(Q39864100)

English

Cultures of HEp-2 cells persistently infected by human respiratory syncytial virus differ in chemokine expression and resistance to apoptosis as compared to lytic infections of the same cell type

scientific article published on 5 April 2009

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scholarly article

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Europe PubMed Central

PubMed publication ID

15 September 2017

Cultures of HEp-2 cells persistently infected by human respiratory syncytial virus differ in chemokine expression and resistance to apoptosis as compared to lytic infections of the same cell type (English)

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Europe PubMed Central

PubMed publication ID

15 September 2017

respiratory syncytial virus

object named as

human respiratory syncytial virus

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author name string

Isidoro Martínez

1

1 reference

Europe PubMed Central

PubMed publication ID

15 September 2017

Luis Lombardía

2

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Europe PubMed Central

PubMed publication ID

15 September 2017

Cristina Herranz

3

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Europe PubMed Central

PubMed publication ID

15 September 2017

Blanca García-Barreno

4

1 reference

Europe PubMed Central

PubMed publication ID

15 September 2017

Orlando Domínguez

5

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Europe PubMed Central

PubMed publication ID

15 September 2017

José A Melero

6

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Europe PubMed Central

PubMed publication ID

15 September 2017

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publication date

5 April 2009

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Europe PubMed Central

PubMed publication ID

15 September 2017

number of pages

11

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Europe PubMed Central

PubMed publication ID

15 September 2017

388

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Europe PubMed Central

PubMed publication ID

15 September 2017

1

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Europe PubMed Central

PubMed publication ID

15 September 2017

31-41

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Europe PubMed Central

PubMed publication ID

15 September 2017

Specific human cytotoxic T cells recognize B-cell lines persistently infected with respiratory syncytial virus

1 reference

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Nonstructural proteins of respiratory syncytial virus suppress premature apoptosis by an NF-kappaB-dependent, interferon-independent mechanism and facilitate virus growth

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Persistent of respiratory syncytial virus in human dendritic cells and influence of nitric oxide

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Establishment of Persistent Infection by Parainfluenza Virus Type 3: Role of a Syncytium Inhibitor

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Persistent infection of mouse cells with Sindbis virus: role of virulence of strains, auto-interfering particles and interferon

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Persistent activation of an innate immune response translates respiratory viral infection into chronic lung disease

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Innate immune responses in respiratory syncytial virus infections

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Respiratory syncytial virus inhibits granulocyte apoptosis through a phosphatidylinositol 3-kinase and NF-kappaB-dependent mechanism

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Structural basis of integrin regulation and signaling

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Antigenic structure of the human respiratory syncytial virus G glycoprotein and relevance of hypermutation events for the generation of antigenic variants

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Reduced expression of surface glycoproteins in mouse fibroblasts persistently infected with human respiratory syncytial virus (HRSV).

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Distinct gene subsets are induced at different time points after human respiratory syncytial virus infection of A549 cells

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Immunohistological demonstration of respiratory syncytial virus antigens in Paget disease of bone

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Activation of the epidermal growth factor receptor by respiratory syncytial virus results in increased inflammation and delayed apoptosis

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Induction of CD95 (Fas) and apoptosis in respiratory epithelial cell cultures following respiratory syncytial virus infection

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Cyclic production of vesicular stomatitis virus caused by defective interfering particles

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Productive infection of isolated human alveolar macrophages by respiratory syncytial virus

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Characteristics of a persistent respiratory syncytial virus infection in HeLa cells

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The association between respiratory syncytial virus infection and the development of childhood asthma: a systematic review of the literature

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Characteristics of a respiratory syncytial virus persistently infected macrophage-like culture

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10.1016/J.VIROL.2009.03.008

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Europe PubMed Central

PubMed publication ID

15 September 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

15 September 2017

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