(Q40000483)

16 September 2017

title

Induction of HIF-2alpha is dependent on mitochondrial O2 consumption in an O2-sensitive adrenomedullary chromaffin cell line (English)

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16 September 2017

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2

Colin A Nurse

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16 September 2017

Stephen T Brown

1

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16 September 2017

publication date

19 March 2008

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American Journal of Physiology - Cell Physiology

16 September 2017

published in

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16 September 2017

volume

294

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16 September 2017

issue

6

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16 September 2017

page(s)

C1305-12

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The Qo site of the mitochondrial complex III is required for the transduction of hypoxic signaling via reactive oxygen species production

16 September 2017

cites work

1 reference

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Oxygen sensing requires mitochondrial ROS but not oxidative phosphorylation

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Metabolic targeting of hypoxia and HIF1 in solid tumors can enhance cytotoxic chemotherapy

21 January 2018

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Mitochondrial reactive oxygen species trigger hypoxia-induced transcription

21 January 2018

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Reactive oxygen species generated at mitochondrial complex III stabilize hypoxia-inducible factor-1alpha during hypoxia: a mechanism of O2 sensing

21 January 2018

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Inhibition of mitochondrial respiration elevates oxygen concentration but leaves regulation of hypoxia-inducible factor (HIF) intact

21 January 2018

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O2-sensitive K+ channels in immortalised rat chromaffin-cell-derived MAH cells.

21 January 2018

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Regulation of gene expression by the hypoxia-inducible factors

21 January 2018

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HIF-1 regulates cytochrome oxidase subunits to optimize efficiency of respiration in hypoxic cells

21 January 2018

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Mitochondrial complex III is required for hypoxia-induced ROS production and cellular oxygen sensing

21 January 2018

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Redistribution of intracellular oxygen in hypoxia by nitric oxide: effect on HIF1alpha

21 January 2018

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Regulation of hypoxia-inducible factor 1alpha is mediated by an O2-dependent degradation domain via the ubiquitin-proteasome pathway

21 January 2018

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Hypoxia-inducible factor 1 as a possible target for cancer chemoprevention

21 January 2018

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Mitochondrial dysfunction resulting from loss of cytochrome c impairs cellular oxygen sensing and hypoxic HIF-alpha activation

21 January 2018

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Hypoxic reduction in cellular glutathione levels requires mitochondrial reactive oxygen species

21 January 2018

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Independent function of two destruction domains in hypoxia-inducible factor-alpha chains activated by prolyl hydroxylation

21 January 2018

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The tumour suppressor protein VHL targets hypoxia-inducible factors for oxygen-dependent proteolysis

21 January 2018

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Hypoxic but not anoxic stabilization of HIF-1alpha requires mitochondrial reactive oxygen species

21 January 2018

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Adrenomedullary function in the neonatal rat: responses to acute hypoxia

21 January 2018

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HIF-1, O(2), and the 3 PHDs: how animal cells signal hypoxia to the nucleus

21 January 2018

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Hydroxylation of HIF-1: oxygen sensing at the molecular level

21 January 2018

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Oxygen sensing and HIF-1 activation does not require an active mitochondrial respiratory chain electron-transfer pathway.

21 January 2018

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The hypoxia-responsive transcription factor EPAS1 is essential for catecholamine homeostasis and protection against heart failure during embryonic development

21 January 2018

1 reference

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Regulation of hypoxia-inducible factor is preserved in the absence of a functioning mitochondrial respiratory chain

21 January 2018

1 reference

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Hypoxia-inducible factor 1 is a basic-helix-loop-helix-PAS heterodimer regulated by cellular O2 tension

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPCELL.00007.2008

Acute oxygen-sensing mechanisms

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPCELL.00007.2008

10.1152/AJPCELL.00007.2008

21 January 2018

Identifiers

DOI

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16 September 2017

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