(Q39440343)

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The generation of a reverse genetics system for Kyasanur Forest Disease Virus and the ability to antagonize the induction of the antiviral state in vitro

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scholarly article

1 reference

Europe PubMed Central

12 September 2017

The generation of a reverse genetics system for Kyasanur Forest Disease Virus and the ability to antagonize the induction of the antiviral state in vitro (English)

1 reference

Europe PubMed Central

12 September 2017

Kyasanur forest disease

1 reference

based on heuristic

inferred from title

Kyasanur Forest disease virus

object named as

Kyasanur Forest Disease Virus

1 reference

based on heuristic

inferred from title

author name string

Bradley W M Cook

1

1 reference

Europe PubMed Central

12 September 2017

Todd A Cutts

2

1 reference

Europe PubMed Central

12 September 2017

Deborah A Court

3

1 reference

Europe PubMed Central

12 September 2017

Steven Theriault

4

1 reference

Europe PubMed Central

12 September 2017

publication date

12 November 2011

1 reference

Europe PubMed Central

12 September 2017

1 reference

Europe PubMed Central

12 September 2017

163

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Europe PubMed Central

12 September 2017

2

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Europe PubMed Central

12 September 2017

431-438

1 reference

Europe PubMed Central

12 September 2017

Inhibition of interferon-stimulated JAK-STAT signaling by a tick-borne flavivirus and identification of NS5 as an interferon antagonist

1 reference

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Chapter 2. New insights into flavivirus nonstructural protein 5.

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Zoonotic tick-borne flaviviruses

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Increased recognition of Powassan encephalitis in the United States, 1999-2005.

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Virus interference. I. The interferon

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Virus interference. II. Some properties of interferon

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The flavivirus NS5 protein is a true RNA guanylyltransferase that catalyzes a two-step reaction to form the RNA cap structure

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Functional analysis of the tick-borne encephalitis virus cyclization elements indicates major differences between mosquito-borne and tick-borne flaviviruses

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Infectious RNA transcribed from stably cloned full-length cDNA of dengue type 4 virus

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The NS5 Protein of the Virulent West Nile Virus NY99 Strain Is a Potent Antagonist of Type I Interferon-Mediated JAK-STAT Signaling

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Identification of a cryptic prokaryotic promoter within the cDNA encoding the 5' end of dengue virus RNA genome

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Analysis of the complete genome of the tick-borne flavivirus Omsk hemorrhagic fever virus

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A Single Amino Acid Substitution in the West Nile Virus Nonstructural Protein NS2A Disables Its Ability To Inhibit Alpha/Beta Interferon Induction and Attenuates Virus Virulence in Mice

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Biological assays for interferons

1 reference

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Recent ancestry of Kyasanur Forest disease virus

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Product review. New mammalian expression vectors

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Inhibition of Alpha/Beta Interferon Signaling by the NS4B Protein of Flaviviruses

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Inhibition of interferon signaling by dengue virus

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Reverse genetics demonstrates that proteolytic processing of the Ebola virus glycoprotein is not essential for replication in cell culture

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Identification of Residues Critical for the Interferon Antagonist Function of Langat Virus NS5 Reveals a Role for the RNA-Dependent RNA Polymerase Domain

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Kyasanur forest disease: an epidemiological view in India

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The interferons: 50 years after their discovery, there is much more to learn

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Rhabdomyolysis and severe muscular weakness in a traveler diagnosed with Alkhurma hemorrhagic fever virus infection

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Ebola virus VP24 binds karyopherin alpha1 and blocks STAT1 nuclear accumulation

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10.1016/J.VIRUSRES.2011.11.002

1 reference

Europe PubMed Central

12 September 2017

1 reference

Europe PubMed Central

12 September 2017

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