome, and cardiovascular diseases: a review of the evidence.

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The relevance of serum levels of long chain omega-3 polyunsaturated fatty acids and prostate cancer risk: A meta-analysis

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n-3 fatty acids: role in neurogenesis and neuroplasticity

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TNF-α signalling and inflammation: interactions between old acquaintances

1 reference

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1 reference

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Polyunsaturated fatty acids in inflammatory bowel diseases: a reappraisal of effects and therapeutic approaches

1 reference

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Autophagy and inflammatory diseases

1 reference

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Dietary omega-3 polyunsaturated fatty acids improve the neurolipidome and restore the DHA status while promoting functional recovery after experimental spinal cord injury

1 reference

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Necrostatin-1 analogues: critical issues on the specificity, activity and in vivo use in experimental disease models

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Fueling the flames: Mammalian programmed necrosis in inflammatory diseases

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9 September 2017

Tumor necrosis factor

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Docosahexaenoic fatty acid (DHA) in the regulation of colon cell growth and cell death: a review

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Resolvins: anti-inflammatory and proresolving mediators derived from omega-3 polyunsaturated fatty acids.

1 reference

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9 September 2017

Dichotomy between RIP1- and RIP3-mediated necroptosis in tumor necrosis factor-α-induced shock.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4205108

9 September 2017

TNF-induced necroptosis in L929 cells is tightly regulated by multiple TNFR1 complex I and II members

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4205108

9 September 2017

RIP1-dependent and independent effects of necrostatin-1 in necrosis and T cell activation

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4205108

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Docosahexaenoic acid-derived neuroprotectin D1 induces neuronal survival via secretase- and PPARγ-mediated mechanisms in Alzheimer's disease models

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4205108

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PARP-1 cleavage fragments: signatures of cell-death proteases in neurodegeneration

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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4205108

9 September 2017

zVAD-induced necroptosis in L929 cells depends on autocrine production of TNFα mediated by the PKC-MAPKs-AP-1 pathway.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4205108

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Inhibition of cytokine signaling in human retinal endothelial cells through downregulation of sphingomyelinases by docosahexaenoic acid

1 reference

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Docosahexaenoic acid (DHA) has neuroprotective effects against oxidative stress in retinal ganglion cells

1 reference

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TNF-mediated inflammatory disease.

1 reference

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Docosanoids are multifunctional regulators of neural cell integrity and fate: significance in aging and disease

1 reference

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Tumor necrosis factor (TNF) biology and cell death

1 reference

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Up-regulation of lysosomal cathepsin L and autophagy during neuronal death induced by reduced serum and potassium

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Palmitic and stearic fatty acids induce caspase-dependent and -independent cell death in nerve growth factor differentiated PC12 cells

1 reference

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Caspase-mediated cleavage of DNA topoisomerase I at unconventional sites during apoptosis

1 reference

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Negatively-regulated necroptosis by autophagy required caspase-6 activation in TNFα-treated murine fibrosarcoma L929 cells

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RIP1-mediated mitochondrial dysfunction and ROS production contributed to tumor necrosis factor alpha-induced L929 cell necroptosis and autophagy

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Lipotoxicity-mediated cell dysfunction and death involve lysosomal membrane permeabilization and cathepsin L activity

1 reference

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1 reference

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Necroptosis, necrosis and secondary necrosis converge on similar cellular disintegration features.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4205108

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ERK and JNK mediate TNFalpha-induced p53 activation in apoptotic and autophagic L929 cell death

1 reference

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C18 ceramide analysis in mammalian cells employing reversed-phase high-performance liquid chromatography tandem mass spectrometry.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4205108

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Autophagy plays a protective role during zVAD-induced necrotic cell death.

1 reference

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Docosahexaenoic acid enrichment can reduce L929 cell necrosis induced by tumor necrosis factor

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Ceramide mediates caspase-independent programmed cell death

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Modes of L929 cell death induced by TNF-alpha and other cytotoxic agents.

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Membrane TNF-alpha-activated programmed necrosis is mediated by Ceramide-induced reactive oxygen species

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Necroptosis turns TNF lethal

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1 reference

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Critical contribution of oxidative stress to TNFα-induced necroptosis downstream of RIPK1 activation

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RIP kinase-dependent necrosis drives lethal systemic inflammatory response syndrome

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1 reference

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12 December 2020

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10.1007/S00011-014-0760-2

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Europe PubMed Central

PMC publication ID

8 September 2017

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PMC publication ID

1 reference

Europe PubMed Central

PMC publication ID

8 September 2017

PubMed publication ID

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Europe PubMed Central

PMC publication ID

8 September 2017

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