(Q39726376)

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Enhanced death ligand-induced apoptosis in cutaneous SCC cells by treatment with diclofenac/hyaluronic acid correlates with downregulation of c-FLIP.

scientific article published on 18 March 2010

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scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

14 September 2017

Enhanced death ligand-induced apoptosis in cutaneous SCC cells by treatment with diclofenac/hyaluronic acid correlates with downregulation of c-FLIP. (English)

1 reference

Europe PubMed Central

PubMed publication ID

14 September 2017

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Eggert Stockfleth

2

object named as

Eggert Stockfleth

1 reference

Europe PubMed Central

PubMed publication ID

14 September 2017

author name string

Lothar F Fecker

1

1 reference

Europe PubMed Central

PubMed publication ID

14 September 2017

Frank K Braun

3

1 reference

Europe PubMed Central

PubMed publication ID

14 September 2017

Paul M Rodust

4

1 reference

Europe PubMed Central

PubMed publication ID

14 September 2017

Constanze Schwarz

5

1 reference

Europe PubMed Central

PubMed publication ID

14 September 2017

Anja Köhler

6

1 reference

Europe PubMed Central

PubMed publication ID

14 September 2017

Martin Leverkus

7

1 reference

Europe PubMed Central

PubMed publication ID

14 September 2017

Jürgen Eberle

8

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Europe PubMed Central

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14 September 2017

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publication date

18 March 2010

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Europe PubMed Central

PubMed publication ID

14 September 2017

Journal of Investigative Dermatology

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Europe PubMed Central

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14 September 2017

130

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Europe PubMed Central

PubMed publication ID

14 September 2017

8

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Europe PubMed Central

PubMed publication ID

14 September 2017

2098-2109

1 reference

Europe PubMed Central

PubMed publication ID

14 September 2017

https://scigraph.springernature.com/pub.10.1038/jid.2010.40

0 references

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Suprabasal spongiosis in acute eczematous dermatitis: cFLIP maintains resistance of basal keratinocytes to T-cell-mediated apoptosis

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Disparate effects of non-steroidal anti-inflammatory drugs on apoptosis in guinea-pig gastric mucous cells: inhibition of basal apoptosis by diclofenac

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Ultraviolet light downregulates CD95 ligand and TRAIL receptor expression facilitating actinic keratosis and squamous cell carcinoma formation

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Blockade of death receptor-mediated pathways early in the signaling cascade coincides with distinct apoptosis resistance in cutaneous T-cell lymphoma cells

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Resistance of cutaneous anaplastic large-cell lymphoma cells to apoptosis by death ligands is enhanced by CD30-mediated overexpression of c-FLIP.

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Protection against Fas-induced liver apoptosis in transgenic mice expressing cyclooxygenase 2 in hepatocytes

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CCAAT/enhancer binding protein homologous protein-dependent death receptor 5 induction and ubiquitin/proteasome-mediated cellular FLICE-inhibitory protein down-regulation contribute to enhancement of tumor necrosis factor-related apoptosis-inducing

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Apoptosis pathways as promising targets for skin cancer therapy

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CD95/Fas signaling in human melanoma cells: conditional expression of CD95L/FasL overcomes the intrinsic apoptosis resistance of malignant melanoma and inhibits growth and progression of human melanoma xenotransplants

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The role of the p53 tumor suppressor gene in squamous cell carcinoma of the head and neck

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Expression of CD95 (Fas) in sun-exposed human skin and cutaneous carcinomas

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Many cuts to ruin: a comprehensive update of caspase substrates

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Apoptosis-based therapies and drug targets

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Extrinsic versus intrinsic apoptosis pathways in anticancer chemotherapy

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TRAIL-mediated apoptosis in malignant glioma cells is augmented by celecoxib through proteasomal degradation of survivin

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Diclofenac induces apoptosis in hepatocytes by alteration of mitochondrial function and generation of ROS.

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Raft ceramide in molecular medicine

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Effective photodynamic therapy of actinic keratoses on the head and face with a novel, self-adhesive 5-aminolaevulinic acid patch

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Celecoxib and a novel COX-2 inhibitor ON09310 upregulate death receptor 5 expression via GADD153/CHOP.

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Molecular mechanism of diclofenac-induced apoptosis of promyelocytic leukemia: dependency on reactive oxygen species, Akt, Bid, cytochrome and caspase pathway

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NSAIDs and apoptosis

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Cyclooxygenase-2 is expressed in neuroblastoma, and nonsteroidal anti-inflammatory drugs induce apoptosis and inhibit tumor growth in vivo

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Cyclooxygenase-2 inhibition induces apoptosis signaling via death receptors and mitochondria in hepatocellular carcinoma

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Apoptosis: A Basic Biological Phenomenon with Wideranging Implications in Tissue Kinetics

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Cyclooxygenase inhibitors induce apoptosis in oral cavity cancer cells by increased expression of nonsteroidal anti-inflammatory drug-activated gene

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Reactive oxygen species are involved in the apoptosis induced by nonsteroidal anti-inflammatory drugs in cultured gastric cells

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p53 mutations in human immortalized epithelial cell lines

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Identifiers

10.1038/JID.2010.40

1 reference

Europe PubMed Central

PubMed publication ID

14 September 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

14 September 2017

ResearchGate publication ID

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