(Q39172025)

9 September 2017

title

Phospho-tyrosine phosphatase inhibitor Bpv(Hopic) enhances C2C12 myoblast migration in vitro. Requirement of PI3K/AKT and MAPK/ERK pathways (English)

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9 September 2017

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Georgi A Dimchev

1

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Nasser Al-Shanti

object named as

Nasser Al-Shanti

series ordinal

2

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Claire E. Stewart

object named as

Claire E Stewart

series ordinal

3

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publication date

4 April 2013

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Journal of Muscle Research & Cell Motility

9 September 2017

published in

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9 September 2017

volume

34

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9 September 2017

issue

2

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9 September 2017

page(s)

125-136

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https://scigraph.springernature.com/pub.10.1007/s10974-013-9340-2

9 September 2017

exact match

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cites work

Growth factor stimulation of matrix metalloproteinase expression and myoblast migration and invasion in vitro

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A semi-automated programme for tracking myoblast migration following mechanical damage: manipulation by chemical inhibitors

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Hepatocyte growth factor is essential for migration of myogenic cells and promotes their proliferation during the early periods of tongue morphogenesis in mouse embryos.

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Chemotaxis of skeletal muscle satellite cells

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Bisperoxovanadium, a phospho-tyrosine phosphatase inhibitor, reprograms myogenic cells to acquire a pluripotent, circulating phenotype

21 January 2018

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Exon skipping and dystrophin restoration in patients with Duchenne muscular dystrophy after systemic phosphorodiamidate morpholino oligomer treatment: an open-label, phase 2, dose-escalation study

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Chemotactic factors enhance myogenic cell migration across an endothelial monolayer.

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In vivo migration of transplanted myoblasts requires matrix metalloproteinase activity.

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PTEN Tumor Suppressor Network in PI3K-Akt Pathway Control

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Tumor suppressor PTEN inhibits integrin- and growth factor-mediated mitogen-activated protein (MAP) kinase signaling pathways

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The fate of individual myoblasts after transplantation into muscles of DMD patients

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N-WASP and WAVE2 acting downstream of phosphatidylinositol 3-kinase are required for myogenic cell migration induced by hepatocyte growth factor

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α-Syntrophin is required for the hepatocyte growth factor-induced migration of cultured myoblasts

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Growth factors improve the in vivo migration of human skeletal myoblasts by modulating their endogenous proteolytic activity.

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Phosphatase and tensin homologue deleted on chromosome ten (PTEN) as a molecular target in lung epithelial wound repair

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Involvement of the ERK/MAP kinase signalling pathway in milli-calpain activation and myogenic cell migration

21 January 2018

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Gelatinase-B (matrix metalloproteinase-9; MMP-9) secretion is involved in the migratory phase of human and murine muscle cell cultures

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PTEN inhibition improves wound healing in lung epithelia through changes in cellular mechanics that enhance migration

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Bisperoxovanadium compounds are potent PTEN inhibitors.

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Transplanted myoblasts can migrate several millimeters to fuse with damaged myofibers in nonhuman primate skeletal muscle

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Problems and solutions in myoblast transfer therapy.

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Involvement of Ras and Ral in chemotactic migration of skeletal myoblasts

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Fibronectin promotes migration, alignment and fusion in an in vitro myoblast cell model

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Negative regulation of myofibroblast differentiation by PTEN (Phosphatase and Tensin Homolog Deleted on chromosome 10)

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Migration of muscle cells

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Tumor Necrosis Factor-α (TNF-α) Stimulates Chemotactic Response in Mouse Myogenic Cells

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12 December 2020

Involvement of calpains in growth factor-mediated migration

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12 December 2020

Growth factor coinjection improves the migration potential of monkey myogenic precursors without affecting cell transplantation success

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12 December 2020

Rapid death of injected myoblasts in myoblast transfer therapy

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12 December 2020

At the leading edge of three-dimensional cell migration

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12 December 2020

Lack of myoblasts migration between transplanted and host muscles of mdx and normal mice

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12 December 2020

10.1007/S10974-013-9340-2

Identifiers

DOI

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9 September 2017

1 reference