(Q40191456)

18 September 2017

title

Enhanced intra-switch region recombination during immunoglobulin class switch recombination in 53BP1-/- B cells (English)

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18 September 2017

author

Michel C. Nussenzweig

series ordinal

4

object named as

Michel C Nussenzweig

1 reference

Bernardo Reina San-Martin

18 September 2017

series ordinal

1

object named as

Bernardo Reina-San-Martin

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18 September 2017

Junjie Chen

2

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18 September 2017

André Nussenzweig

series ordinal

3

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18 September 2017

language of work or name

English

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publication date

1 January 2007

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European Journal of Immunology

18 September 2017

published in

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18 September 2017

volume

37

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18 September 2017

issue

1

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18 September 2017

page(s)

235-239

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Class-switch recombination: interplay of transcription, DNA deamination and DNA repair

18 September 2017

cites work

1 reference

https://api.crossref.org/works/10.1002%2FEJI.200636789

7 January 2021

Ku80 is required for immunoglobulin isotype switching

1 reference

https://api.crossref.org/works/10.1002%2FEJI.200636789

7 January 2021

Ku70 is required for late B cell development and immunoglobulin heavy chain class switching

1 reference

https://api.crossref.org/works/10.1002%2FEJI.200636789

7 January 2021

H2AX is required for recombination between immunoglobulin switch regions but not for intra-switch region recombination or somatic hypermutation

1 reference

https://api.crossref.org/works/10.1002%2FEJI.200636789

7 January 2021

The SCID but not the RAG-2 gene product is required for S mu-S epsilon heavy chain class switching.

1 reference

https://api.crossref.org/works/10.1002%2FEJI.200636789

7 January 2021

DNA-dependent protein kinase activity is not required for immunoglobulin class switching

1 reference

https://api.crossref.org/works/10.1002%2FEJI.200636789

7 January 2021

Reduced switching in SCID B cells is associated with altered somatic mutation of recombined S regions

1 reference

https://api.crossref.org/works/10.1002%2FEJI.200636789

7 January 2021

IgH class switch recombination to IgG1 in DNA-PKcs-deficient B cells

1 reference

https://api.crossref.org/works/10.1002%2FEJI.200636789

7 January 2021

Internal IgH class switch region deletions are position-independent and enhanced by AID expression

1 reference

https://api.crossref.org/works/10.1002%2FEJI.200636789

7 January 2021

ATM is required for efficient recombination between immunoglobulin switch regions

1 reference

https://api.crossref.org/works/10.1002%2FEJI.200636789

7 January 2021

Role of genomic instability and p53 in AID-induced c-myc-Igh translocations.

1 reference

https://api.crossref.org/works/10.1002%2FEJI.200636789

7 January 2021

AID is required for c-myc/IgH chromosome translocations in vivo

1 reference

https://api.crossref.org/works/10.1002%2FEJI.200636789

7 January 2021

Class switch recombination and hypermutation require activation-induced cytidine deaminase (AID), a potential RNA editing enzyme

1 reference

https://api.crossref.org/works/10.1002%2FEJI.200636789

7 January 2021

Activation-induced cytidine deaminase (AID) deficiency causes the autosomal recessive form of the Hyper-IgM syndrome (HIGM2)

1 reference

https://api.crossref.org/works/10.1002%2FEJI.200636789

7 January 2021

AID mutates E. coli suggesting a DNA deamination mechanism for antibody diversification

1 reference

https://api.crossref.org/works/10.1002%2FEJI.200636789

7 January 2021

AID to overcome the limitations of genomic information

1 reference

https://api.crossref.org/works/10.1002%2FEJI.200636789

7 January 2021

Mismatch recognition and uracil excision provide complementary paths to both Ig switching and the A/T-focused phase of somatic mutation

1 reference

https://api.crossref.org/works/10.1002%2FEJI.200636789

7 January 2021

Inducible DNA breaks in Ig S regions are dependent on AID and UNG

1 reference

https://api.crossref.org/works/10.1002%2FEJI.200636789

7 January 2021

Nibrin functions in Ig class-switch recombination

1 reference

https://api.crossref.org/works/10.1002%2FEJI.200636789

7 January 2021

Immunoglobulin class switch recombination is impaired in Atm-deficient mice

1 reference

https://api.crossref.org/works/10.1002%2FEJI.200636789

7 January 2021

53BP1 links DNA damage-response pathways to immunoglobulin heavy chain class-switch recombination.

1 reference

https://api.crossref.org/works/10.1002%2FEJI.200636789

7 January 2021

AID is required to initiate Nbs1/gamma-H2AX focus formation and mutations at sites of class switching

1 reference

https://api.crossref.org/works/10.1002%2FEJI.200636789

7 January 2021

Genomic instability, endoreduplication, and diminished Ig class-switch recombination in B cells lacking Nbs1.

1 reference

https://api.crossref.org/works/10.1002%2FEJI.200636789

7 January 2021

53BP1 is required for class switch recombination

1 reference

https://api.crossref.org/works/10.1002%2FEJI.200636789

7 January 2021

53BP1, an activator of ATM in response to DNA damage

1 reference

https://api.crossref.org/works/10.1002%2FEJI.200636789

7 January 2021

Methylated lysine 79 of histone H3 targets 53BP1 to DNA double-strand breaks

1 reference

https://api.crossref.org/works/10.1002%2FEJI.200636789

7 January 2021

DNA damage-induced G2-M checkpoint activation by histone H2AX and 53BP1

1 reference

https://api.crossref.org/works/10.1002%2FEJI.200636789

7 January 2021

Histone H2AX phosphorylation is dispensable for the initial recognition of DNA breaks

1 reference

https://api.crossref.org/works/10.1002%2FEJI.200636789

7 January 2021

Accumulation of Checkpoint Protein 53BP1 at DNA Breaks Involves Its Binding to Phosphorylated Histone H2AX

1 reference

https://api.crossref.org/works/10.1002%2FEJI.200636789

7 January 2021

Megabase chromatin domains involved in DNA double-strand breaks in vivo

1 reference

https://api.crossref.org/works/10.1002%2FEJI.200636789

7 January 2021

Role for the BRCA1 C-terminal repeats (BRCT) protein 53BP1 in maintaining genomic stability

1 reference

https://api.crossref.org/works/10.1002%2FEJI.200636789

7 January 2021

p53 Binding protein 53BP1 is required for DNA damage responses and tumor suppression in mice

1 reference

https://api.crossref.org/works/10.1002%2FEJI.200636789

7 January 2021

53BP1 and p53 synergize to suppress genomic instability and lymphomagenesis.

1 reference

https://api.crossref.org/works/10.1002%2FEJI.200636789

7 January 2021

53BP1 cooperates with p53 and functions as a haploinsufficient tumor suppressor in mice

1 reference

https://api.crossref.org/works/10.1002%2FEJI.200636789

7 January 2021

H2AX prevents DNA breaks from progressing to chromosome breaks and translocations

1 reference

https://api.crossref.org/works/10.1002%2FEJI.200636789

7 January 2021

Identifiers

DOI

10.1002/EJI.200636789

1 reference

18 September 2017

1 reference