(Q39632457)

13 September 2017

title

Suppression of NADPH oxidase 2 substantially restores glucose-induced dysfunction of pancreatic NIT-1 cells (English)

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13 September 2017

Huiping Yuan

1

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13 September 2017

Yonggang Lu

2

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13 September 2017

Xiuqing Huang

3

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13 September 2017

Qinghua He

4

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13 September 2017

Yong Man

5

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13 September 2017

Yingsheng Zhou

6

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13 September 2017

Shu Wang

7

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13 September 2017

Jian Li

8

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13 September 2017

language of work or name

English

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publication date

12 November 2010

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13 September 2017

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13 September 2017

volume

277

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13 September 2017

issue

24

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13 September 2017

page(s)

5061-5071

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Thioredoxin-interacting protein: a critical link between glucose toxicity and beta-cell apoptosis

13 September 2017

cites work

1 reference

https://api.crossref.org/works/10.1111%2FJ.1742-4658.2010.07911.X

Glucolipotoxicity: fuel excess and beta-cell dysfunction.

21 January 2018

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Diabetes associated cell stress and dysfunction: role of mitochondrial and non-mitochondrial ROS production and activity

21 January 2018

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https://api.crossref.org/works/10.1111%2FJ.1742-4658.2010.07911.X

The NOX family of ROS-generating NADPH oxidases: physiology and pathophysiology

21 January 2018

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https://api.crossref.org/works/10.1111%2FJ.1742-4658.2010.07911.X

Insights into the critical role of NADPH oxidase(s) in the normal and dysregulated pancreatic beta cell

21 January 2018

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https://api.crossref.org/works/10.1111%2FJ.1742-4658.2010.07911.X

Association of NAD(P)H oxidase with glucose-induced insulin secretion by pancreatic beta-cells.

21 January 2018

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https://api.crossref.org/works/10.1111%2FJ.1742-4658.2010.07911.X

Reactive nitrogen species induced by hyperglycemia suppresses Akt signaling and triggers apoptosis by upregulating phosphatase PTEN (phosphatase and tensin homologue deleted on chromosome 10) in an LKB1-dependent manner

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FJ.1742-4658.2010.07911.X

The forkhead transcription factor Foxo1 bridges the JNK pathway and the transcription factor PDX-1 through its intracellular translocation

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FJ.1742-4658.2010.07911.X

Normalizing mitochondrial superoxide production blocks three pathways of hyperglycaemic damage

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FJ.1742-4658.2010.07911.X

Glucose and leptin induce apoptosis in human beta-cells and impair glucose-stimulated insulin secretion through activation of c-Jun N-terminal kinases

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FJ.1742-4658.2010.07911.X

Inhibition of Foxo1 protects pancreatic islet beta-cells against fatty acid and endoplasmic reticulum stress-induced apoptosis

21 January 2018

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https://api.crossref.org/works/10.1111%2FJ.1742-4658.2010.07911.X

A central role for JNK in obesity and insulin resistance

21 January 2018

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https://api.crossref.org/works/10.1111%2FJ.1742-4658.2010.07911.X

Oxidative stress induces nucleo-cytoplasmic translocation of pancreatic transcription factor PDX-1 through activation of c-Jun NH(2)-terminal kinase.

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FJ.1742-4658.2010.07911.X

The emerging role of FOXO transcription factors in pancreatic beta cells.

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FJ.1742-4658.2010.07911.X

Regulation of insulin action and pancreatic beta-cell function by mutated alleles of the gene encoding forkhead transcription factor Foxo1.

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FJ.1742-4658.2010.07911.X

Elimination of Mcl-1 is required for the initiation of apoptosis following ultraviolet irradiation

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FJ.1742-4658.2010.07911.X

ER stress and SREBP-1 activation are implicated in beta-cell glucolipotoxicity

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FJ.1742-4658.2010.07911.X

Expression of an uncleavable N-terminal RasGAP fragment in insulin-secreting cells increases their resistance toward apoptotic stimuli without affecting their glucose-induced insulin secretion

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FJ.1742-4658.2010.07911.X

10.1111/J.1742-4658.2010.07911.X

21 January 2018

Identifiers

DOI

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13 September 2017

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