(Q39622621)

13 September 2017

Copper depletion increases the mitochondrial-associated SOD1 in neuronal cells (English)

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13 September 2017

Mario Arciello

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Mauro Cozzolino

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Mauro Cozzolino

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Alberto Ferri

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Alberto Ferri

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author name string

Concetta Rosa Capo

2

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13 September 2017

Sara D'Annibale

3

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13 September 2017

Maria Teresa Carrì

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13 September 2017

Luisa Rossi

7

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13 September 2017

8 December 2010

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13 September 2017

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13 September 2017

24

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13 September 2017

269-278

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13 September 2017

2

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https://scigraph.springernature.com/pub.10.1007/s10534-010-9392-3

13 September 2017

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cites work

Inactivation of cytochrome c oxidase by mutant SOD1s in mouse motoneuronal NSC-34 cells is independent from copper availability but is because of nitric oxide.

1 reference

https://api.crossref.org/works/10.1007%2FS10534-010-9392-3

Metals in Alzheimer's and Parkinson's diseases

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10534-010-9392-3

Copper modulates the degradation of copper chaperone for Cu,Zn superoxide dismutase by the 26 S proteosome.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10534-010-9392-3

Neuroblastoma x spinal cord (NSC) hybrid cell lines resemble developing motor neurons

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10534-010-9392-3

Importing mitochondrial proteins: machineries and mechanisms

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10534-010-9392-3

Amyotrophic lateral sclerosis: from current developments in the laboratory to clinical implications

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10534-010-9392-3

Oligomerization of mutant SOD1 in mitochondria of motoneuronal cells drives mitochondrial damage and cell toxicity

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10534-010-9392-3

Conversion to the amyotrophic lateral sclerosis phenotype is associated with intermolecular linked insoluble aggregates of SOD1 in mitochondria

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10534-010-9392-3

Copper activation of superoxide dismutase in rat erythrocytes

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10534-010-9392-3

Familial ALS-superoxide dismutases associate with mitochondria and shift their redox potentials.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10534-010-9392-3

Factors controlling the uptake of yeast copper/zinc superoxide dismutase into mitochondria.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10534-010-9392-3

Oxygen-induced maturation of SOD1: a key role for disulfide formation by the copper chaperone CCS.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10534-010-9392-3

The copper chelator d-penicillamine delays onset of disease and extends survival in a transgenic mouse model of familial amyotrophic lateral sclerosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10534-010-9392-3

Function, structure, and mechanism of intracellular copper trafficking proteins

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10534-010-9392-3

Prolonged copper depletion induces expression of antioxidants and triggers apoptosis in SH-SY5Y neuroblastoma cells

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10534-010-9392-3

Mutated human SOD1 causes dysfunction of oxidative phosphorylation in mitochondria of transgenic mice.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10534-010-9392-3

Subcellular distribution of superoxide dismutases (SOD) in rat liver: Cu,Zn-SOD in mitochondria

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10534-010-9392-3

Metallochaperone for Cu,Zn-superoxide dismutase (CCS) protein but not mRNA is higher in organs from copper-deficient mice and rats.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10534-010-9392-3

Overexpression of CCS in G93A-SOD1 mice leads to accelerated neurological deficits with severe mitochondrial pathology

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10534-010-9392-3

Mia40, a novel factor for protein import into the intermembrane space of mitochondria is able to bind metal ions

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10534-010-9392-3

Ammonium tetrathiomolybdate delays onset, prolongs survival, and slows progression of disease in a mouse model for amyotrophic lateral sclerosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10534-010-9392-3

Dysregulation of intracellular copper trafficking pathway in a mouse model of mutant copper/zinc superoxide dismutase-linked familial amyotrophic lateral sclerosis.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10534-010-9392-3

Copper-zinc superoxide dismutase and amyotrophic lateral sclerosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10534-010-9392-3

Posttranslational regulation of copper transporters

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10534-010-9392-3

Selective association of misfolded ALS-linked mutant SOD1 with the cytoplasmic face of mitochondria.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10534-010-9392-3

Altered microglial copper homeostasis in a mouse model of Alzheimer's disease

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10534-010-9392-3

Genetically Decreased Spinal Cord Copper Concentration Prolongs Life in a Transgenic Mouse Model of Amyotrophic Lateral Sclerosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS10534-010-9392-3

10.1007/S10534-010-9392-3

21 January 2018

Identifiers

DOI

1 reference

13 September 2017

1 reference