(Q39847273)

15 September 2017

title

An agonistic monoclonal antibody against DR5 induces ROS production, sustained JNK activation and Endo G release in Jurkat leukemia cells (English)

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15 September 2017

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Caifeng Chen

1

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15 September 2017

Yanxin Liu

series ordinal

2

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15 September 2017

Dexian Zheng

series ordinal

3

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15 September 2017

publication date

1 August 2009

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15 September 2017

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15 September 2017

volume

19

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15 September 2017

issue

8

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15 September 2017

page(s)

984-995

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https://scigraph.springernature.com/pub.10.1038/cr.2009.60

15 September 2017

exact match

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cites work

Targeting death and decoy receptors of the tumour-necrosis factor superfamily

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A novel anti-human DR5 monoclonal antibody with tumoricidal activity induces caspase-dependent and caspase-independent cell death

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Apoptosis signaling by death receptors.

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TRAIL receptor-2 signals apoptosis through FADD and caspase-8

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Sustained activation of p38 mitogen-activated protein kinase and c-Jun N-terminal kinase pathways by hepatitis B virus X protein mediates apoptosis via induction of Fas/FasL and tumor necrosis factor (TNF) receptor 1/TNF-alpha expression

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Activation of the p38 mitogen-activated protein kinase pathway by Epstein-Barr virus-encoded latent membrane protein 1 coregulates interleukin-6 and interleukin-8 production

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Reactive oxygen special (ROS), troublemakers between nuclear factor-kB (NF-kB) and c-Jun NH2-terminal kinase (JNK)

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Reactive oxygen species promote TNFalpha-induced death and sustained JNK activation by inhibiting MAP kinase phosphatases

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NF-kappaB inhibits TNF-induced accumulation of ROS that mediate prolonged MAPK activation and necrotic cell death

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The Fas death signaling pathway connecting reactive oxygen species generation and FLICE inhibitory protein down-regulation

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Endogenous interleukin-6 enhances the renal injury, dysfunction, and inflammation caused by ischemia/reperfusion

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JNK potentiates TNF-stimulated necrosis by increasing the production of cytotoxic reactive oxygen species

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Tunicamycin enhances tumor necrosis factor-related apoptosis-inducing ligand-induced apoptosis in human prostate cancer cells

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Bisindolylmaleimide VIII enhances DR5-mediated apoptosis through the MKK4/JNK/p38 kinase and the mitochondrial pathways

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Fas triggers an alternative, caspase-8-independent cell death pathway using the kinase RIP as effector molecule

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Arsenic trioxide sensitizes human glioma cells, but not normal astrocytes, to TRAIL-induced apoptosis via CCAAT/enhancer-binding protein homologous protein-dependent DR5 up-regulation

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Artesunate induces ROS-mediated apoptosis in doxorubicin-resistant T leukemia cells

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Sulforaphane enhances the therapeutic potential of TRAIL in prostate cancer orthotopic model through regulation of apoptosis, metastasis, and angiogenesis

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BH3-only proteins in control: specificity regulates MCL-1 and BAK-mediated apoptosis

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Redox regulation of resveratrol-mediated switching of death signal into survival signal

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Cytochrome c is released from mitochondria in a reactive oxygen species (ROS)-dependent fashion and can operate as a ROS scavenger and as a respiratory substrate in cerebellar neurons undergoing excitotoxic death

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Phytosphingosine in combination with ionizing radiation enhances apoptotic cell death in radiation-resistant cancer cells through ROS-dependent and -independent AIF release

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Induction of endonuclease G-mediated apopotosis in human oral squamous cell carcinoma cells by protein kinase C inhibitor safingol

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Molecular determinants of kinase pathway activation by Apo2 ligand/tumor necrosis factor-related apoptosis-inducing ligand

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Reactive oxygen species mediate crosstalk between NF-kappaB and JNK.

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Tumoricidal activity of a novel anti-human DR5 monoclonal antibody without hepatocyte cytotoxicity.

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A human scFv antibody against TRAIL receptor 2 induces autophagic cell death in both TRAIL-sensitive and TRAIL-resistant cancer cells.

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Does the redox status of cytochrome C act as a fail-safe mechanism in the regulation of programmed cell death?

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21 January 2018

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Sustained JNK activation in response to tumor necrosis factor is mediated by caspases in a cell type-specific manner

21 January 2018

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https://api.crossref.org/works/10.1038%2FCR.2009.60

Necrotic death as a cell fate

21 January 2018

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Role of JNK activation in apoptosis: a double-edged sword

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FCR.2009.60

Endonuclease G is an apoptotic DNase when released from mitochondria

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FCR.2009.60

Bcl-2-family proteins: the role of the BH3 domain in apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FCR.2009.60

Exploiting different ways to die.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FCR.2009.60

More than one way to die: apoptosis, necrosis and reactive oxygen damage

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FCR.2009.60

A JNK-Dependent Pathway Is Required for TNFα-Induced Apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FCR.2009.60

inferred from DOI database lookup

7 January 2021

based on heuristic

Overexpression of manganese superoxide dismutase by N-acetylcysteine in hyperoxic lung injury

1 reference

https://api.crossref.org/works/10.1038%2FCR.2009.60

inferred from DOI database lookup

7 January 2021

based on heuristic

Identifiers

DOI

10.1038/CR.2009.60

1 reference

15 September 2017

1 reference