(Q39155413)

9 September 2017

title

ENaC contribution to epithelial wound healing is independent of the healing mode and of any increased expression in the channel (English)

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9 September 2017

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5

Silvia Chifflet

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9 September 2017

Cristian Justet

1

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9 September 2017

Frances Evans

series ordinal

2

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9 September 2017

Elena Vasilskis

series ordinal

3

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9 September 2017

Julio A Hernández

series ordinal

4

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9 September 2017

publication date

7 May 2013

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9 September 2017

published in

Cell and Tissue Research

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9 September 2017

volume

353

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9 September 2017

issue

1

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9 September 2017

page(s)

53-64

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Mechanisms of ENaC regulation and clinical implications

9 September 2017

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A possible role for membrane depolarization in epithelial wound healing

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Regulation of epithelial sodium channel trafficking by ubiquitination

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Polarity proteins in migration and invasion

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Multiple rows of cells behind an epithelial wound edge extend cryptic lamellipodia to collectively drive cell-sheet movement

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Amiloride and its analogs as tools to inhibit Na+ transport via the Na+ channel, the Na+/H+ antiport and the Na+/Ca2+ exchanger

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Roles of wound geometry, wound size, and extracellular matrix in the healing response of bovine corneal endothelial cells in culture

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Cells move when ions and water flow

21 January 2018

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Inhibition of sodium-calcium and sodium-proton exchangers by amiloride congeners in arterial muscle cells

21 January 2018

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Wound repair: toward understanding and integration of single-cell and multicellular wound responses

21 January 2018

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21 January 2018

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The role of the ENaC-regulatory complex in aldosterone-mediated sodium transport

21 January 2018

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21 January 2018

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21 January 2018

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21 January 2018

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21 January 2018

Identifiers

DOI

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9 September 2017

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